ADULT ACQUIRED FLATFOOT DEFORMITY
Progressive Collapse | PTT Dysfunction | Staged Reconstruction
Johnson and Strom Classification
Critical Must-Knows
- Johnson and Strom classification - guides surgical reconstruction strategy
- Rigid vs flexible - determines need for osteotomy vs arthrodesis
- Too many toes sign - pathognomonic for hindfoot valgus on clinical exam
- Single heel rise test - inability indicates PTT incompetence
- Stage II divide - IIA (flexible hindfoot) vs IIB (forefoot driven, fixed forefoot varus)
Examiner's Pearls
- "Stage IIB requires cotton test and first ray plantar flexion osteotomy
- "FDL is preferred tendon transfer over FHL (better excursion, less donor morbidity)
- "Lateral column lengthening (Evans) corrects forefoot abduction
- "Stage III requires subtalar or triple arthrodesis for rigid deformity
Clinical Imaging
Imaging Gallery



Critical Adult Flatfoot Exam Points
Classification Is Key
Johnson and Strom staging drives all treatment decisions. Stage I = conservative. Stage II = reconstruction (osteotomy + transfer). Stage III = arthrodesis. Stage IV = ankle fusion. Know the flexibility test!
Deformity Components
Three-plane deformity: Hindfoot valgus (coronal), arch collapse (sagittal), forefoot abduction (axial). Examiners ask: "Describe the deformity." Answer systematically in all three planes.
Clinical Tests
Too many toes sign (view from behind) and single heel rise test (PTT function). Examiners will show photos. Also test: flexibility with hindfoot valgus stress and Silfverskiöld test for gastrocnemius contracture.
Surgical Decision Tree
Flexible Stage II = calcaneal osteotomy (medializing or Koutsogiannis) plus FDL transfer plus possible lateral column lengthening. Stage IIB adds cotton test and medial cuneiform osteotomy. Rigid Stage III = triple arthrodesis. Know when to do what!
Quick Decision Guide: AAFD Management
| Stage | Deformity | Treatment | Key Pearl |
|---|---|---|---|
| Stage I | PTT tenosynovitis, no deformity | Immobilization (CAM boot 6-12 weeks), NSAIDs, orthotics | Exam: medial ankle pain, no too many toes sign |
| Stage IIA | Flexible hindfoot valgus, passively correctable | Medializing calcaneal osteotomy + FDL transfer ± LCL | Cotton test negative, hindfoot corrects with stress |
| Stage IIB | Flexible hindfoot + fixed forefoot varus | As IIA PLUS medial cuneiform plantar flexion osteotomy | Cotton test positive, forefoot drives hindfoot valgus |
| Stage III | Rigid/fixed deformity, subtalar arthritis | Subtalar arthrodesis or triple arthrodesis | Hindfoot does NOT correct passively, talonavicular arthritis |
| Stage IV | Ankle valgus tilt (deltoid insufficiency) | Tibiotalocalcaneal arthrodesis (TTC fusion) | Lateral ankle mortise widening on AP radiograph |
MAINPosterior Tibial Tendon Anatomy
Memory Hook:PTT is the MAIN support of the medial arch - lose it and the arch collapses!
TFRFJohnson and Strom Classification
Memory Hook:AAFD progression: Tendon inflames → Flexibility lost in hindfoot → Rigidity sets in → Further spread to ankle!
COLTSurgical Reconstruction Components (Stage II)
Memory Hook:Fixing Stage II AAFD is like training a young COLT - need all components working together for stable gait!
UNIONComplications to Discuss in Viva
Memory Hook:Aim for UNION of all corrected joints - but watch for complications that prevent perfect healing!
Overview and Epidemiology
Clinical Significance
Adult acquired flatfoot deformity (AAFD) represents a progressive collapse of the medial longitudinal arch due to posterior tibial tendon (PTT) dysfunction. It is the most common cause of acquired flatfoot in adults and represents a spectrum from isolated tenosynovitis to severe fixed deformity with ankle involvement. Understanding the staged progression is critical for treatment selection.
