Flatback Syndrome
FLATBACK SYNDROME - SAGITTAL IMBALANCE
Loss of lumbar lordosis causing forward trunk lean with inability to stand upright
Osteotomy Types
Critical Must-Knows
- Flatback = loss of lumbar lordosis causing sagittal imbalance with forward trunk lean
- SVA (sagittal vertical axis): distance from C7 plumb line to S1; normal under 5cm
- PI-LL mismatch: difference between pelvic incidence and lumbar lordosis; normal under 10 degrees
- Three-column osteotomies: SPO (10 degrees), PSO (30 degrees), VCR (40 degrees)
- Iatrogenic most common cause: distraction instrumentation (Harrington rods)
Examiner's Pearls
- "Patients cannot stand upright without compensation (knee flexion, hip extension)
- "ODI and SF-36 correlate with SVA - most important predictor of disability
- "Always assess pelvic compensation (pelvic tilt, pelvic incidence-lumbar lordosis)
- "Rod fracture risk high if SVA not corrected adequately
High Yield Exam Points
SVA is King
Sagittal Vertical Axis (SVA) is the MOST important parameter! Measure from C7 plumb line to posterosuperior S1. Normal under 5cm. Symptoms when over 5cm. Disability severe when over 10cm. This predicts patient outcomes better than any other parameter.
PI-LL Mismatch
Pelvic Incidence minus Lumbar Lordosis should be under 10 degrees. PI is FIXED (anatomic), LL is VARIABLE (can lose with degeneration). Mismatch over 10 degrees = sagittal imbalance requiring correction. Target: PI-LL under 10 degrees.
Osteotomy Hierarchy
Three-column osteotomies by correction power: SPO (Smith-Petersen) = 1 degree/level, PSO (Pedicle Subtraction) = 30 degrees/level, VCR (Vertebral Column Resection) = 40 degrees/level. PSO most common for flatback. VCR for severe/rigid deformities.
Iatrogenic is Most Common
Harrington rod instrumentation was the classic cause (distraction without lordosis preservation). Now: long fusions to sacrum without lordosis, adjacent segment degeneration, pseudarthrosis. Always assess prior hardware and fusion mass on imaging.
At a Glance
| Aspect | Key Information |
|---|---|
| Definition | Loss of lumbar lordosis causing sagittal imbalance |
| Most common cause | Iatrogenic (Harrington rods, long fusion) |
| Cardinal symptom | Forward trunk lean, cannot stand upright |
| Key measurement | SVA (C7 plumb to S1) - normal under 5cm |
| PI-LL target | Under 10 degrees mismatch |
| Gold standard imaging | Standing full-length lateral spine X-ray |
| First-line surgical | Pedicle Subtraction Osteotomy (PSO) |
| PSO correction | 25-30 degrees lordosis per level |
| Neurologic risk | 10-30% with PSO/VCR |
FLATBACKFlatback Syndrome Features
Memory Hook:The spine is FLAT in the BACK!
SVA-PILLRadiographic Parameters
Memory Hook:Take a PILL to measure SVA!
SPVOsteotomy Options
Memory Hook:SPecial Vertebral procedures - ascending power!
Overview and Epidemiology
Flatback syndrome is a debilitating spinal deformity characterized by loss of normal lumbar lordosis, resulting in sagittal plane imbalance where patients cannot maintain an upright posture without significant compensatory mechanisms.
Definition
Flatback syndrome occurs when the lumbar spine loses its normal lordotic curvature (typically 40-60 degrees from L1-S1), resulting in positive sagittal balance where the C7 plumb line falls anterior to the posterosuperior corner of S1.
Key Concept: Normal spine in sagittal plane = head centered over pelvis with minimal energy expenditure. Flatback = head anterior to pelvis requiring constant muscular compensation.
Epidemiology
Prevalence
| Parameter | Value |
|---|---|
| Historic prevalence | 20-40% after Harrington rod instrumentation |
| Current prevalence | 5-10% after modern spinal fusion |
| Peak age | 50-70 years (iatrogenic), variable (degenerative) |
| Gender | Female predominance (2:1) - more fusions for scoliosis |
Risk Factors
| Category | Specific Factors |
|---|---|
| Iatrogenic | Harrington rods, long fusion to sacrum, flat rod contouring |
| Degenerative | Disc collapse, compression fractures, ankylosing spondylitis |
| Post-surgical | Pseudarthrosis, junctional kyphosis, hardware failure |
| Patient factors | Osteoporosis, smoking, obesity, prior laminectomy |
Historical Context
Exam Pearl
Harrington Era (1960s-1990s): Distraction instrumentation for scoliosis correction. Straightened spine in coronal plane but ELIMINATED lumbar lordosis. Result: epidemic of flatback syndrome. Modern instrumentation preserves lordosis with contoured rods and pedicle screw fixation.
Pathophysiology
Sagittal Balance Concepts
Normal Sagittal Alignment
The spine maintains sagittal balance through reciprocal curves:
- Cervical lordosis: 20-40 degrees
- Thoracic kyphosis: 20-40 degrees
- Lumbar lordosis: 40-60 degrees (L1-S1)
Energy Efficiency: When C7 plumb line falls within 2-5cm of posterosuperior S1, minimal muscular effort required to maintain upright posture.
