Skip to main content
OrthoVellum
Knowledge Hub

Study

  • Topics
  • MCQs
  • ISAWE
  • Operative Surgery
  • Flashcards

Company

  • About Us
  • Editorial Policy
  • Contact
  • FAQ
  • Blog

Legal

  • Terms of Service
  • Privacy Policy
  • Cookie Policy
  • Medical Disclaimer
  • Copyright & DMCA
  • Refund Policy

Support

  • Help Center
  • Accessibility
  • Report an Issue
OrthoVellum

© 2026 OrthoVellum. For educational purposes only.

Not affiliated with the Royal Australasian College of Surgeons.

Flatback Syndrome

Back to Topics
Contents
0%

Flatback Syndrome

Comprehensive FRACS exam guide to flatback syndrome - loss of lumbar lordosis, sagittal imbalance, pathophysiology, radiographic assessment (SVA, PI-LL mismatch), conservative management, and surgical correction with Smith-Petersen, PSO, and VCR osteotomies.

complete
Updated: 2025-12-25

Flatback Syndrome

High Yield Overview

FLATBACK SYNDROME - SAGITTAL IMBALANCE

Loss of lumbar lordosis causing forward trunk lean with inability to stand upright

5cmSVA threshold for symptoms
10°PI-LL mismatch threshold
30°PSO correction per level
80%Patient satisfaction post-correction

Osteotomy Types

SPO
PatternSmith-Petersen (posterior only)
Treatment10 degrees per level
PSO
PatternPedicle Subtraction (3-column)
Treatment30 degrees per level
VCR
PatternVertebral Column Resection
Treatment40+ degrees per level

Critical Must-Knows

  • Flatback = loss of lumbar lordosis causing sagittal imbalance with forward trunk lean
  • SVA (sagittal vertical axis): distance from C7 plumb line to S1; normal under 5cm
  • PI-LL mismatch: difference between pelvic incidence and lumbar lordosis; normal under 10 degrees
  • Three-column osteotomies: SPO (10 degrees), PSO (30 degrees), VCR (40 degrees)
  • Iatrogenic most common cause: distraction instrumentation (Harrington rods)

Examiner's Pearls

  • "
    Patients cannot stand upright without compensation (knee flexion, hip extension)
  • "
    ODI and SF-36 correlate with SVA - most important predictor of disability
  • "
    Always assess pelvic compensation (pelvic tilt, pelvic incidence-lumbar lordosis)
  • "
    Rod fracture risk high if SVA not corrected adequately

High Yield Exam Points

SVA is King

Sagittal Vertical Axis (SVA) is the MOST important parameter! Measure from C7 plumb line to posterosuperior S1. Normal under 5cm. Symptoms when over 5cm. Disability severe when over 10cm. This predicts patient outcomes better than any other parameter.

PI-LL Mismatch

Pelvic Incidence minus Lumbar Lordosis should be under 10 degrees. PI is FIXED (anatomic), LL is VARIABLE (can lose with degeneration). Mismatch over 10 degrees = sagittal imbalance requiring correction. Target: PI-LL under 10 degrees.

Osteotomy Hierarchy

Three-column osteotomies by correction power: SPO (Smith-Petersen) = 1 degree/level, PSO (Pedicle Subtraction) = 30 degrees/level, VCR (Vertebral Column Resection) = 40 degrees/level. PSO most common for flatback. VCR for severe/rigid deformities.

Iatrogenic is Most Common

Harrington rod instrumentation was the classic cause (distraction without lordosis preservation). Now: long fusions to sacrum without lordosis, adjacent segment degeneration, pseudarthrosis. Always assess prior hardware and fusion mass on imaging.

At a Glance

AspectKey Information
DefinitionLoss of lumbar lordosis causing sagittal imbalance
Most common causeIatrogenic (Harrington rods, long fusion)
Cardinal symptomForward trunk lean, cannot stand upright
Key measurementSVA (C7 plumb to S1) - normal under 5cm
PI-LL targetUnder 10 degrees mismatch
Gold standard imagingStanding full-length lateral spine X-ray
First-line surgicalPedicle Subtraction Osteotomy (PSO)
PSO correction25-30 degrees lordosis per level
Neurologic risk10-30% with PSO/VCR
Mnemonic

FLATBACKFlatback Syndrome Features

F
Forward trunk lean
Cannot stand upright, compensatory knee/hip flexion
L
Lordosis loss
Lumbar spine flat or kyphotic
A
Anterior imbalance
C7 plumb line falls anterior to S1
T
Ten degrees PI-LL
Mismatch threshold for surgery (over 10 degrees)
B
Back pain severe
Paraspinal fatigue from constant compensation
A
Assessment with SVA
Sagittal vertical axis - key measurement
C
Correction with osteotomy
PSO (30 degrees), VCR (40 degrees)
K
Knee and hip compensation
Flexed knees, extended hips to maintain balance

Memory Hook:The spine is FLAT in the BACK!

Mnemonic

SVA-PILLRadiographic Parameters

S
Sagittal Vertical Axis
C7 plumb to S1, normal under 5cm
V
Vertical alignment
Head over pelvis alignment
A
Anterior displacement
Positive SVA = C7 anterior to S1
P
Pelvic Incidence
Fixed anatomic parameter (angle from S1 to hip axis)
I
Incidence minus lordosis
PI-LL mismatch calculation
L
Lumbar Lordosis
Measured L1-S1, should match PI within 10 degrees
L
Less than 10 degrees
Target: PI-LL under 10 degrees mismatch

Memory Hook:Take a PILL to measure SVA!

Mnemonic

SPVOsteotomy Options

S
Smith-Petersen Osteotomy (SPO)
Posterior column only, 1 degree/level, mobile disc needed
P
Pedicle Subtraction Osteotomy (PSO)
3-column, 30 degrees/level, workhorse for flatback
V
Vertebral Column Resection (VCR)
Complete vertebrectomy, 40 degrees, severe deformity

Memory Hook:SPecial Vertebral procedures - ascending power!


Overview and Epidemiology

Flatback syndrome is a debilitating spinal deformity characterized by loss of normal lumbar lordosis, resulting in sagittal plane imbalance where patients cannot maintain an upright posture without significant compensatory mechanisms.

Definition

Flatback syndrome occurs when the lumbar spine loses its normal lordotic curvature (typically 40-60 degrees from L1-S1), resulting in positive sagittal balance where the C7 plumb line falls anterior to the posterosuperior corner of S1.

Key Concept: Normal spine in sagittal plane = head centered over pelvis with minimal energy expenditure. Flatback = head anterior to pelvis requiring constant muscular compensation.

Epidemiology

Prevalence

ParameterValue
Historic prevalence20-40% after Harrington rod instrumentation
Current prevalence5-10% after modern spinal fusion
Peak age50-70 years (iatrogenic), variable (degenerative)
GenderFemale predominance (2:1) - more fusions for scoliosis

Risk Factors

CategorySpecific Factors
IatrogenicHarrington rods, long fusion to sacrum, flat rod contouring
DegenerativeDisc collapse, compression fractures, ankylosing spondylitis
Post-surgicalPseudarthrosis, junctional kyphosis, hardware failure
Patient factorsOsteoporosis, smoking, obesity, prior laminectomy

Historical Context

Exam Pearl

Harrington Era (1960s-1990s): Distraction instrumentation for scoliosis correction. Straightened spine in coronal plane but ELIMINATED lumbar lordosis. Result: epidemic of flatback syndrome. Modern instrumentation preserves lordosis with contoured rods and pedicle screw fixation.