Demographics and Risk Factors
- Age: Peak 40-60 years
- Gender: Female predominance (3:1 ratio)
- Obesity: Major risk factor (increased mechanical load)
- Diabetes: Associated with tendon degeneration
- Hypertension: Vascular compromise to tendon
- Previous trauma: Medial ankle injuries
- Inflammatory arthropathy: RA, seronegative arthritis
- Steroid use: Tendon weakening
Natural History and Impact
- Progressive deformity: Without treatment, stage I → stage IV over years
- Functional limitation: Pain with prolonged walking/standing
- Gait alteration: Compensatory external rotation, antalgic gait
- Quality of life: Significant impact on mobility and footwear
- Adjacent joint arthritis: Talonavicular, subtalar, ankle involvement
- Bilateral progression: 20% develop contralateral AAFD
Pathophysiology and Mechanisms
PTT Is the PRIMARY Dynamic Arch Stabilizer
The posterior tibial tendon provides over 50% of the dynamic support to the medial longitudinal arch during stance phase. Loss of PTT function results in progressive arch collapse, hindfoot valgus, and forefoot abduction - the classic flatfoot deformity. Unlike static ligamentous stabilizers (spring ligament, plantar fascia), the PTT actively resists arch collapse during gait, making it irreplaceable.
Posterior Tibial Tendon Course and Function
| Anatomical Component | Clinical Significance | Exam Relevance |
|---|---|---|
| Origin: Posterior tibia, fibula, interosseous membrane | Strongest invertor of foot (tibialis posterior muscle) | Loss causes unopposed eversion (peroneus brevis dominates) |
| Course: Posterior to medial malleolus in fibro-osseous tunnel | Hypovasular zone 2-4cm proximal to navicular insertion | Site of degenerative tendinopathy and eventual rupture |
| Primary insertion: Navicular tuberosity | Main arch keystone stabilizer | Detachment here causes immediate arch collapse |
| Secondary insertions: All 3 cuneiforms, MT 2-4 bases | Provides midfoot stability and transverse arch support | Multiple slips allow distributed force transfer |
Static Stabilizers (Spring Ligament Complex)
The spring ligament (plantar calcaneonavicular ligament) is the primary static stabilizer of the medial arch. It consists of two components:
- Superomedial calcaneonavicular ligament: Main load-bearing structure, supports talar head
- Inferior calcaneonavicular ligament: Reinforces plantar aspect
In AAFD, the spring ligament undergoes attritional failure as the PTT loses function, leading to progressive talar head uncovering and arch collapse.
Three-Plane Deformity Components
Coronal Plane
Hindfoot valgus
- Subtalar joint eversion
- Lateral talar shift
- Posterior facet uncovering
- "Too many toes" sign
Sagittal Plane
Arch collapse (planus)
- Talus plantar flexion
- Navicular sag
- Increased talo-first metatarsal angle
- Spring ligament attenuation
Axial Plane
Forefoot abduction
- Talonavicular uncovering
- Lateral border of forefoot
- "Too many toes" sign
- Compensatory tibial external rotation
Classification Systems
Johnson and Strom Classification (Most Widely Used)
| Stage | Clinical Findings | Radiographic Features | Treatment |
|---|---|---|---|
| Stage I | PTT pain, swelling medial ankle. Normal alignment. Single heel rise positive. No too many toes sign. | Normal alignment. No arch collapse. No hindfoot valgus. | Conservative: CAM boot 6-12 weeks, UCBL orthotic, NSAIDs, PT |
| Stage IIA | Hindfoot valgus flexible. Arch collapses weight-bearing. Single heel rise negative. Too many toes positive. Cotton test negative. | Hindfoot valgus (AP: talar head uncovering). Lateral: sag at talonavicular, increased talo-1st MT angle. | Medializing calcaneal osteotomy + FDL transfer ± lateral column lengthening |
| Stage IIB | As IIA PLUS fixed forefoot varus (does not correct). Cotton test positive (forefoot drives hindfoot valgus). | As IIA PLUS forefoot varus visible on lateral radiograph with hindfoot corrected. | As IIA PLUS medial cuneiform plantar flexion osteotomy (Cotton osteotomy) |
| Stage III | Rigid hindfoot valgus (does NOT correct passively). Subtalar stiffness. Talonavicular pain/arthritis. | Subtalar arthritis. Talonavicular arthritis. Fixed hindfoot valgus. No passive correction on stress views. | Subtalar arthrodesis or triple arthrodesis (± tendon transfer if Stage IIIA) |
| Stage IV | As Stage III PLUS lateral ankle pain. Deltoid insufficiency. Ankle valgus tilt. | Ankle valgus tilt on AP ankle (tilted talus). Tibiotalar arthritis. Lateral ankle mortise widening. | Tibiotalocalcaneal (TTC) arthrodesis. Consider total ankle replacement in select cases. |
Stage II Subdivisions (Myerson Modification)
Stage IIA: Flexible hindfoot valgus, hindfoot corrects when forefoot is unloaded. Cotton test negative. Treat with hindfoot procedures only.