Pelvic Parameters (CRITICAL for Exam)
| Parameter | Definition | Normal Range | Clinical Significance |
|---|---|---|---|
| Pelvic Incidence (PI) | Angle from S1 midpoint to hip axis | 35-80 degrees | FIXED anatomic parameter (cannot change) |
| Pelvic Tilt (PT) | Vertical to S1-hip axis line | 10-25 degrees | VARIABLE - increases with compensation |
| Sacral Slope (SS) | S1 endplate to horizontal | 30-50 degrees | VARIABLE - decreases in flatback |
| PI = PT + SS | Mathematical relationship | Always true | Use to check measurement accuracy |
Lumbar Lordosis (LL): Should approximately equal Pelvic Incidence (PI-LL under 10 degrees)
Pathophysiology of Imbalance
Stage 1: Loss of Lordosis
- Iatrogenic: Flat rod contouring, distraction forces
- Degenerative: Disc collapse, compression fractures
- Result: Lumbar lordosis decreases below PI
Stage 2: Pelvic Compensation
- Pelvis rotates posteriorly (increased pelvic tilt)
- Sacral slope decreases
- Goal: Bring C7 plumb line back over sacrum
- Energy cost: Moderate increase
Stage 3: Hip and Knee Compensation
- Pelvic compensation exhausted
- Hip extension (limited by hip flexors)
- Knee flexion (to shift center of mass posterior)
- Energy cost: Severe increase, early fatigue
Stage 4: Decompensation
- All compensatory mechanisms exhausted
- Fixed forward trunk lean
- Inability to stand upright without support
- Severe disability
Compensation Exhaustion: Patients with high pelvic tilt (over 25 degrees) have exhausted pelvic compensation. These patients have SEVERE disability and limited reserve for further degeneration. Surgical correction should restore balance to avoid progression.
Etiologies
Iatrogenic Flatback (Most Common)
Harrington Rod Era
- Distraction instrumentation without lordosis preservation
- Long fusions (often to L5 or sacrum)
- Flat rod contouring (no sagittal profile)
- Result: Complete loss of lumbar lordosis
Modern Instrumentation Issues
- Undercorrection of lordosis in long fusions
- Flat rod placement in degenerative fusions
- Pseudarthrosis with rod fracture and kyphosis
- Adjacent segment degeneration above fusion
Prevention Strategies (Modern)
- Contoured rods matching normal lordosis (60-70 degrees L1-S1)
- Pedicle screw fixation for lordosis control
- Interbody cages with lordotic angles (20-30 degrees)
- Avoid long fusions unless necessary
This section describes the causes of iatrogenic flatback syndrome.
Clinical Presentation
Symptoms
Cardinal Symptom: Forward Trunk Inclination
Patients describe:
- Inability to stand upright
- "Looking at the ground" when walking
- Needing to lean on shopping cart or walker
- Severe fatigue with standing or walking
Pain Distribution
| Location | Mechanism | Characteristics |
|---|---|---|
| Paraspinal muscles | Constant isometric contraction | Burning, aching, fatigue |
| Thighs (anterior) | Hip extension compensation | Cramping, tightness |
| Knees | Flexion compensation | Aching, early arthritis |
| Buttocks | Gluteal fatigue | Weakness, pain with standing |
Functional Limitations
- Standing tolerance: Often under 15-30 minutes
- Walking distance: Limited by fatigue, not neurogenic claudication
- Social impact: Unable to make eye contact, difficulty with activities
- ADL impairment: Cannot stand to cook, shop, socialize
Physical Examination
Inspection
| Finding | Significance |
|---|---|
| Forward trunk lean | Cannot stand upright without support |
| Knee flexion | Compensatory mechanism |
| Hip extension | Limited by tight hip flexors |
| Gait | Shuffling, antalgic, requires assistive device |
Palpation
- Paraspinal muscle spasm and tenderness
- Loss of lumbar lordotic curve
- Step-off if spondylolisthesis present
- Hardware palpable if prior surgery
Range of Motion
- Lumbar extension: Severely limited or absent
- Lumbar flexion: Often preserved
- Hip extension: Limited (Thomas test positive)
Neurologic Examination
- Usually normal (not a neurologic condition)
- Assess for radiculopathy if foraminal stenosis
- Assess for myelopathy if cervical compensation
Special Tests
Flatback Special Tests
| Test | Technique | Positive Finding |
|---|---|---|
| Sagittal balance test | Patient flexes forward, attempts to stand upright | Cannot achieve upright position without knee flexion |
| Wall test | Patient stands with back to wall, attempts to touch wall with back of head | Head far anterior to wall (over 15cm abnormal) |
| Scoliosis forward bend test | Patient bends forward from waist | Assess coronal deformity if scoliosis present |
Impact on Quality of Life
Patient-Reported Outcomes
Studies show:
- ODI (Oswestry Disability Index) correlates strongly with SVA
- SF-36 physical function severely impaired
- Depression and anxiety common (chronic pain, disability)
Correlation with SVA
| SVA | Disability Level |
|---|---|
| Under 5cm | Minimal disability |
| 5-10cm | Moderate disability |
| Over 10cm | Severe disability |
Investigations
Radiographic Assessment (ESSENTIAL)
Standing Full-Length Lateral Spine Radiograph
Technique Requirements:
- True standing position (no leaning, no support)
- Arms positioned: Fists on clavicles or grasping horizontal bars
- Film includes: Skull to femoral heads
- Cassette: 36-inch length minimum
CRITICAL: Radiographs MUST be taken STANDING. Supine films cannot assess sagittal balance and will underestimate deformity. Patients must stand unsupported with arms positioned to clear thoracic spine.