Pathophysiology

Sagittal Balance Concepts

Normal Sagittal Alignment

The spine maintains sagittal balance through reciprocal curves:

  • Cervical lordosis: 20-40 degrees
  • Thoracic kyphosis: 20-40 degrees
  • Lumbar lordosis: 40-60 degrees (L1-S1)

Energy Efficiency: When C7 plumb line falls within 2-5cm of posterosuperior S1, minimal muscular effort required to maintain upright posture.

Pelvic Parameters (CRITICAL for Exam)

ParameterDefinitionNormal RangeClinical Significance
Pelvic Incidence (PI)Angle from S1 midpoint to hip axis35-80 degreesFIXED anatomic parameter (cannot change)
Pelvic Tilt (PT)Vertical to S1-hip axis line10-25 degreesVARIABLE - increases with compensation
Sacral Slope (SS)S1 endplate to horizontal30-50 degreesVARIABLE - decreases in flatback
PI = PT + SSMathematical relationshipAlways trueUse to check measurement accuracy

Lumbar Lordosis (LL): Should approximately equal Pelvic Incidence (PI-LL under 10 degrees)

Pathophysiology of Imbalance

Stage 1: Loss of Lordosis

  • Iatrogenic: Flat rod contouring, distraction forces
  • Degenerative: Disc collapse, compression fractures
  • Result: Lumbar lordosis decreases below PI

Stage 2: Pelvic Compensation

  • Pelvis rotates posteriorly (increased pelvic tilt)
  • Sacral slope decreases
  • Goal: Bring C7 plumb line back over sacrum
  • Energy cost: Moderate increase

Stage 3: Hip and Knee Compensation

  • Pelvic compensation exhausted
  • Hip extension (limited by hip flexors)
  • Knee flexion (to shift center of mass posterior)
  • Energy cost: Severe increase, early fatigue

Stage 4: Decompensation

  • All compensatory mechanisms exhausted
  • Fixed forward trunk lean
  • Inability to stand upright without support
  • Severe disability

Compensation Exhaustion: Patients with high pelvic tilt (over 25 degrees) have exhausted pelvic compensation. These patients have SEVERE disability and limited reserve for further degeneration. Surgical correction should restore balance to avoid progression.

Etiologies

Iatrogenic Flatback (Most Common)

Harrington Rod Era

  • Distraction instrumentation without lordosis preservation
  • Long fusions (often to L5 or sacrum)
  • Flat rod contouring (no sagittal profile)
  • Result: Complete loss of lumbar lordosis

Modern Instrumentation Issues

  • Undercorrection of lordosis in long fusions
  • Flat rod placement in degenerative fusions
  • Pseudarthrosis with rod fracture and kyphosis
  • Adjacent segment degeneration above fusion

Prevention Strategies (Modern)

  • Contoured rods matching normal lordosis (60-70 degrees L1-S1)
  • Pedicle screw fixation for lordosis control
  • Interbody cages with lordotic angles (20-30 degrees)
  • Avoid long fusions unless necessary

This section describes the causes of iatrogenic flatback syndrome.

Degenerative Flatback

Disc-Related

  • Multiple level disc collapse (height loss anteriorly)
  • Progressive loss of disc lordosis
  • Facet arthropathy preventing extension

Vertebral-Related

  • Osteoporotic compression fractures
  • Scheuermann kyphosis (thoracolumbar)
  • Ankylosing spondylitis (fixed kyphosis)

Post-Laminectomy

  • Extensive posterior element removal
  • Facet incompetence
  • Progressive kyphosis (especially thoracolumbar junction)

Degenerative causes progressively decrease lumbar lordosis over time.

Post-Traumatic Flatback

Vertebral Fractures

  • Burst fractures with anterior column collapse
  • Compression fractures (especially multiple levels)
  • Fracture-dislocation with malunion

Pelvic Fractures

  • Sacral fractures changing sacral slope
  • Acetabular fractures altering hip mechanics

Trauma can alter sagittal alignment through bony collapse or malunion.

Clinical Presentation

Symptoms

Cardinal Symptom: Forward Trunk Inclination

Patients describe:

  • Inability to stand upright
  • "Looking at the ground" when walking
  • Needing to lean on shopping cart or walker
  • Severe fatigue with standing or walking

Pain Distribution

LocationMechanismCharacteristics
Paraspinal musclesConstant isometric contractionBurning, aching, fatigue
Thighs (anterior)Hip extension compensationCramping, tightness
KneesFlexion compensationAching, early arthritis
ButtocksGluteal fatigueWeakness, pain with standing

Functional Limitations

  • Standing tolerance: Often under 15-30 minutes
  • Walking distance: Limited by fatigue, not neurogenic claudication
  • Social impact: Unable to make eye contact, difficulty with activities
  • ADL impairment: Cannot stand to cook, shop, socialize

Physical Examination

Inspection

FindingSignificance
Forward trunk leanCannot stand upright without support
Knee flexionCompensatory mechanism
Hip extensionLimited by tight hip flexors
GaitShuffling, antalgic, requires assistive device

Palpation

  • Paraspinal muscle spasm and tenderness
  • Loss of lumbar lordotic curve
  • Step-off if spondylolisthesis present
  • Hardware palpable if prior surgery

Range of Motion

  • Lumbar extension: Severely limited or absent
  • Lumbar flexion: Often preserved
  • Hip extension: Limited (Thomas test positive)

Neurologic Examination

  • Usually normal (not a neurologic condition)
  • Assess for radiculopathy if foraminal stenosis
  • Assess for myelopathy if cervical compensation

Special Tests

Flatback Special Tests

TestTechniquePositive Finding
Sagittal balance testPatient flexes forward, attempts to stand uprightCannot achieve upright position without knee flexion
Wall testPatient stands with back to wall, attempts to touch wall with back of headHead far anterior to wall (over 15cm abnormal)
Scoliosis forward bend testPatient bends forward from waistAssess coronal deformity if scoliosis present

Impact on Quality of Life

Patient-Reported Outcomes

Studies show:

  • ODI (Oswestry Disability Index) correlates strongly with SVA
  • SF-36 physical function severely impaired
  • Depression and anxiety common (chronic pain, disability)

Correlation with SVA

SVADisability Level
Under 5cmMinimal disability
5-10cmModerate disability
Over 10cmSevere disability

Investigations

Radiographic Assessment (ESSENTIAL)

Standing Full-Length Lateral Spine Radiograph

Technique Requirements:

  • True standing position (no leaning, no support)
  • Arms positioned: Fists on clavicles or grasping horizontal bars
  • Film includes: Skull to femoral heads
  • Cassette: 36-inch length minimum

CRITICAL: Radiographs MUST be taken STANDING. Supine films cannot assess sagittal balance and will underestimate deformity. Patients must stand unsupported with arms positioned to clear thoracic spine.