Stage IIB: Flexible hindfoot valgus BUT fixed forefoot varus (first ray dorsiflexed). Cotton test positive (elevating heel under first ray does NOT correct arch). The forefoot varus drives the hindfoot valgus (compensatory). MUST address forefoot with medial cuneiform plantar flexion osteotomy PLUS hindfoot procedures. Missing this = failure.
Clinical Assessment
History
- Pain location: Medial ankle (PTT), lateral (peroneal overload, sinus tarsi), plantar (arch strain)
- Onset: Acute trauma vs insidious (degenerative)
- Progression: Worsening deformity over months to years
- Function: Walking distance, stairs, uneven ground tolerance
- Footwear: Difficulty fitting shoes, wearing out medial heel
- Red flags: Inflammatory symptoms (RA), neuropathy (diabetes), acute swelling (DVT)
Examination Sequence
Standing (weight-bearing):
- Too many toes sign (view from behind)
- Arch height vs normal side
- Hindfoot valgus alignment
- Forefoot abduction
- Compensatory external tibial rotation
Seated (non-weight-bearing):
- PTT palpation: swelling, tenderness, gap
- Single heel rise test (each foot separately)
- Hindfoot flexibility: valgus stress test
- Cotton test (Stage IIB screening)
- Silfverskiöld test (gastrocnemius contracture)
- Neurovascular examination
Key Clinical Tests
Examination Tests for AAFD
Technique: Examiner stands behind patient who stands with feet shoulder-width apart.
Positive test: More than 2 toes visible lateral to heel on affected side (normally only 5th and part of 4th visible). Indicates forefoot abduction and hindfoot valgus.
Sensitivity: 90% for Stage II or greater AAFD.
Technique: Patient stands on one leg, rises up on toes (heel off ground). Observe hindfoot from behind.
Normal: Hindfoot inverts (heel shifts to varus) as PTT contracts and arch rises.
Abnormal (PTT dysfunction): Hindfoot remains in valgus or everts further. Patient may be unable to perform heel rise (complete PTT rupture/dysfunction).
Gold standard for PTT function. Inability to perform = Stage II or greater.
Technique: With patient seated (unloaded), examiner grasps heel and attempts to correct hindfoot valgus into neutral or varus.
Flexible (Stage II): Hindfoot corrects passively to neutral. Indicates soft tissue deformity (PTT, spring ligament) without fixed bony/arthritic changes.
Rigid (Stage III): Hindfoot does NOT correct. Indicates subtalar or talonavicular arthritis, fixed bony deformity. Changes treatment to arthrodesis.
Technique: With patient seated, elevate first ray (plantarflex first metatarsal-cuneiform joint) and observe hindfoot alignment.
Negative (Stage IIA): Hindfoot valgus CORRECTS when first ray is elevated. Forefoot is flexible.
Positive (Stage IIB): Hindfoot valgus PERSISTS despite first ray elevation. Indicates fixed forefoot varus driving hindfoot valgus (compensatory). MUST address forefoot with medial cuneiform plantar flexion osteotomy.
Technique: With knee extended, dorsiflex ankle. Then flex knee to 90 degrees and dorsiflex ankle.
Positive: Dorsiflexion improves with knee flexed (isolated gastrocnemius contracture). May require gastrocnemius recession in addition to hindfoot reconstruction.
Significance: Gastrocnemius contracture exacerbates hindfoot valgus (equinus drives valgus). Affects surgical planning.