Key Radiographic Measurements
Sagittal Vertical Axis (SVA)
| Measurement | Technique | Normal | Abnormal |
|---|---|---|---|
| SVA | Plumb line from C7 vertebral body center to posterosuperior S1 | Under 5cm | Over 5cm |
| Interpretation | Positive = C7 anterior to S1 (flatback) | 0-5cm | Over 10cm = severe |
Pelvic Parameters
| Parameter | Measurement | Normal | Flatback |
|---|---|---|---|
| Pelvic Incidence (PI) | Angle: S1 midpoint to hip axis, perpendicular to S1 | 35-80 degrees | FIXED (anatomic) |
| Pelvic Tilt (PT) | Angle: vertical to line from S1 to hip axis | 10-25 degrees | Over 25 degrees (compensated) |
| Sacral Slope (SS) | Angle: S1 endplate to horizontal | 30-50 degrees | Under 30 degrees |
Lumbar Lordosis (LL)
- Measurement: Cobb angle from superior endplate L1 to superior endplate S1
- Normal: 40-60 degrees (approximately equals PI)
- Flatback: Under 30 degrees
PI-LL Mismatch
- Calculation: Pelvic Incidence minus Lumbar Lordosis
- Normal: Under 10 degrees
- Symptomatic: Over 10 degrees
- Severe: Over 20 degrees
Exam Pearl
PI-LL Mismatch is the KEY surgical parameter: Goal of surgery is to restore PI-LL to under 10 degrees. For example, if PI = 60 degrees, target LL = 50-70 degrees (within 10 degrees). This predicts outcomes better than SVA alone.
Advanced Imaging
CT Scan
Indications:
- Surgical planning (bony anatomy, pedicle size)
- Assessment of prior fusion mass
- Hardware evaluation (loosening, fracture)
- Osteotomy level selection
MRI
Indications:
- Assess neural compression (stenosis, radiculopathy)
- Disc degeneration and remaining disc health
- Pseudarthrosis evaluation (fluid signal at fusion site)
- Spinal cord integrity if myelopathy suspected
Flexion-Extension Radiographs
- Assess mobility at adjacent segments
- Identify pseudarthrosis (motion at fusion site)
- Plan osteotomy location (mobile vs. fused segments)
Classification of Flatback Deformity
Ames-ISSG Classification (Most Commonly Used)
Based on SVA and PT:
| Type | SVA | PT | Interpretation |
|---|---|---|---|
| Type 0 | Under 5cm | Under 25 degrees | Compensated, minimal symptoms |
| Type I | 5-10cm | Under 25 degrees | Mild imbalance |
| Type II | Over 10cm | Under 25 degrees | Severe imbalance, compensation failing |
| Type III | Any | Over 25 degrees | Severe, pelvic compensation exhausted |
Surgical Implications:
- Type 0: Conservative management
- Type I: Consider surgery if symptomatic
- Type II-III: Surgery usually indicated
Management Algorithm

Non-Operative Treatment
Efficacy: Limited for established flatback syndrome. May provide symptomatic relief but does NOT correct deformity.
Physiotherapy
Goals:
- Strengthen core musculature
- Improve hip flexor flexibility
- Maintain existing lumbar motion
Exercises:
| Exercise Type | Examples | Benefit |
|---|---|---|
| Core strengthening | Planks, bridges, dead bugs | Improve paraspinal endurance |
| Hip flexor stretching | Thomas stretch, kneeling lunge | Reduce compensatory hip extension |
| Hamstring stretching | Seated hamstring stretch | Allow pelvic rotation |
| Postural training | Wall slides, chin tucks | Awareness and positioning |
Limitations: Cannot restore lost lordosis or correct fixed deformity.
Medications
| Class | Examples | Use | Limitations |
|---|---|---|---|
| NSAIDs | Ibuprofen, naproxen | Muscle inflammation, pain | GI side effects, limited efficacy |
| Muscle relaxants | Cyclobenzaprine | Paraspinal spasm | Sedation, not for long-term |
| Neuropathic agents | Gabapentin, pregabalin | If radicular component | Minimal benefit for mechanical pain |
Interventional Procedures
Epidural Steroid Injections
- Indication: Radiculopathy from foraminal stenosis
- Efficacy: Temporary relief (weeks to months)
- NOT effective for mechanical back pain from imbalance
Facet Injections
- Indication: Facet-mediated pain
- Efficacy: Variable, temporary
- NOT a treatment for sagittal imbalance
Assistive Devices
| Device | Use | Limitation |
|---|---|---|
| Walker with forearm supports | Unloads spine, allows forward lean | Dependence, social stigma |
| Cane | Minimal support | Insufficient for severe flatback |
| Corset/brace | May reduce pain | Does NOT correct deformity |
Natural History
Untreated Flatback Syndrome:
- Progressive worsening of SVA and PT
- Increasing disability and pain
- Adjacent segment degeneration above prior fusion
- Hip and knee arthritis from compensatory postures
Indications for Surgery:
- Failed conservative management (3-6 months trial)
- Severe disability (ODI over 40)
- SVA over 5cm with symptoms
- PI-LL mismatch over 10 degrees with symptoms
- Progressive deformity
Conservative management provides symptom control but rarely resolves flatback.