Key Radiographic Measurements

Sagittal Vertical Axis (SVA)

MeasurementTechniqueNormalAbnormal
SVAPlumb line from C7 vertebral body center to posterosuperior S1Under 5cmOver 5cm
InterpretationPositive = C7 anterior to S1 (flatback)0-5cmOver 10cm = severe

Pelvic Parameters

ParameterMeasurementNormalFlatback
Pelvic Incidence (PI)Angle: S1 midpoint to hip axis, perpendicular to S135-80 degreesFIXED (anatomic)
Pelvic Tilt (PT)Angle: vertical to line from S1 to hip axis10-25 degreesOver 25 degrees (compensated)
Sacral Slope (SS)Angle: S1 endplate to horizontal30-50 degreesUnder 30 degrees

Lumbar Lordosis (LL)

  • Measurement: Cobb angle from superior endplate L1 to superior endplate S1
  • Normal: 40-60 degrees (approximately equals PI)
  • Flatback: Under 30 degrees

PI-LL Mismatch

  • Calculation: Pelvic Incidence minus Lumbar Lordosis
  • Normal: Under 10 degrees
  • Symptomatic: Over 10 degrees
  • Severe: Over 20 degrees

Exam Pearl

PI-LL Mismatch is the KEY surgical parameter: Goal of surgery is to restore PI-LL to under 10 degrees. For example, if PI = 60 degrees, target LL = 50-70 degrees (within 10 degrees). This predicts outcomes better than SVA alone.

Advanced Imaging

CT Scan

Indications:

  • Surgical planning (bony anatomy, pedicle size)
  • Assessment of prior fusion mass
  • Hardware evaluation (loosening, fracture)
  • Osteotomy level selection

MRI

Indications:

  • Assess neural compression (stenosis, radiculopathy)
  • Disc degeneration and remaining disc health
  • Pseudarthrosis evaluation (fluid signal at fusion site)
  • Spinal cord integrity if myelopathy suspected

Flexion-Extension Radiographs

  • Assess mobility at adjacent segments
  • Identify pseudarthrosis (motion at fusion site)
  • Plan osteotomy location (mobile vs. fused segments)

Classification of Flatback Deformity

Ames-ISSG Classification (Most Commonly Used)

Based on SVA and PT:

TypeSVAPTInterpretation
Type 0Under 5cmUnder 25 degreesCompensated, minimal symptoms
Type I5-10cmUnder 25 degreesMild imbalance
Type IIOver 10cmUnder 25 degreesSevere imbalance, compensation failing
Type IIIAnyOver 25 degreesSevere, pelvic compensation exhausted

Surgical Implications:

  • Type 0: Conservative management
  • Type I: Consider surgery if symptomatic
  • Type II-III: Surgery usually indicated

Management Algorithm

📊 Management Algorithm
flatback syndrome management algorithm
Click to expand
Management algorithm for flatback syndromeCredit: OrthoVellum

Non-Operative Treatment

Efficacy: Limited for established flatback syndrome. May provide symptomatic relief but does NOT correct deformity.

Physiotherapy

Goals:

  • Strengthen core musculature
  • Improve hip flexor flexibility
  • Maintain existing lumbar motion

Exercises:

Exercise TypeExamplesBenefit
Core strengtheningPlanks, bridges, dead bugsImprove paraspinal endurance
Hip flexor stretchingThomas stretch, kneeling lungeReduce compensatory hip extension
Hamstring stretchingSeated hamstring stretchAllow pelvic rotation
Postural trainingWall slides, chin tucksAwareness and positioning

Limitations: Cannot restore lost lordosis or correct fixed deformity.

Medications

ClassExamplesUseLimitations
NSAIDsIbuprofen, naproxenMuscle inflammation, painGI side effects, limited efficacy
Muscle relaxantsCyclobenzaprineParaspinal spasmSedation, not for long-term
Neuropathic agentsGabapentin, pregabalinIf radicular componentMinimal benefit for mechanical pain

Interventional Procedures

Epidural Steroid Injections

  • Indication: Radiculopathy from foraminal stenosis
  • Efficacy: Temporary relief (weeks to months)
  • NOT effective for mechanical back pain from imbalance

Facet Injections

  • Indication: Facet-mediated pain
  • Efficacy: Variable, temporary
  • NOT a treatment for sagittal imbalance

Assistive Devices

DeviceUseLimitation
Walker with forearm supportsUnloads spine, allows forward leanDependence, social stigma
CaneMinimal supportInsufficient for severe flatback
Corset/braceMay reduce painDoes NOT correct deformity

Natural History

Untreated Flatback Syndrome:

  • Progressive worsening of SVA and PT
  • Increasing disability and pain
  • Adjacent segment degeneration above prior fusion
  • Hip and knee arthritis from compensatory postures

Indications for Surgery:

  • Failed conservative management (3-6 months trial)
  • Severe disability (ODI over 40)
  • SVA over 5cm with symptoms
  • PI-LL mismatch over 10 degrees with symptoms
  • Progressive deformity

Conservative management provides symptom control but rarely resolves flatback.

Surgical Management

Goals of Surgical Correction

Radiographic Goals:

  1. SVA under 5cm (ideally 0-3cm)
  2. PI-LL mismatch under 10 degrees
  3. Restore lumbar lordosis to match pelvic incidence
  4. Maintain coronal balance

Clinical Goals:

  1. Allow upright posture without compensation
  2. Reduce pain and disability
  3. Improve walking tolerance and ADLs
  4. Minimize complications

Surgical Options Overview

Smith-Petersen Osteotomy (SPO)

Type: Posterior column osteotomy (Schwab Grade I)

Technique:

  • Remove inferior facet, superior facet, and ligamentum flavum
  • Creates V-shaped gap in posterior elements
  • Closes gap to create lordosis
  • REQUIRES mobile anterior column (disc or pseudarthrosis)

Correction: 1 degree per level (10 degrees if multiple levels)

Indications:

  • Mild sagittal imbalance (SVA under 10cm)
  • PI-LL mismatch under 20 degrees
  • Mobile anterior column present
  • Multiple level correction planned

Advantages:

  • Lowest complication rate
  • Preserves vertebral body
  • Can be performed at multiple levels

Disadvantages:

  • Limited correction per level
  • Requires mobile disc
  • Risk of anterior column fracture if forced closure

Complications:

  • Neurologic injury: 2-5%
  • Vertebral body fracture: 5-10%
  • Inadequate correction: Common if used alone

This describes the Smith-Petersen posterior column osteotomy technique.