Do Not Miss Bilateral Disease
Approximately 20% of AAFD patients have bilateral involvement (often asymmetric). Always examine BOTH feet and compare. The "normal" side may be early-stage asymptomatic AAFD. Failure to identify bilateral disease leads to:
- Incorrect comparison (thinking abnormal is "normal for patient")
- Missed opportunity for early intervention on asymptomatic side
- Patient dissatisfaction when contralateral side progresses post-operatively
Investigations
Imaging Protocol for AAFD
AP foot: Talonavicular uncovering (lateral subluxation of navicular on talus), forefoot abduction, talo-first metatarsal angle.
Lateral foot: Calcaneal pitch angle (normal 20-30 degrees, decreased in AAFD), talo-first metatarsal angle (normal 0-5 degrees, increased Meary angle in AAFD indicates arch collapse), talus plantar flexion/sagging.
AP ankle (if Stage III/IV suspected): Assess for ankle valgus tilt, tibiotalar arthritis, lateral mortise widening (deltoid insufficiency).
Hindfoot alignment view (Saltzman view): Quantifies hindfoot valgus. Taken with patient standing, X-ray beam angled 20 degrees cephalad from behind ankle. Measures weight-bearing axis of hindfoot.
Critical: Must be weight-bearing to assess deformity magnitude. Non-weight-bearing films underestimate deformity.
Indications: Uncertain diagnosis, assessing PTT integrity (surgical planning), evaluating spring ligament status, ruling out occult fractures or other pathology.
Findings:
- PTT: Increased T2 signal (tenosynovitis), thinning, discontinuity (tear), fluid in sheath
- Spring ligament: Attenuation, tear, increased signal
- Bone marrow edema: Talar head (overload), navicular stress
- Subtalar/talonavicular arthritis: Cartilage loss, subchondral changes
Bluman MRI Classification: Grade 1 (intact tendon, peritendinitis), Grade 2 (partial tear less than 50%), Grade 3 (greater than 50% or complete rupture).
Indications: Assessing subtalar or talonavicular arthritis (planning arthrodesis), evaluating tarsal coalition (rigid flatfoot differential), planning complex osteotomies.
Advantages: Better bony detail than MRI. Can perform 3D reconstructions for preoperative planning (especially useful for lateral column lengthening or complex deformity correction).
Limited role: Dynamic assessment of PTT (real-time movement), identifying complete rupture vs partial tear. Operator-dependent. Not routinely used in Australia.
Key Radiographic Measurements
| Measurement | Normal Value | AAFD Finding | Clinical Significance |
|---|---|---|---|
| Calcaneal pitch angle (lateral) | 20-30 degrees | Less than 18 degrees (arch collapse) | Decreased angle indicates loss of arch height |
| Talo-first metatarsal angle (lateral, Meary angle) | 0-5 degrees | Greater than 10 degrees (arch sag) | Measures alignment of talus and first metatarsal - should be collinear |
| AP talonavicular coverage angle | Less than 7 degrees | Greater than 20 degrees (uncovering) | Lateral subluxation of navicular off talar head - forefoot abduction |
| Hindfoot alignment (Saltzman view) | 0 ± 3mm from midline | Greater than 5mm lateral deviation (valgus) | Quantifies hindfoot valgus deformity for surgical planning |
Management Algorithm

Conservative Management Indications
Appropriate for:
- Stage I (tenosynovitis without deformity)
- Mild Stage IIA with minimal symptoms
- Patients unfit for surgery (medical comorbidities)
- Patient preference (informed consent)
Conservative Treatment Protocol
Goals: Reduce inflammation, offload PTT, control pain
- Immobilization: CAM walker boot or short leg cast, non-weight-bearing or protected weight-bearing
- NSAIDs: Oral anti-inflammatories (if no contraindications)
- Ice: 15-20 minutes TDS over medial ankle
- Activity modification: Avoid prolonged standing, stairs, uneven ground
- Rest: PTT needs unloading to allow healing/reduction of inflammation
Goals: Restore function, prevent recurrence, transition to orthotic
- Progressive weight-bearing: Gradual transition from boot to supportive footwear
- Physiotherapy: PTT strengthening (resisted inversion), intrinsic muscle strengthening, proprioception
- Orthotic prescription: UCBL (University of California Berkeley Laboratory) insert or AFO (ankle-foot orthosis)
- Footwear advice: Firm heel counter, medial arch support, avoid flat flexible shoes
Goals: Prevent progression, maintain function, surveillance
- Orthotic compliance: Daily use in all footwear
- Weight management: Reduce mechanical load on PTT
- Activity modification: Avoid high-impact activities, use walking aids if needed
- Surveillance: 6-12 monthly review to assess for progression to Stage II
- Progression criteria: Increasing deformity, too many toes sign, failed single heel rise = surgical referral
When Conservative Fails
Indications for surgery in Stage I patients:
- Persistent pain despite 6 months of appropriate conservative treatment
- Progressive deformity (transition to Stage II)
- Failed single heel rise test (PTT incompetence)
- MRI showing Grade 3 PTT tear (complete rupture) - these will not heal
Important: Not all Stage I patients progress. Approximately 50% respond to conservative treatment with orthotics. However, those with Grade 3 MRI tears should be offered early surgical intervention (FDL transfer before deformity develops).