Surgical Management
Goals of Surgical Correction
Radiographic Goals:
- SVA under 5cm (ideally 0-3cm)
- PI-LL mismatch under 10 degrees
- Restore lumbar lordosis to match pelvic incidence
- Maintain coronal balance
Clinical Goals:
- Allow upright posture without compensation
- Reduce pain and disability
- Improve walking tolerance and ADLs
- Minimize complications
Surgical Options Overview
Smith-Petersen Osteotomy (SPO)
Type: Posterior column osteotomy (Schwab Grade I)
Technique:
- Remove inferior facet, superior facet, and ligamentum flavum
- Creates V-shaped gap in posterior elements
- Closes gap to create lordosis
- REQUIRES mobile anterior column (disc or pseudarthrosis)
Correction: 1 degree per level (10 degrees if multiple levels)
Indications:
- Mild sagittal imbalance (SVA under 10cm)
- PI-LL mismatch under 20 degrees
- Mobile anterior column present
- Multiple level correction planned
Advantages:
- Lowest complication rate
- Preserves vertebral body
- Can be performed at multiple levels
Disadvantages:
- Limited correction per level
- Requires mobile disc
- Risk of anterior column fracture if forced closure
Complications:
- Neurologic injury: 2-5%
- Vertebral body fracture: 5-10%
- Inadequate correction: Common if used alone
This describes the Smith-Petersen posterior column osteotomy technique.
Surgical Planning
Preoperative Assessment:
| Parameter | Assessment | Surgical Plan |
|---|---|---|
| SVA | 5-10cm | Single-level PSO may suffice |
| SVA | Over 10cm | May need 2-level PSO or PSO + SPO |
| PI-LL | 20-30 degrees | Single-level PSO (30 degrees correction) |
| PI-LL | Over 40 degrees | Consider VCR or 2-level PSO |
| Prior fusion | Solid fusion mass | Osteotomy through fusion or adjacent |
| Osteoporosis | DEXA T-score under -2.5 | Optimize bone health, consider cement augmentation |
Osteotomy Level Selection:
| Level | Advantages | Disadvantages |
|---|---|---|
| L2 | Large vertebral body, high lordosis potential | High in lumbar spine, junctional stress |
| L3 | Most common PSO level, large body, safer | Moderate lordosis contribution |
| L4 | Good lordosis, mid-lumbar | Smaller body, L5 root at risk |
| L5 | Maximum lordosis potential | Small body, sacral roots at risk, NOT recommended |
Most Common PSO Level: L3 (balance of safety and correction)
Surgical Technique: Pedicle Subtraction Osteotomy (PSO)
Patient Positioning:
- Prone on Jackson table or radiolucent frame
- Hips extended (allows lordosis)
- Arms abducted 90 degrees
- Neuromonitoring leads placed
Instrumentation:
- Exposure: Midline incision, subperiosteal dissection
- Screw placement: Pedicle screws 3-4 levels above and below osteotomy
- Temporary rods: Place to maintain stability
Osteotomy Steps:
- Laminectomy: Complete removal of lamina, spinous process at osteotomy level
- Facetectomy: Remove inferior and superior facets bilaterally
- Pedicle resection: Remove pedicles bilaterally (identify nerve roots)
- Posterior body resection:
- Remove posterior 1/3 to 1/2 of vertebral body
- Create 30-40 degree wedge
- Taper resection laterally (egg-shaped)
- Nerve root decompression: Mobilize nerve roots, protect during closure
Closure:
- Remove temporary rods
- Place final rods (pre-contoured with lordosis)
- Close osteotomy site SLOWLY:
- 1-2mm at a time
- Check neuromonitoring after each increment
- Use cantilever maneuver
- Compress instrumentation
- Decorticate fusion bed
- Apply bone graft and BMP
Closure and Hemostasis:
- Large drains (hemovac)
- Layered closure over drains
- Subfascial drain placement
Operative time for single-level PSO is typically 4-6 hours.