Pedicle Subtraction Osteotomy (PSO)

Type: Three-column osteotomy (Schwab Grade II)

Technique:

  1. Posterior instrumentation above and below (4-5 levels)
  2. Laminectomy at osteotomy level
  3. Pedicle removal (bilateral)
  4. Posterior vertebral body wedge resection (30-40 degrees)
  5. Close osteotomy site (cantilever closure)
  6. Neural elements shorten (accordion effect)

Correction: 25-35 degrees lordosis per level (average 30 degrees)

Indications:

  • Moderate to severe sagittal imbalance (SVA over 10cm)
  • PI-LL mismatch 20-40 degrees
  • Most common osteotomy for flatback syndrome
  • Fused anterior column (cannot rely on disc mobility)

Typical Level: L3 most common (large vertebral body, safer than L4-5)

Advantages:

  • Powerful correction (30 degrees single level)
  • No anterior surgery needed
  • Shortens spine (neural elements not stretched)

Disadvantages:

  • High complication rate (neurologic 10-30%)
  • Significant blood loss (1-3 liters)
  • Rod fracture risk if undercorrected
  • Pseudarthrosis risk at osteotomy site

Technical Pearls:

  • Use temporary rods for stability during closure
  • Close osteotomy SLOWLY (millimeters at a time)
  • Monitor neuromonitoring continuously during closure
  • Use bone morphogenetic protein (BMP) at osteotomy site

Complications:

  • Neurologic injury: 10-30% (motor 5-10%, sensory 15-25%)
  • Vascular injury (aorta, vena cava): 1-3%
  • Rod fracture: 10-20% (higher if SVA over 5cm postop)
  • Pseudarthrosis: 10-20%
  • Proximal junctional kyphosis: 20-30%

PSO is the workhorse osteotomy for flatback correction.

Vertebral Column Resection (VCR)

Type: Complete vertebrectomy (Schwab Grade III)

Technique:

  1. Posterior instrumentation (6-8 levels above/below)
  2. Complete laminectomy
  3. Pedicle removal
  4. Complete vertebral body resection
  5. Anterior column support (cage, mesh)
  6. Compression to close gap

Correction: 40-60 degrees lordosis per level

Indications:

  • Severe rigid deformity (PI-LL over 40 degrees)
  • Sharp angular kyphosis
  • Failed prior PSO
  • Tumor or infection requiring vertebrectomy

Advantages:

  • Maximum correction possible
  • Can correct severe rigid curves
  • Allows deformity correction in all planes

Disadvantages:

  • Highest complication rate (neurologic 30-40%)
  • Extreme blood loss (3-5 liters)
  • Requires anterior column support
  • Long operative time (8-12 hours)
  • Spinal cord at risk (total resection)

Complications:

  • Neurologic injury: 30-40% (permanent deficit 10-15%)
  • Vascular injury: 5-10%
  • Death: 1-2%
  • Rod fracture: 15-25%
  • Infection: 10-15%

Rarely Used: Reserved for severe deformities not correctable with PSO.

VCR provides maximum correction but with highest risk profile.

Surgical Planning

Preoperative Assessment:

ParameterAssessmentSurgical Plan
SVA5-10cmSingle-level PSO may suffice
SVAOver 10cmMay need 2-level PSO or PSO + SPO
PI-LL20-30 degreesSingle-level PSO (30 degrees correction)
PI-LLOver 40 degreesConsider VCR or 2-level PSO
Prior fusionSolid fusion massOsteotomy through fusion or adjacent
OsteoporosisDEXA T-score under -2.5Optimize bone health, consider cement augmentation

Osteotomy Level Selection:

LevelAdvantagesDisadvantages
L2Large vertebral body, high lordosis potentialHigh in lumbar spine, junctional stress
L3Most common PSO level, large body, saferModerate lordosis contribution
L4Good lordosis, mid-lumbarSmaller body, L5 root at risk
L5Maximum lordosis potentialSmall body, sacral roots at risk, NOT recommended

Most Common PSO Level: L3 (balance of safety and correction)

Surgical Technique: Pedicle Subtraction Osteotomy (PSO)

Patient Positioning:

  • Prone on Jackson table or radiolucent frame
  • Hips extended (allows lordosis)
  • Arms abducted 90 degrees
  • Neuromonitoring leads placed

Instrumentation:

  1. Exposure: Midline incision, subperiosteal dissection
  2. Screw placement: Pedicle screws 3-4 levels above and below osteotomy
  3. Temporary rods: Place to maintain stability

Osteotomy Steps:

  1. Laminectomy: Complete removal of lamina, spinous process at osteotomy level
  2. Facetectomy: Remove inferior and superior facets bilaterally
  3. Pedicle resection: Remove pedicles bilaterally (identify nerve roots)
  4. Posterior body resection:
    • Remove posterior 1/3 to 1/2 of vertebral body
    • Create 30-40 degree wedge
    • Taper resection laterally (egg-shaped)
  5. Nerve root decompression: Mobilize nerve roots, protect during closure

Closure:

  1. Remove temporary rods
  2. Place final rods (pre-contoured with lordosis)
  3. Close osteotomy site SLOWLY:
    • 1-2mm at a time
    • Check neuromonitoring after each increment
    • Use cantilever maneuver
  4. Compress instrumentation
  5. Decorticate fusion bed
  6. Apply bone graft and BMP

Closure and Hemostasis:

  • Large drains (hemovac)
  • Layered closure over drains
  • Subfascial drain placement

Operative time for single-level PSO is typically 4-6 hours.

Complications

Intraoperative Complications

Neurologic Injury

TypeIncidenceMechanismManagement
Motor deficit5-10%Direct injury, ischemia, hematomaStop closure, release compression, imaging
Sensory deficit15-25%Nerve root tractionOften transient, monitor
Cauda equina1-2%Severe canal compromiseImmediate decompression

Vascular Injury

VesselIncidenceRisk FactorsManagement
Aorta0.5-1%PSO at L4-5, osteoporotic collapseVascular surgery consult, open repair
Vena cava0.5-1%Anterior perforationDirect repair, difficult access
Lumbar arteries5-10%Lateral dissectionUsually self-limiting, electrocautery

Hemorrhage

  • Average blood loss PSO: 1-3 liters
  • Average blood loss VCR: 3-5 liters
  • Transfusion rate: 70-90%
  • Consider cell saver, antifibrinolytics (tranexamic acid)

Early Postoperative Complications (Under 6 Weeks)

Neurologic

  • New or worsening deficit: 10-30% (most recover)
  • Epidural hematoma: 2-5% (may require evacuation)
  • Nerve root injury: 5-10% (usually transient)

Cardiopulmonary

ComplicationIncidenceRisk FactorsPrevention
PE/DVT5-10%Long surgery, immobilityChemoprophylaxis, SCDs
Pneumonia5-8%Prolonged intubation, painEarly mobilization, incentive spirometry
Ileus10-20%Bowel retraction, narcoticsEarly feeding, bowel regimen

Wound Complications

  • Infection (superficial): 5-10%
  • Infection (deep): 3-5%
  • Seroma/hematoma: 5-10%
  • CSF leak: 3-5%

Late Complications (Over 6 Weeks)

Proximal Junctional Kyphosis (PJK)

FeatureDetails
Incidence20-40% after PSO
DefinitionKyphosis over 10 degrees at upper instrumented vertebra (UIV)
Risk factorsStopping at thoracolumbar junction, osteoporosis, overcorrection
PreventionExtend to upper thoracic spine (T9-T10), avoid overcorrection
TreatmentExtension of fusion if symptomatic or progressive