Surgical Technique: Stage II Reconstruction
Pre-operative Planning
Consent Points
- Undercorrection: 20-30% recurrence risk, may need revision
- Overcorrection: Hindfoot varus (painful rigid foot), lateral column overload
- Nerve injury: Sural nerve (lateral approaches), saphenous nerve, tibial nerve branches
- Infection: Superficial 5-10%, deep 2-5% (higher if diabetic)
- Nonunion: Calcaneal osteotomy (5%), Evans osteotomy if bone graft used (10%)
- Donor site morbidity: FDL harvest (toe clawing rare, flexion weakness), iliac crest if used (pain)
- Prolonged recovery: 3 months non-weight-bearing, 6-12 months full recovery
- Adjacent joint arthritis: Ankle, subtalar, talonavicular (long-term risk)
Equipment Checklist
- Implants: Cannulated screws (6.5mm or 7.0mm for calcaneus), interference screw or suture anchor (FDL), Evans plate if LCL planned
- Power tools: Oscillating saw (osteotomies), drill, reamer
- Imaging: C-arm with ankle/foot capabilities, ensure can obtain hindfoot alignment view
- Bone graft: Allograft tricortical iliac crest or femoral head (Evans), consider autograft harvest set if preferred
- Tendon instruments: Nerve hooks, right-angle clamps, drill for navicular tunnel
- Retractors: Self-retaining (Weitlaner), Army-Navy, Hohmann
Patient Positioning and Setup
Setup Checklist
Supine on radiolucent table (Jackson table or standard with radiolucent foot extension).
- Head: Neutral, adequate airway access
- Upper body: Arms on arm boards or across chest (depends on anaesthetic preference)
- Pelvis: Centered on table, neutral alignment
- Operative leg: Hip neutral rotation (or slight external rotation), knee extended or slight flexion (bump under knee for relaxation)
- Non-operative leg: Abducted and supported on leg holder or pillow (out of C-arm path)
- Bony prominences: Sacrum, heels (non-operative), elbows
- Nerves at risk: Ulnar nerve (elbow padding), peroneal nerve at fibular head (if leg externally rotated or in leg holder)
- Pressure areas: Ensure heel of operative foot OFF table (ankle hanging free or on bolster)
Thigh tourniquet (standard):
- Apply over cast padding to proximal thigh
- Exsanguinate with Esmarch or elevation
- Pressure: 250-300 mmHg (adjust for patient size/BP)
- Expected time: 90-120 minutes (multiple osteotomies + transfer)
Advantages: Bloodless field aids dissection, tendon identification, osteotomy precision Disadvantages: Tourniquet pain (limited by anaesthetic), time limit (deflate and re-exsanguinate if exceeds 2 hours)
- Prep: Circumferential ankle and foot to mid-calf. Include proximal calf if anticipating gastrocnemius recession.
- Draping: Stockinette over foot, U-drape or split sheet isolating foot and ankle
- Landmarks exposed: Medial malleolus to toes (medial approach for FDL, medial cuneiform), lateral calcaneus (for MCO through lateral or oblique approach, Evans if needed)
- C-arm positioning: Ensure adequate AP, lateral, oblique views of foot AND hindfoot alignment view possible without repositioning patient. Test BEFORE prep/drape.