Complications
Intraoperative Complications
Neurologic Injury
| Type | Incidence | Mechanism | Management |
|---|---|---|---|
| Motor deficit | 5-10% | Direct injury, ischemia, hematoma | Stop closure, release compression, imaging |
| Sensory deficit | 15-25% | Nerve root traction | Often transient, monitor |
| Cauda equina | 1-2% | Severe canal compromise | Immediate decompression |
Vascular Injury
| Vessel | Incidence | Risk Factors | Management |
|---|---|---|---|
| Aorta | 0.5-1% | PSO at L4-5, osteoporotic collapse | Vascular surgery consult, open repair |
| Vena cava | 0.5-1% | Anterior perforation | Direct repair, difficult access |
| Lumbar arteries | 5-10% | Lateral dissection | Usually self-limiting, electrocautery |
Hemorrhage
- Average blood loss PSO: 1-3 liters
- Average blood loss VCR: 3-5 liters
- Transfusion rate: 70-90%
- Consider cell saver, antifibrinolytics (tranexamic acid)
Early Postoperative Complications (Under 6 Weeks)
Neurologic
- New or worsening deficit: 10-30% (most recover)
- Epidural hematoma: 2-5% (may require evacuation)
- Nerve root injury: 5-10% (usually transient)
Cardiopulmonary
| Complication | Incidence | Risk Factors | Prevention |
|---|---|---|---|
| PE/DVT | 5-10% | Long surgery, immobility | Chemoprophylaxis, SCDs |
| Pneumonia | 5-8% | Prolonged intubation, pain | Early mobilization, incentive spirometry |
| Ileus | 10-20% | Bowel retraction, narcotics | Early feeding, bowel regimen |
Wound Complications
- Infection (superficial): 5-10%
- Infection (deep): 3-5%
- Seroma/hematoma: 5-10%
- CSF leak: 3-5%
Late Complications (Over 6 Weeks)
Proximal Junctional Kyphosis (PJK)
| Feature | Details |
|---|---|
| Incidence | 20-40% after PSO |
| Definition | Kyphosis over 10 degrees at upper instrumented vertebra (UIV) |
| Risk factors | Stopping at thoracolumbar junction, osteoporosis, overcorrection |
| Prevention | Extend to upper thoracic spine (T9-T10), avoid overcorrection |
| Treatment | Extension of fusion if symptomatic or progressive |
Rod Fracture
| Parameter | Details |
|---|---|
| Incidence | 10-20% overall (higher if SVA over 5cm postop) |
| Timing | 6 months to 2 years |
| Risk factors | Undercorrection (SVA over 5cm), pseudarthrosis, single rods |
| Prevention | Adequate SVA correction, dual rods, solid fusion |
| Treatment | Revision if symptomatic or progressive deformity |
Pseudarthrosis
- Incidence: 10-20% at osteotomy site
- Risk factors: Smoking, osteoporosis, infection, rod fracture
- Diagnosis: CT (motion, lucency), rod fracture
- Treatment: Revision fusion, BMP, anterior support
Implant Failure
- Screw pullout: 5-10% (osteoporosis, overcorrection forces)
- Screw fracture: 2-5%
- Cross-link fracture: 5-10%
Complication Prevention Strategies
Complication Prevention
| Complication | Prevention Strategy |
|---|---|
| Neurologic injury | Slow closure, neuromonitoring, adequate decompression, avoid overstretching |
| Vascular injury | Careful anterior dissection, stay midline, palpate aorta |
| Rod fracture | Adequate SVA correction (under 5cm), dual rods, solid fusion |
| PJK | Extend to upper thoracic, avoid overcorrection, prophylactic vertebroplasty UIV |
| Infection | Prophylactic antibiotics, meticulous hemostasis, vancomycin powder |
| Pseudarthrosis | BMP, bone graft, avoid smoking, optimize nutrition |
Postoperative Care
Immediate Postoperative (Day 0-2)
ICU Monitoring:
- Hemodynamic stability (blood loss often significant)
- Neurologic examination every 2 hours
- Drain output monitoring
- Pain control (PCA, epidural)
Early Mobilization:
- Out of bed to chair Day 1 (if stable)
- Walking with PT Day 2-3
- No brace typically required (rigid internal fixation)
Hospital Stay (Days 3-7)
Monitoring:
- Daily neurologic examination
- Drain removal when output under 30mL per 8 hours
- Early radiographs (AP and lateral) to assess alignment
- Transition to oral pain medications
Physical Therapy:
- Progressive ambulation
- Core strengthening (isometric)
- ADL training
- Stair climbing prior to discharge
Discharge Criteria:
- Hemodynamically stable
- Neurologically stable or improving
- Pain controlled on oral medications
- Ambulating independently or with walker
- Drains removed
Outpatient Follow-Up
Timeline:
| Timepoint | Assessment | Imaging |
|---|---|---|
| 2 weeks | Wound check, suture removal | None (unless concern) |
| 6 weeks | Clinical exam, pain assessment | AP/Lateral X-rays (standing) |
| 3 months | Neurologic exam, function | AP/Lateral X-rays (standing) |
| 6 months | Function, return to activities | AP/Lateral X-rays |
| 1 year | Final assessment | AP/Lateral X-rays, CT if concern for fusion |
| 2 years | Long-term outcome | As needed |
Radiographic Assessment:
- SVA (goal: under 5cm)
- PI-LL mismatch (goal: under 10 degrees)
- Hardware integrity (rod fracture?)
- Fusion mass (bridging bone?)
- Proximal junctional kyphosis assessment
Activity Restrictions
| Activity | Restriction Period | Rationale |
|---|---|---|
| Lifting | No lifting over 10 lbs for 3 months | Protect fusion, prevent hardware failure |
| Driving | No driving for 6 weeks (narcotics) | Safety, pain control |
| Return to work (desk) | 3 months | Depends on pain, function |
| Return to work (manual) | 6-12 months | Heavy labor risk to fusion |
| Sports | Light activities 6 months, full 1 year | Fusion maturation |
Bracing
Typically NOT Required:
- Rigid pedicle screw fixation provides stability
- Bracing does NOT improve fusion rates in modern instrumentation
- May use soft lumbar corset for comfort (not structural)
Exceptions:
- Osteoporotic bone (concern for screw pullout)
- Extended laminectomy (concern for instability)
- Patient preference for comfort
Postoperative care focuses on neurologic monitoring and progressive mobilization.