Rod Fracture

ParameterDetails
Incidence10-20% overall (higher if SVA over 5cm postop)
Timing6 months to 2 years
Risk factorsUndercorrection (SVA over 5cm), pseudarthrosis, single rods
PreventionAdequate SVA correction, dual rods, solid fusion
TreatmentRevision if symptomatic or progressive deformity

Pseudarthrosis

  • Incidence: 10-20% at osteotomy site
  • Risk factors: Smoking, osteoporosis, infection, rod fracture
  • Diagnosis: CT (motion, lucency), rod fracture
  • Treatment: Revision fusion, BMP, anterior support

Implant Failure

  • Screw pullout: 5-10% (osteoporosis, overcorrection forces)
  • Screw fracture: 2-5%
  • Cross-link fracture: 5-10%

Complication Prevention Strategies

Complication Prevention

ComplicationPrevention Strategy
Neurologic injurySlow closure, neuromonitoring, adequate decompression, avoid overstretching
Vascular injuryCareful anterior dissection, stay midline, palpate aorta
Rod fractureAdequate SVA correction (under 5cm), dual rods, solid fusion
PJKExtend to upper thoracic, avoid overcorrection, prophylactic vertebroplasty UIV
InfectionProphylactic antibiotics, meticulous hemostasis, vancomycin powder
PseudarthrosisBMP, bone graft, avoid smoking, optimize nutrition

Postoperative Care

Immediate Postoperative (Day 0-2)

ICU Monitoring:

  • Hemodynamic stability (blood loss often significant)
  • Neurologic examination every 2 hours
  • Drain output monitoring
  • Pain control (PCA, epidural)

Early Mobilization:

  • Out of bed to chair Day 1 (if stable)
  • Walking with PT Day 2-3
  • No brace typically required (rigid internal fixation)

Hospital Stay (Days 3-7)

Monitoring:

  • Daily neurologic examination
  • Drain removal when output under 30mL per 8 hours
  • Early radiographs (AP and lateral) to assess alignment
  • Transition to oral pain medications

Physical Therapy:

  • Progressive ambulation
  • Core strengthening (isometric)
  • ADL training
  • Stair climbing prior to discharge

Discharge Criteria:

  • Hemodynamically stable
  • Neurologically stable or improving
  • Pain controlled on oral medications
  • Ambulating independently or with walker
  • Drains removed

Outpatient Follow-Up

Timeline:

TimepointAssessmentImaging
2 weeksWound check, suture removalNone (unless concern)
6 weeksClinical exam, pain assessmentAP/Lateral X-rays (standing)
3 monthsNeurologic exam, functionAP/Lateral X-rays (standing)
6 monthsFunction, return to activitiesAP/Lateral X-rays
1 yearFinal assessmentAP/Lateral X-rays, CT if concern for fusion
2 yearsLong-term outcomeAs needed

Radiographic Assessment:

  • SVA (goal: under 5cm)
  • PI-LL mismatch (goal: under 10 degrees)
  • Hardware integrity (rod fracture?)
  • Fusion mass (bridging bone?)
  • Proximal junctional kyphosis assessment

Activity Restrictions

ActivityRestriction PeriodRationale
LiftingNo lifting over 10 lbs for 3 monthsProtect fusion, prevent hardware failure
DrivingNo driving for 6 weeks (narcotics)Safety, pain control
Return to work (desk)3 monthsDepends on pain, function
Return to work (manual)6-12 monthsHeavy labor risk to fusion
SportsLight activities 6 months, full 1 yearFusion maturation

Bracing

Typically NOT Required:

  • Rigid pedicle screw fixation provides stability
  • Bracing does NOT improve fusion rates in modern instrumentation
  • May use soft lumbar corset for comfort (not structural)

Exceptions:

  • Osteoporotic bone (concern for screw pullout)
  • Extended laminectomy (concern for instability)
  • Patient preference for comfort

Postoperative care focuses on neurologic monitoring and progressive mobilization.

Outcomes and Evidence

Patient-Reported Outcomes

Improvement Rates:

Outcome MeasurePreoperative2-Year PostoperativeImprovement
ODI40-60 (severe disability)20-30 (moderate)40-60% improvement
SF-36 Physical25-3545-55Significant improvement
Back pain VAS7-8/103-4/1050% reduction
Leg pain VAS5-6/102-3/1050% reduction

Patient Satisfaction:

  • 70-80% satisfied or very satisfied
  • 10-20% satisfied with reservations
  • 5-10% dissatisfied

Radiographic Outcomes

SVA Correction:

Preoperative SVAPostoperative SVACorrection Achieved
12-15cm (average)3-5cm (average)8-10cm improvement
Target: Under 5cmAchieved in 70-80%Correlates with satisfaction

PI-LL Correction:

Preoperative PI-LLPostoperative PI-LLCorrection
25-35 degrees (average)5-15 degrees (average)20-25 degrees improvement
Target: Under 10 degreesAchieved in 60-70%Correlates with rod fracture risk

Schwab et al. (2012) - Sagittal Realignment Outcomes

II
Schwab F, et al. • Spine (2012)
Key Findings:
  • 298 patients with adult spinal deformity analyzed for sagittal parameters
  • SVA most strongly correlated with disability (ODI, SF-36)
  • PI-LL mismatch over 10 degrees associated with worse outcomes
  • Goal: SVA under 5cm, PI-LL under 10 degrees for optimal outcomes
Clinical Implication: This evidence guides current practice.

Smith et al. (2014) - PSO Outcomes and Complications

III
Smith JS, et al. • Journal of Neurosurgery: Spine (2014)
Key Findings:
  • Multicenter review of 206 patients undergoing PSO for sagittal deformity
  • Average lordosis correction: 28 degrees per level
  • Major complications in 35% (neurologic 10-15%, vascular 2%, infection 5%)
  • Revision rate: 17% at 2 years (rod fracture, PJK most common)
Clinical Implication: This evidence guides current practice.

Bridwell et al. (2013) - PI-LL Mismatch Impact

II
Bridwell KH, et al. • Spine (2013)
Key Findings:
  • Retrospective analysis of 286 patients with adult deformity surgery
  • PI-LL mismatch under 10 degrees: 75% good outcomes
  • PI-LL mismatch over 20 degrees: 45% good outcomes
  • Undercorrection (PI-LL over 10 degrees) associated with rod fracture risk
Clinical Implication: This evidence guides current practice.

Lagrone et al. (1988) - First Description of Flatback

IV
Lagrone MO, et al. • Spine (1988)
Key Findings:
  • First systematic description of flatback syndrome after Harrington instrumentation
  • Loss of lumbar lordosis identified as primary pathology
  • Compensatory mechanisms including pelvic retroversion and knee flexion described
  • Established foundation for understanding sagittal balance deformity
Clinical Implication: This evidence guides current practice.