Positioning Pearl for Hindfoot Alignment View
The Saltzman hindfoot alignment view (20-degree cephalad beam from posterior) is critical for assessing intraoperative correction. Position the C-arm BEFORE starting - you need to confirm adequate valgus correction post-osteotomy. If you cannot obtain this view intraoperatively, you are operating "blind" on alignment.
Complications
| Complication | Incidence | Risk Factors | Management |
|---|---|---|---|
| Undercorrection / Recurrent deformity | 20-30% (varies by study and definition) | Stage IIB missed (no forefoot correction), inadequate MCO translation, FDL transfer insufficient tension, patient non-compliance (early weight-bearing) | Mild: Orthotics, activity modification. Moderate-Severe: Revision surgery (repeat MCO, revision transfer, consider arthrodesis if progression to Stage III) |
| Overcorrection (Hindfoot varus) | 5-10% | Excessive MCO translation (greater than 12mm), over-lengthening Evans (greater than 12mm), FDL transfer overtightened | Painful rigid foot, lateral column overload. Mild: Orthotics (lateral heel wedge), shoe modifications. Severe: Revision osteotomy (lateralizing calcaneus) or takedown and redo |
| Nonunion (Calcaneal osteotomy) | 5-10% (higher if smokers, diabetics) | Smoking, diabetes, inadequate fixation, early weight-bearing, infection | If asymptomatic: Observe. If painful: Revision fixation (ORIF with bone graft, consider bone stimulator) |
| Nonunion (Evans osteotomy graft) | 10-15% (if bone graft used) | Allograft (higher than autograft), smoking, inadequate fixation | Often asymptomatic (graft provides structural support even without union). If painful: Revision with autograft and rigid fixation |
| Sural nerve injury | 5-15% (numbness, neuroma) | Lateral approaches (MCO, Evans), inadequate identification and protection | Numbness lateral foot/ankle (variable distribution). Usually improves over 6-12 months. Painful neuroma: Nerve blocks, desensitization, rarely neuroma excision |
| Infection (Superficial) | 5-10% | Diabetes, obesity, smoking, prolonged surgery, inadequate wound care | Antibiotics (oral if cellulitis), wound care, dressing changes. Rarely needs debridement. |
| Infection (Deep / Osteomyelitis) | 2-5% (higher in diabetics) | Diabetes, immunosuppression, contamination, hardware | Debridement, hardware removal (once union achieved), IV antibiotics (6 weeks minimum), consider VAC therapy. May require staged reconstruction or amputation if severe. |
| FDL donor site morbidity (Toe clawing, weak flexion) | Less than 5% (rare) | Tight closure of FDL harvest site, loss of FDL to lesser toes | Usually minimal functional impact (FHL compensates). Clawing: Toe taping, silicone sleeves. Rarely needs surgical correction (IP fusion if fixed deformity) |
| Calcaneocuboid arthritis (Post-Evans) | 10-20% radiographic, 5-10% symptomatic | Over-distraction (greater than 12mm), intra-articular osteotomy, graft malposition | Mild: NSAIDs, activity modification, orthotics. Moderate-Severe: Calcaneocuboid fusion (salvage) |
| Progression to Stage III/IV (Adjacent joint arthritis) | 10-15% long-term (10+ years) | Undercorrection, high-demand activities, obesity, age at surgery | Surveillance with serial radiographs. Symptomatic arthritis: Arthrodesis (subtalar, triple, or TTC depending on location) |
Beware the Diabetic Foot in AAFD
Patients with diabetes and AAFD have:
- Higher infection risk (2-3× baseline)
- Impaired wound healing (neuropathy, vascular disease)
- Higher nonunion rates (metabolic factors)
- Risk of Charcot neuroarthropathy progression (especially if neuropathy present)
Pre-operative optimization: HbA1c less than 7.5% (ideally less than 7%), vascular assessment (ABI, consider angiography if abnormal), neuropathy assessment. Consider staged procedures (e.g., FDL transfer first, then osteotomies later) to reduce single-operation complexity. Extended non-weight-bearing (12-16 weeks vs 8-12 weeks) to ensure healing.