Outcomes and Evidence
Patient-Reported Outcomes
Improvement Rates:
| Outcome Measure | Preoperative | 2-Year Postoperative | Improvement |
|---|---|---|---|
| ODI | 40-60 (severe disability) | 20-30 (moderate) | 40-60% improvement |
| SF-36 Physical | 25-35 | 45-55 | Significant improvement |
| Back pain VAS | 7-8/10 | 3-4/10 | 50% reduction |
| Leg pain VAS | 5-6/10 | 2-3/10 | 50% reduction |
Patient Satisfaction:
- 70-80% satisfied or very satisfied
- 10-20% satisfied with reservations
- 5-10% dissatisfied
Radiographic Outcomes
SVA Correction:
| Preoperative SVA | Postoperative SVA | Correction Achieved |
|---|---|---|
| 12-15cm (average) | 3-5cm (average) | 8-10cm improvement |
| Target: Under 5cm | Achieved in 70-80% | Correlates with satisfaction |
PI-LL Correction:
| Preoperative PI-LL | Postoperative PI-LL | Correction |
|---|---|---|
| 25-35 degrees (average) | 5-15 degrees (average) | 20-25 degrees improvement |
| Target: Under 10 degrees | Achieved in 60-70% | Correlates with rod fracture risk |
Schwab et al. (2012) - Sagittal Realignment Outcomes
- 298 patients with adult spinal deformity analyzed for sagittal parameters
- SVA most strongly correlated with disability (ODI, SF-36)
- PI-LL mismatch over 10 degrees associated with worse outcomes
- Goal: SVA under 5cm, PI-LL under 10 degrees for optimal outcomes
Smith et al. (2014) - PSO Outcomes and Complications
- Multicenter review of 206 patients undergoing PSO for sagittal deformity
- Average lordosis correction: 28 degrees per level
- Major complications in 35% (neurologic 10-15%, vascular 2%, infection 5%)
- Revision rate: 17% at 2 years (rod fracture, PJK most common)
Bridwell et al. (2013) - PI-LL Mismatch Impact
- Retrospective analysis of 286 patients with adult deformity surgery
- PI-LL mismatch under 10 degrees: 75% good outcomes
- PI-LL mismatch over 20 degrees: 45% good outcomes
- Undercorrection (PI-LL over 10 degrees) associated with rod fracture risk
Lagrone et al. (1988) - First Description of Flatback
- First systematic description of flatback syndrome after Harrington instrumentation
- Loss of lumbar lordosis identified as primary pathology
- Compensatory mechanisms including pelvic retroversion and knee flexion described
- Established foundation for understanding sagittal balance deformity
Complication Rates (Meta-Analysis)
| Complication | Incidence | Severity |
|---|---|---|
| Neurologic (any) | 10-30% | 50% resolve by 1 year |
| Neurologic (permanent) | 5-10% | Motor deficit most concerning |
| Rod fracture | 10-20% | Higher if SVA over 5cm postop |
| PJK | 20-40% | 10-15% require revision |
| Pseudarthrosis | 10-20% | 5-10% require revision |
| Infection | 5-10% | Deep infection 3-5% |
| Reoperation (any reason) | 20-30% | At 2-5 years |
Factors Predicting Outcomes
Good Outcome Predictors:
- Postoperative SVA under 5cm
- PI-LL mismatch under 10 degrees
- Solid fusion at 1 year
- No neurologic complications
Poor Outcome Predictors:
- Undercorrection (SVA over 5cm)
- Rod fracture
- Proximal junctional kyphosis
- Neurologic injury
- Infection
References
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Smith JS, Shaffrey CI, Ames CP, et al. Assessment of symptomatic rod fracture after posterior instrumented fusion for adult spinal deformity. Neurosurgery. 2012;71(4):862-7.
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Bridwell KH, Glassman S, Horton W, et al. Does treatment (nonoperative and operative) improve the two-year quality of life in patients with adult symptomatic lumbar scoliosis: a prospective multicenter evidence-based medicine study. Spine. 2009;34(20):2171-8.
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Bridwell KH, Lewis SJ, Lenke LG, et al. Pedicle subtraction osteotomy for the treatment of fixed sagittal imbalance. J Bone Joint Surg Am. 2003;85(3):454-63.
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Kim YJ, Bridwell KH, Lenke LG, et al. Proximal junctional kyphosis in adult spinal deformity after segmental posterior spinal instrumentation and fusion: minimum five-year follow-up. Spine. 2008;33(20):2179-84.
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Yagi M, Akilah KB, Boachie-Adjei O. Incidence, risk factors and classification of proximal junctional kyphosis: surgical outcomes review of adult idiopathic scoliosis. Spine. 2011;36(1):E60-8.
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Smith JS, Klineberg E, Schwab F, et al. Change in classification grade by the SRS-Schwab Adult Spinal Deformity Classification predicts impact on health-related quality of life measures: prospective analysis of operative and nonoperative treatment. Spine. 2013;38(19):1663-71.
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Schwab FJ, Blondel B, Bess S, et al. Radiographical spinopelvic parameters and disability in the setting of adult spinal deformity: a prospective multicenter analysis. Spine. 2013;38(13):E803-12.
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Kim HJ, Bridwell KH, Lenke LG, et al. Patients with proximal junctional kyphosis requiring revision surgery have higher postoperative lumbar lordosis and larger sagittal balance corrections. Spine. 2014;39(9):E576-80.