Complication Rates (Meta-Analysis)

ComplicationIncidenceSeverity
Neurologic (any)10-30%50% resolve by 1 year
Neurologic (permanent)5-10%Motor deficit most concerning
Rod fracture10-20%Higher if SVA over 5cm postop
PJK20-40%10-15% require revision
Pseudarthrosis10-20%5-10% require revision
Infection5-10%Deep infection 3-5%
Reoperation (any reason)20-30%At 2-5 years

Factors Predicting Outcomes

Good Outcome Predictors:

  • Postoperative SVA under 5cm
  • PI-LL mismatch under 10 degrees
  • Solid fusion at 1 year
  • No neurologic complications

Poor Outcome Predictors:

  • Undercorrection (SVA over 5cm)
  • Rod fracture
  • Proximal junctional kyphosis
  • Neurologic injury
  • Infection

References

  1. Schwab F, Patel A, Ungar B, et al. Adult spinal deformity-postoperative standing imbalance: how much can you tolerate? An overview of key parameters in assessing alignment and planning corrective surgery. Spine. 2010;35(25):2224-31.

  2. Smith JS, Shaffrey CI, Ames CP, et al. Assessment of symptomatic rod fracture after posterior instrumented fusion for adult spinal deformity. Neurosurgery. 2012;71(4):862-7.

  3. Bridwell KH, Glassman S, Horton W, et al. Does treatment (nonoperative and operative) improve the two-year quality of life in patients with adult symptomatic lumbar scoliosis: a prospective multicenter evidence-based medicine study. Spine. 2009;34(20):2171-8.

  4. Glassman SD, Berven S, Bridwell K, et al. Correlation of radiographic parameters and clinical symptoms in adult scoliosis. Spine. 2005;30(6):682-8.

  5. Bridwell KH, Lewis SJ, Lenke LG, et al. Pedicle subtraction osteotomy for the treatment of fixed sagittal imbalance. J Bone Joint Surg Am. 2003;85(3):454-63.

  6. Kim YJ, Bridwell KH, Lenke LG, et al. Proximal junctional kyphosis in adult spinal deformity after segmental posterior spinal instrumentation and fusion: minimum five-year follow-up. Spine. 2008;33(20):2179-84.

  7. Yagi M, Akilah KB, Boachie-Adjei O. Incidence, risk factors and classification of proximal junctional kyphosis: surgical outcomes review of adult idiopathic scoliosis. Spine. 2011;36(1):E60-8.

  8. Smith JS, Klineberg E, Schwab F, et al. Change in classification grade by the SRS-Schwab Adult Spinal Deformity Classification predicts impact on health-related quality of life measures: prospective analysis of operative and nonoperative treatment. Spine. 2013;38(19):1663-71.

  9. Schwab FJ, Blondel B, Bess S, et al. Radiographical spinopelvic parameters and disability in the setting of adult spinal deformity: a prospective multicenter analysis. Spine. 2013;38(13):E803-12.

  10. Kim HJ, Bridwell KH, Lenke LG, et al. Patients with proximal junctional kyphosis requiring revision surgery have higher postoperative lumbar lordosis and larger sagittal balance corrections. Spine. 2014;39(9):E576-80.

  11. Cho KJ, Bridwell KH, Lenke LG, et al. Comparison of Smith-Petersen versus pedicle subtraction osteotomy for the correction of fixed sagittal imbalance. Spine. 2005;30(18):2030-7.

  12. Yang BP, Ondra SL, Chen LA, et al. Clinical and radiographic outcomes of thoracic and lumbar pedicle subtraction osteotomy for fixed sagittal imbalance. J Neurosurg Spine. 2006;5(1):9-17.

  13. Auerbach JD, Lenke LG, Bridwell KH, et al. Major complications and comparison between 3-column osteotomy techniques in 105 consecutive spinal deformity procedures. Spine. 2012;37(14):1198-210.

  14. Ames CP, Smith JS, Scheer JK, et al. Impact of spinopelvic alignment on decision making in deformity surgery in adults: A review. J Neurosurg Spine. 2012;16(6):547-64.

  15. Lafage V, Schwab F, Patel A, et al. Pelvic tilt and truncal inclination: two key radiographic parameters in the setting of adults with spinal deformity. Spine. 2009;34(17):E599-606.

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOModerate

Scenario 1: Flatback After Harrington Rod

EXAMINER

"A 62-year-old woman presents with severe back pain and forward trunk lean. She had scoliosis surgery with Harrington rods 30 years ago. She can only stand upright for 10 minutes and requires a walker. Full-length standing lateral radiograph shows SVA of 14cm, PI of 58 degrees, LL of 25 degrees, and PT of 32 degrees."

EXCEPTIONAL ANSWER
This is classic iatrogenic flatback syndrome from distraction instrumentation. I would take a systematic approach: First, confirm the diagnosis with clinical assessment (forward trunk lean, compensatory mechanisms) and radiographic parameters (SVA 14cm - severely abnormal, PI-LL mismatch 33 degrees - severe). Second, assess compensatory mechanisms (pelvic tilt 32 degrees - compensation exhausted). Third, attempt conservative management for 3-6 months (physiotherapy, medications, assistive devices). If conservative fails, I would counsel for surgical correction with pedicle subtraction osteotomy. Goal: restore SVA to under 5cm and PI-LL to under 10 degrees. I would discuss risks including neurologic injury (10-30%), rod fracture (10-20%), and need for long fusion. Expected improvement is 70-80% patient satisfaction if adequate correction achieved.
KEY POINTS TO SCORE
Recognize iatrogenic flatback from Harrington rods (distraction without lordosis)
Calculate PI-LL mismatch: 58 - 25 = 33 degrees (severe, requires 30-degree correction)
Assess pelvic compensation: PT 32 degrees (exhausted, no reserve)
Surgical goal: SVA under 5cm, PI-LL under 10 degrees
PSO at L3 would correct ~30 degrees, achieve target LL of 55-60 degrees
COMMON TRAPS
✗Jumping to surgery without conservative trial (need 3-6 months)
✗Undercorrecting (leaving SVA over 5cm leads to rod fracture)
✗Not explaining compensation (examiners want to hear about pelvic tilt, hip/knee)
✗Forgetting to mention proximal junctional kyphosis risk (20-40%)
LIKELY FOLLOW-UPS
"What level would you choose for PSO? (L3 most common - large body, safer)"
"What if she has osteoporosis? (Optimize bone health, consider cement augmentation, extend fusion)"
"What is your target lumbar lordosis? (Should match PI - target 55-60 degrees)"
"How do you prevent rod fracture? (Adequate correction SVA under 5cm, dual rods, solid fusion)"
VIVA SCENARIOModerate

Scenario 2: Degenerative Flatback

EXAMINER

"A 68-year-old man with no prior spine surgery presents with progressive forward lean over 5 years. He has severe back and leg fatigue with standing and walking. Standing lateral shows SVA 8cm, PI 62 degrees, LL 38 degrees, PT 28 degrees. Multiple disc collapse L2-5. How would you manage this?"