Postoperative Care and Rehabilitation
Rehabilitation Timeline After Stage II Reconstruction
Goals: Protect osteotomies and FDL transfer, prevent swelling and complications.
- Immobilization: Below-knee backslab (posterior slab), transition to cast at 2 weeks after suture removal
- Weight-bearing: Strict non-weight-bearing (crutches or walker, NO weight on operative foot)
- Elevation: Leg elevated above heart level as much as possible (reduces swelling)
- Ice: 20 minutes every 2-3 hours over cast (NOT directly on skin)
- DVT prophylaxis: Aspirin 100mg daily OR enoxaparin (if high risk - obesity, prior DVT, prolonged immobility). Mechanical prophylaxis (foot pumps, TED stockings) if possible.
- Pain management: Multimodal (paracetamol + NSAID + opioid PRN), wean opioids by week 2
- Wound check: POD 10-14 for suture/staple removal, inspect for infection
Goals: Continue protection, begin gentle ankle motion (out of cast), monitor for healing.
- Immobilization: Below-knee circumferential cast or CAM walker boot (removable)
- Weight-bearing: Continue non-weight-bearing (osteotomies not yet healed)
- Physiotherapy: Gentle ankle range of motion exercises OUT of cast/boot (plantarflexion, dorsiflexion only - NO inversion/eversion). Knee and hip exercises to prevent stiffness.
- Radiographs: 6-week X-rays (AP, lateral foot, hindfoot alignment) to assess osteotomy healing, hardware position
Goals: Transition to weight-bearing, advance range of motion, restore gait.
- Immobilization: CAM walker boot (allows progressive weight-bearing)
- Weight-bearing: Progressive weight-bearing if radiographs show healing:
- Weeks 6-8: Touch weight-bearing to partial (25-50% body weight), progress as tolerated
- Weeks 8-10: Partial to full weight-bearing in boot
- Weeks 10-12: Full weight-bearing in boot, transition to supportive shoe + orthotic
- Physiotherapy: Active ankle ROM (all planes), proprioception exercises (balance board), gait re-education (normal heel-toe pattern), FDL strengthening (resisted inversion)
- Precautions: NO high-impact activities (running, jumping), avoid uneven ground
Goals: Restore full function, strengthen supporting muscles, prevent recurrence.
- Footwear: Transition to supportive athletic or walking shoes with custom orthotic (UCBL or similar to support arch)
- Weight-bearing: Full unrestricted weight-bearing (out of boot)
- Physiotherapy: Progressive resistance exercises (ankle strengthening - inversion, eversion, plantarflexion, dorsiflexion), calf strengthening (heel raises), proprioception, sport-specific training if applicable
- Activity progression: Low-impact activities (walking, cycling, swimming) → gradual return to higher-impact if appropriate
- Radiographs: 3-month and 6-month follow-up X-rays to confirm union, assess alignment
Goals: Maintain correction, monitor for recurrence, prevent adjacent joint arthritis.
- Orthotic compliance: Daily use in ALL footwear (critical to prevent recurrence)
- Footwear: Firm heel counter, medial arch support, avoid flat flexible shoes
- Activity modification: Avoid prolonged barefoot walking, high-impact repetitive loading
- Surveillance: Annual radiographs for first 2-3 years to monitor for recurrence, then as needed if symptomatic
- Weight management: Maintain healthy BMI to reduce mechanical load
- Patient education: Recognize signs of recurrence (increasing arch sag, hindfoot valgus, pain) and report early
Why Extended Non-Weight-Bearing?
AAFD reconstruction involves multiple osteotomies (calcaneus ± Evans ± cuneiform) and soft tissue transfer (FDL). Early weight-bearing risks:
- Osteotomy nonunion or malunion (especially calcaneus with metal translation)
- Hardware failure (screw pullout, plate breakage)
- FDL transfer failure (anchor pullout, tendon elongation with loss of tension)
- Recurrent deformity (hindfoot settles back into valgus)
8-12 weeks non-weight-bearing is standard. Some surgeons extend to 12 weeks for complex reconstructions (Stage IIB with multiple osteotomies) or high-risk patients (diabetics, osteoporotic).