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Exam Viva Scenarios
Practice these scenarios to excel in your viva examination
Scenario 1: Flatback After Harrington Rod
"A 62-year-old woman presents with severe back pain and forward trunk lean. She had scoliosis surgery with Harrington rods 30 years ago. She can only stand upright for 10 minutes and requires a walker. Full-length standing lateral radiograph shows SVA of 14cm, PI of 58 degrees, LL of 25 degrees, and PT of 32 degrees."
Scenario 2: Degenerative Flatback
"A 68-year-old man with no prior spine surgery presents with progressive forward lean over 5 years. He has severe back and leg fatigue with standing and walking. Standing lateral shows SVA 8cm, PI 62 degrees, LL 38 degrees, PT 28 degrees. Multiple disc collapse L2-5. How would you manage this?"
Scenario 3: Postoperative Neurologic Deficit
"You are performing a PSO at L3 for flatback correction. After closing the osteotomy, the neurophysiologist reports loss of motor evoked potentials in bilateral lower extremities. What do you do?"
MCQ Practice Points
Exam Pearl
Q: What is flatback syndrome and what are its causes?
A: Flatback syndrome: Loss of normal lumbar lordosis causing positive sagittal balance (sagittal vertical axis greater than 5cm anterior to S1). Patients lean forward and cannot stand upright without hip/knee flexion. Causes: (1) Iatrogenic - distraction instrumentation (Harrington rods), hypolordotic fusion constructs; (2) Degenerative disc disease with disc height loss; (3) Vertebral fractures; (4) Ankylosing spondylitis; (5) Adjacent segment degeneration after fusion.
Exam Pearl
Q: What are the clinical features and compensatory mechanisms in flatback syndrome?
A: Symptoms: Back pain (fatigue), inability to stand erect, forward stooped posture, need to lean on objects. Compensatory mechanisms (from spine distally): Thoracic hyperkyphosis; Hip hyperextension; Knee flexion; Ankle dorsiflexion. With exhaustion, compensatory mechanisms fail and patient leans progressively forward. Physical exam: Forward trunk inclination; Positive sagittal balance; Hip flexion contractures may develop; Diminished lumbar lordosis or frank kyphosis.
Exam Pearl
Q: How do you assess sagittal balance radiographically?
A: Standing full-spine radiographs essential. Key measurements: SVA (sagittal vertical axis): C7 plumb line to posterior S1 - normal less than 5cm, positive values indicate anterior shift. Pelvic incidence (PI): Fixed anatomic value. Lumbar lordosis (LL): Should approximately equal PI ± 10°. Pelvic tilt (PT): Increases with compensation (pelvis retroversion). T1 pelvic angle: Global sagittal alignment measure. Goal is to restore PI-LL match and normalize SVA.
Exam Pearl
Q: What are the surgical options for correcting flatback syndrome?
A: Osteotomy techniques (increasing correction): Smith-Petersen osteotomy (SPO): Posterior column shortening through facets, 10° per level. Pedicle subtraction osteotomy (PSO): Wedge resection through all three columns, 30-35° correction per level. Vertebral column resection (VCR): Complete removal of vertebral segment, greatest correction but highest risk. Extension of fusion: Address adjacent segment disease. Selection based on magnitude of deformity and prior fusion status. Often multiple osteotomies required.
Exam Pearl
Q: What are the complications specific to flatback correction surgery?
A: Neurological injury: Especially with PSO/VCR - cord monitoring essential; Root injury from nerve stretch or direct trauma. Pseudarthrosis: High mechanical load at osteotomy site. Hardware failure: Rod fracture at osteotomy site (stress riser). Proximal junctional kyphosis: Failure above fusion construct. Adjacent segment disease: Increased stress at adjacent levels. Medical complications: High blood loss, prolonged surgery, age-related comorbidities. Mortality rates 1-5% in revision deformity surgery.
Australian Context
Tertiary Referral: Flatback syndrome surgery is performed at major spine centres with fellowship-trained deformity surgeons. Intraoperative neuromonitoring (SSEPs, MEPs, EMG) is mandatory for osteotomy procedures.
PBS-Subsidised Care: Bone-targeted agents for osteoporosis optimization are PBS-listed. Physiotherapy and pain management are available through Enhanced Primary Care (EPC) plans.
Pre-operative Workup: Bone densitometry (DEXA), nutritional assessment, and cardiopulmonary optimization are standard before major deformity surgery. High-risk patients require multidisciplinary perioperative care.
Rehabilitation: Inpatient rehabilitation following flatback correction is typically 2-4 weeks in dedicated spinal units. Long-term physiotherapy for postural retraining and core strengthening is recommended.