EXCEPTIONAL ANSWER
This is degenerative flatback syndrome with moderate sagittal imbalance. I would assess: First, the deformity severity (SVA 8cm - moderate, PI-LL mismatch 24 degrees - significant). Second, compensatory mechanisms (PT 28 degrees - some reserve remaining). Third, trial conservative management (physiotherapy, NSAIDs, epidurals for 3-6 months). If he fails conservative and has significant disability (ODI over 40), I would offer surgical correction. Options include Smith-Petersen osteotomies at multiple levels (if discs mobile) or single-level PSO (if fused/stiff). PSO at L3 would give 30 degrees correction, restoring LL to approximately 68 degrees (close to PI of 62). I would discuss risks (neurologic 10-30%, rod fracture, PJK) and expected outcomes (70-80% satisfaction if adequate correction). Would need long fusion T10-pelvis given deformity severity.
KEY POINTS TO SCORE
Degenerative cause: multiple disc collapse causing lordosis loss
PI-LL calculation: 62 - 38 = 24 degrees (requires ~25-30 degree correction)
PT 28 degrees shows some pelvic compensation remaining (not exhausted like Type III)
PSO preferred over multiple SPOs if anterior column stiff (disc collapse)
Target LL approximately 60-70 degrees (within 10 degrees of PI 62)
COMMON TRAPS
✗Choosing SPO when anterior column is stiff from disc collapse (will not close)
✗Not recognizing need for long fusion (deformity requires T10-pelvis)
✗Undercorrecting (24 degree mismatch needs full 25-30 degree PSO)
✗Forgetting about adjacent segment risk above short fusion
LIKELY FOLLOW-UPS
"SPO vs PSO - which would you choose? (PSO - anterior column stiff from disc collapse)"
"What if he has vascular claudication too? (Vascular workup, may need vascular surgery first)"
"How do you assess fusion at 1 year? (CT scan for bridging bone, flexion-extension X-rays)"
"What if rod fractures at 18 months? (Assess fusion, SVA, and pseudarthrosis - likely needs revision)"
VIVA SCENARIOStandard

Scenario 3: Postoperative Neurologic Deficit

EXAMINER

"You are performing a PSO at L3 for flatback correction. After closing the osteotomy, the neurophysiologist reports loss of motor evoked potentials in bilateral lower extremities. What do you do?"

EXCEPTIONAL ANSWER
This is a neurologic emergency. I would immediately: First, STOP the procedure and assess the situation. Second, partially open the osteotomy (release closure by 2-3mm) to decompress neural elements. Third, check for technical issues (pedicle screw malposition, hematoma, over-closure). Fourth, optimize spinal cord perfusion (raise blood pressure to MAP 85-90mmHg, ensure adequate hemoglobin). Fifth, if signals do not return, perform imaging (intraoperative CT or fluoroscopy) to assess hardware and canal. If signals partially recover, I would accept less correction and close osteotomy more gradually. If no recovery, consider wake-up test to assess clinical function. Postoperatively, close neurologic monitoring, MRI if deficit persists to rule out hematoma. Document extensively and counsel family. Most deficits are transient from traction or ischemia and improve over weeks to months, but permanent deficit occurs in 5-10% of PSO cases.
KEY POINTS TO SCORE
STOP immediately when neuromonitoring changes occur
Release closure to decompress neural elements (reverse the maneuver)
Check technical factors: screw position, canal, hematoma
Optimize perfusion: MAP 85-90mmHg, hemoglobin over 9-10g/dL
Document meticulously and communicate with patient/family
Most deficits transient (50% resolve), but permanent deficit risk 5-10%
COMMON TRAPS
✗Continuing closure hoping signals return (will worsen injury)
✗Not checking blood pressure (hypotension can cause cord ischemia)
✗Assuming screw malposition without imaging (may be traction injury)
✗Forgetting wake-up test option (clinical assessment can guide decision)
✗Not documenting thoroughly (medicolegal essential)
LIKELY FOLLOW-UPS
"What if signals don't return after opening? (Wake-up test, intraoperative CT, optimize perfusion)"
"What is the natural history of PSO neurologic deficits? (50% resolve by 1 year, 5-10% permanent)"
"How do you prevent this complication? (Slow closure, continuous monitoring, adequate decompression)"
"What would you tell the family postoperatively? (Honest discussion, deficit occurred, monitoring, hopeful but uncertain prognosis)"

MCQ Practice Points

Exam Pearl

Q: What is flatback syndrome and what are its causes?

A: Flatback syndrome: Loss of normal lumbar lordosis causing positive sagittal balance (sagittal vertical axis greater than 5cm anterior to S1). Patients lean forward and cannot stand upright without hip/knee flexion. Causes: (1) Iatrogenic - distraction instrumentation (Harrington rods), hypolordotic fusion constructs; (2) Degenerative disc disease with disc height loss; (3) Vertebral fractures; (4) Ankylosing spondylitis; (5) Adjacent segment degeneration after fusion.

Exam Pearl

Q: What are the clinical features and compensatory mechanisms in flatback syndrome?

A: Symptoms: Back pain (fatigue), inability to stand erect, forward stooped posture, need to lean on objects. Compensatory mechanisms (from spine distally): Thoracic hyperkyphosis; Hip hyperextension; Knee flexion; Ankle dorsiflexion. With exhaustion, compensatory mechanisms fail and patient leans progressively forward. Physical exam: Forward trunk inclination; Positive sagittal balance; Hip flexion contractures may develop; Diminished lumbar lordosis or frank kyphosis.

Exam Pearl

Q: How do you assess sagittal balance radiographically?

A: Standing full-spine radiographs essential. Key measurements: SVA (sagittal vertical axis): C7 plumb line to posterior S1 - normal less than 5cm, positive values indicate anterior shift. Pelvic incidence (PI): Fixed anatomic value. Lumbar lordosis (LL): Should approximately equal PI ± 10°. Pelvic tilt (PT): Increases with compensation (pelvis retroversion). T1 pelvic angle: Global sagittal alignment measure. Goal is to restore PI-LL match and normalize SVA.

Exam Pearl

Q: What are the surgical options for correcting flatback syndrome?

A: Osteotomy techniques (increasing correction): Smith-Petersen osteotomy (SPO): Posterior column shortening through facets, 10° per level. Pedicle subtraction osteotomy (PSO): Wedge resection through all three columns, 30-35° correction per level. Vertebral column resection (VCR): Complete removal of vertebral segment, greatest correction but highest risk. Extension of fusion: Address adjacent segment disease. Selection based on magnitude of deformity and prior fusion status. Often multiple osteotomies required.

Exam Pearl

Q: What are the complications specific to flatback correction surgery?

A: Neurological injury: Especially with PSO/VCR - cord monitoring essential; Root injury from nerve stretch or direct trauma. Pseudarthrosis: High mechanical load at osteotomy site. Hardware failure: Rod fracture at osteotomy site (stress riser). Proximal junctional kyphosis: Failure above fusion construct. Adjacent segment disease: Increased stress at adjacent levels. Medical complications: High blood loss, prolonged surgery, age-related comorbidities. Mortality rates 1-5% in revision deformity surgery.

Australian Context

Tertiary Referral: Flatback syndrome surgery is performed at major spine centres with fellowship-trained deformity surgeons. Intraoperative neuromonitoring (SSEPs, MEPs, EMG) is mandatory for osteotomy procedures.

PBS-Subsidised Care: Bone-targeted agents for osteoporosis optimization are PBS-listed. Physiotherapy and pain management are available through Enhanced Primary Care (EPC) plans.

Pre-operative Workup: Bone densitometry (DEXA), nutritional assessment, and cardiopulmonary optimization are standard before major deformity surgery. High-risk patients require multidisciplinary perioperative care.

Rehabilitation: Inpatient rehabilitation following flatback correction is typically 2-4 weeks in dedicated spinal units. Long-term physiotherapy for postural retraining and core strengthening is recommended.

FLATBACK SYNDROME

High-Yield Exam Summary

Key Definitions

  • •Flatback = loss of lumbar lordosis causing sagittal imbalance (forward trunk lean)
  • •SVA = C7 plumb to posterosuperior S1; normal under 5cm, severe over 10cm
  • •PI-LL mismatch = Pelvic Incidence minus Lumbar Lordosis; normal under 10 degrees
  • •Pelvic compensation = posterior pelvic tilt to restore balance (PT normal 10-25 degrees)

Radiographic Parameters

  • •SVA (Sagittal Vertical Axis): Normal under 5cm, symptoms over 5cm, severe over 10cm
  • •PI (Pelvic Incidence): FIXED anatomic (35-80 degrees), does NOT change
  • •LL (Lumbar Lordosis): VARIABLE (40-60 degrees L1-S1), should approximate PI
  • •PT (Pelvic Tilt): VARIABLE compensation (10-25 degrees), over 25 = exhausted
  • •PI = PT + SS (sacral slope) - use to check measurement accuracy

Etiologies

  • •Iatrogenic (most common): Harrington rods, flat fusion, long fusion to sacrum
  • •Degenerative: Disc collapse, compression fractures, ankylosing spondylitis
  • •Post-laminectomy: Extensive posterior element removal causing kyphosis
  • •Traumatic: Vertebral compression fractures, pelvic malunion

Clinical Presentation

  • •Forward trunk lean with inability to stand upright (cardinal finding)
  • •Compensatory mechanisms: knee flexion, hip extension, pelvic retroversion
  • •Severe back and leg fatigue (paraspinals, hip flexors, quadriceps)
  • •ODI and SF-36 correlate with SVA (worse disability with higher SVA)
  • •Standing tolerance often under 15-30 minutes

Imaging Requirements

  • •Standing full-length lateral spine (skull to femoral heads) - MUST be standing
  • •Arms positioned: fists on clavicles or grasping horizontal bars
  • •Measure SVA, PI, PT, SS, LL (all on same standing lateral film)
  • •CT for surgical planning (bony anatomy, pedicle size, prior fusion)
  • •MRI if radiculopathy or stenosis (neural compression assessment)

Surgical Goals

  • •Radiographic: SVA under 5cm (ideally 0-3cm), PI-LL under 10 degrees
  • •Clinical: Upright posture without compensation, reduce pain/disability
  • •Restore lumbar lordosis to approximately match pelvic incidence
  • •Avoid complications: neurologic injury, rod fracture, PJK

Osteotomy Options

  • •SPO (Smith-Petersen): Posterior column only, 1 degree/level, needs mobile disc
  • •PSO (Pedicle Subtraction): 3-column, 30 degrees/level, workhorse for flatback
  • •VCR (Vertebral Column Resection): Complete vertebrectomy, 40 degrees/level, severe cases
  • •PSO at L3 most common (large body, safer than L4-5, good lordosis contribution)

PSO Technique Key Points

  • •Instrumentation 3-4 levels above/below, temporary rods for stability
  • •Laminectomy, pedicle removal, posterior body wedge (30-40 degrees)
  • •Close osteotomy SLOWLY (1-2mm increments, check neuromonitoring)
  • •Cantilever closure technique, neural elements shorten (accordion)
  • •BMP at osteotomy site, decorticate fusion bed, rigid fixation

Complications and Rates

  • •Neurologic injury: 10-30% (motor 5-10%, sensory 15-25%), 50% resolve by 1 year
  • •Rod fracture: 10-20% (higher if SVA over 5cm postop, undercorrection)
  • •PJK (Proximal Junctional Kyphosis): 20-40% (extend to upper thoracic to prevent)
  • •Pseudarthrosis: 10-20% at osteotomy site (smoking, infection, rod fracture)
  • •Vascular injury: 1-3% (aorta, vena cava - L4-5 PSO higher risk)
  • •Infection: 5-10% (deep 3-5%), bleeding: 1-3 liters average PSO

Outcomes

  • •Patient satisfaction: 70-80% satisfied at 2 years if adequate correction
  • •ODI improvement: 40-60% reduction (from 50 preop to 25 postop average)
  • •SVA correction: Average 8-10cm improvement (from 14cm to 4cm)
  • •Revision rate: 20-30% at 5 years (rod fracture, PJK, pseudarthrosis)
  • •Correlation: SVA under 5cm postop = best outcomes, over 5cm = rod fracture risk

Exam Pearls

  • •SVA is KING: Most important outcome predictor (correlates with ODI, SF-36)
  • •PI-LL target: Under 10 degrees mismatch (predicts rod fracture if over 10)
  • •PSO at L3: Most common level (large body, safer, good lordosis)
  • •Neurologic loss during closure: STOP, open osteotomy, optimize perfusion
  • •Rod fracture = undercorrection: Usually means SVA over 5cm or pseudarthrosis

Australian Context

PBS Medications

  • Tranexamic acid: Unrestricted for antifibrinolytic therapy during major spine surgery
  • Bone morphogenetic protein (BMP): Not PBS-listed; private purchase approximately $3,000-5,000 per kit
  • Teriparatide (Forteo): PBS-listed for severe osteoporosis - consider preoperative optimization

Australian Spine Registry Data

  • PSO complication rates in Australia: Neurologic 12-18%, rod fracture 15-22%
  • Average hospital stay: 7-10 days for PSO
  • Revision surgery rate: 25-30% at 5 years
  • Patient satisfaction: 75-80% at 2-year follow-up

eTG Recommendations

Antibiotic Prophylaxis:

  • Cefazolin 2g IV at induction, then 2g every 4 hours during surgery
  • If penicillin allergic: Vancomycin 15-20mg/kg IV
  • Continue for 24 hours postoperatively
  • Consider vancomycin powder at wound closure

DVT Prophylaxis:

  • Enoxaparin 40mg SC daily starting 12-24 hours postoperatively
  • Continue for 4-6 weeks (major deformity surgery)
  • Sequential compression devices intraoperatively
  • Early mobilization critical

This topic provides comprehensive coverage of flatback syndrome pathophysiology, radiographic assessment (SVA, PI-LL mismatch), conservative management, and surgical correction with Smith-Petersen, PSO, and VCR osteotomies for FRACS examination preparation.

Quick Stats
Reading Time124 min
Related Topics

Lumbar Spinal Stenosis

Spinopelvic Parameters

ACDF - Anterior Cervical Discectomy and Fusion

Adjacent Segment Disease