FLATBACK SYNDROME
High-Yield Exam Summary
Key Definitions
- •Flatback = loss of lumbar lordosis causing sagittal imbalance (forward trunk lean)
- •SVA = C7 plumb to posterosuperior S1; normal under 5cm, severe over 10cm
- •PI-LL mismatch = Pelvic Incidence minus Lumbar Lordosis; normal under 10 degrees
- •Pelvic compensation = posterior pelvic tilt to restore balance (PT normal 10-25 degrees)
Radiographic Parameters
- •SVA (Sagittal Vertical Axis): Normal under 5cm, symptoms over 5cm, severe over 10cm
- •PI (Pelvic Incidence): FIXED anatomic (35-80 degrees), does NOT change
- •LL (Lumbar Lordosis): VARIABLE (40-60 degrees L1-S1), should approximate PI
- •PT (Pelvic Tilt): VARIABLE compensation (10-25 degrees), over 25 = exhausted
- •PI = PT + SS (sacral slope) - use to check measurement accuracy
Etiologies
- •Iatrogenic (most common): Harrington rods, flat fusion, long fusion to sacrum
- •Degenerative: Disc collapse, compression fractures, ankylosing spondylitis
- •Post-laminectomy: Extensive posterior element removal causing kyphosis
- •Traumatic: Vertebral compression fractures, pelvic malunion
Clinical Presentation
- •Forward trunk lean with inability to stand upright (cardinal finding)
- •Compensatory mechanisms: knee flexion, hip extension, pelvic retroversion
- •Severe back and leg fatigue (paraspinals, hip flexors, quadriceps)
- •ODI and SF-36 correlate with SVA (worse disability with higher SVA)
- •Standing tolerance often under 15-30 minutes
Imaging Requirements
- •Standing full-length lateral spine (skull to femoral heads) - MUST be standing
- •Arms positioned: fists on clavicles or grasping horizontal bars
- •Measure SVA, PI, PT, SS, LL (all on same standing lateral film)
- •CT for surgical planning (bony anatomy, pedicle size, prior fusion)
- •MRI if radiculopathy or stenosis (neural compression assessment)
Surgical Goals
- •Radiographic: SVA under 5cm (ideally 0-3cm), PI-LL under 10 degrees
- •Clinical: Upright posture without compensation, reduce pain/disability
- •Restore lumbar lordosis to approximately match pelvic incidence
- •Avoid complications: neurologic injury, rod fracture, PJK
Osteotomy Options
- •SPO (Smith-Petersen): Posterior column only, 1 degree/level, needs mobile disc
- •PSO (Pedicle Subtraction): 3-column, 30 degrees/level, workhorse for flatback
- •VCR (Vertebral Column Resection): Complete vertebrectomy, 40 degrees/level, severe cases
- •PSO at L3 most common (large body, safer than L4-5, good lordosis contribution)
PSO Technique Key Points
- •Instrumentation 3-4 levels above/below, temporary rods for stability
- •Laminectomy, pedicle removal, posterior body wedge (30-40 degrees)
- •Close osteotomy SLOWLY (1-2mm increments, check neuromonitoring)
- •Cantilever closure technique, neural elements shorten (accordion)
- •BMP at osteotomy site, decorticate fusion bed, rigid fixation
Complications and Rates
- •Neurologic injury: 10-30% (motor 5-10%, sensory 15-25%), 50% resolve by 1 year
- •Rod fracture: 10-20% (higher if SVA over 5cm postop, undercorrection)
- •PJK (Proximal Junctional Kyphosis): 20-40% (extend to upper thoracic to prevent)
- •Pseudarthrosis: 10-20% at osteotomy site (smoking, infection, rod fracture)
- •Vascular injury: 1-3% (aorta, vena cava - L4-5 PSO higher risk)
- •Infection: 5-10% (deep 3-5%), bleeding: 1-3 liters average PSO
Outcomes
- •Patient satisfaction: 70-80% satisfied at 2 years if adequate correction
- •ODI improvement: 40-60% reduction (from 50 preop to 25 postop average)
- •SVA correction: Average 8-10cm improvement (from 14cm to 4cm)
- •Revision rate: 20-30% at 5 years (rod fracture, PJK, pseudarthrosis)
- •Correlation: SVA under 5cm postop = best outcomes, over 5cm = rod fracture risk
Exam Pearls
- •SVA is KING: Most important outcome predictor (correlates with ODI, SF-36)
- •PI-LL target: Under 10 degrees mismatch (predicts rod fracture if over 10)
- •PSO at L3: Most common level (large body, safer, good lordosis)
- •Neurologic loss during closure: STOP, open osteotomy, optimize perfusion
- •Rod fracture = undercorrection: Usually means SVA over 5cm or pseudarthrosis
Australian Context
PBS Medications
- Tranexamic acid: Unrestricted for antifibrinolytic therapy during major spine surgery
- Bone morphogenetic protein (BMP): Not PBS-listed; private purchase approximately $3,000-5,000 per kit
- Teriparatide (Forteo): PBS-listed for severe osteoporosis - consider preoperative optimization
Australian Spine Registry Data
- PSO complication rates in Australia: Neurologic 12-18%, rod fracture 15-22%
- Average hospital stay: 7-10 days for PSO
- Revision surgery rate: 25-30% at 5 years
- Patient satisfaction: 75-80% at 2-year follow-up
eTG Recommendations
Antibiotic Prophylaxis:
- Cefazolin 2g IV at induction, then 2g every 4 hours during surgery
- If penicillin allergic: Vancomycin 15-20mg/kg IV
- Continue for 24 hours postoperatively
- Consider vancomycin powder at wound closure
DVT Prophylaxis:
- Enoxaparin 40mg SC daily starting 12-24 hours postoperatively
- Continue for 4-6 weeks (major deformity surgery)
- Sequential compression devices intraoperatively
- Early mobilization critical
This topic provides comprehensive coverage of flatback syndrome pathophysiology, radiographic assessment (SVA, PI-LL mismatch), conservative management, and surgical correction with Smith-Petersen, PSO, and VCR osteotomies for FRACS examination preparation.