Outcomes and Prognosis
Functional Outcomes by Stage
| Procedure | Pain Relief | Function / Satisfaction | Durability |
|---|---|---|---|
| Stage I Conservative (Orthotic) | 50-60% achieve adequate pain relief at 1 year | Moderate functional improvement, ongoing orthotic dependence, compliance variable | 30-40% progress to Stage II within 5 years if Grade 3 PTT tear on MRI |
| Stage II Reconstruction (MCO + FDL + LCL) | 70-80% good to excellent pain relief | 75-85% patient satisfaction, improved gait, ability to return to low-impact activities | 20-30% recurrence (residual symptoms or progressive deformity) at 5-10 years. Undercorrection most common cause. |
| Stage III Triple Arthrodesis | 80-85% significant pain relief (if solid fusion achieved) | 65-75% satisfaction (lower than Stage II due to loss of motion). Stable plantigrade foot. Functional limitations on uneven ground. | 10-15% nonunion. 20-30% develop adjacent joint arthritis (ankle, naviculocuneiform) by 10 years post-fusion. |
| Stage IV TTC Fusion | 70-75% pain relief (if solid fusion achieved) | 50-60% satisfaction (significant functional limitation - complete loss of ankle and hindfoot motion). Salvage procedure. | High nonunion risk (10-15%). Patients need lifelong rocker-bottom shoes, significant gait alteration. Quality of life impact substantial. |
Predictors of Poor Outcome
Factors associated with WORSE outcomes after Stage II reconstruction:
- Obesity (BMI greater than 35): Increased mechanical load, higher recurrence
- Diabetes: Impaired healing, higher infection, neuropathy (balance issues)
- Smoking: Nonunion risk, wound complications
- Advanced age (greater than 70): Lower functional reserve, slower rehabilitation
- Inflammatory arthropathy (RA, seronegative): Ongoing systemic disease progression
- Stage IIB without forefoot correction: Cotton test missed, inevitable recurrence
- Delayed presentation (longstanding severe deformity): Fixed changes, worse baseline alignment
Counsel patients pre-operatively about realistic expectations based on risk factors. Consider arthrodesis earlier (Stage II → Stage III procedure) in high-risk patients.
Evidence Base and Key Trials
Systematic Review: FDL vs FHL Transfer for Stage II AAFD
- Systematic review of 15 studies comparing FDL and FHL transfers in AAFD reconstruction
- FDL transfer: 78% good-excellent outcomes, 5% donor site morbidity (toe weakness/clawing)
- FHL transfer: 76% good-excellent outcomes, 12% donor site morbidity (hallux weakness, IP joint stiffness)
- No significant difference in pain relief or radiographic correction between FDL and FHL
- FDL had lower donor morbidity and easier harvest (medial approach vs deep posterior for FHL)
Medializing Calcaneal Osteotomy vs Lateral Column Lengthening for Stage II AAFD
- Comparative study of MCO alone (n=42) vs MCO + Evans LCL (n=38) in Stage IIA AAFD
- Both groups underwent FDL transfer in addition to osteotomy
- MCO + LCL group: Greater radiographic correction of forefoot abduction and arch height (talo-1st MT angle improved 8 degrees more than MCO alone)
- MCO alone group: 30% had residual forefoot abduction symptoms at 2 years
- MCO + LCL group: 15% lateral column pain (calcaneocuboid overload), but 85% satisfied
- Recurrence rate: MCO alone 25%, MCO + LCL 12% at 5 years
Long-term Outcomes After Triple Arthrodesis for Stage III AAFD
- Retrospective cohort of 87 patients (92 feet) with triple arthrodesis for Stage III AAFD, mean follow-up 12 years
- Solid fusion achieved in 85% (nonunion 15%, highest rate at talonavicular joint)
- Pain relief: 80% good-excellent, 12% moderate, 8% poor (persistent pain despite fusion)
- Adjacent joint arthritis: 28% developed ankle arthritis, 18% naviculocuneiform arthritis by 10 years
- Reoperation rate: 22% (hardware removal 12%, revision fusion for nonunion 6%, ankle fusion 4%)
- Patient satisfaction: 72% satisfied despite functional limitations (stiff hindfoot)
Cotton Test Accuracy for Predicting Stage IIB (Forefoot-Driven) AAFD
- Prospective study of 62 patients with Stage II AAFD undergoing reconstruction
- Cotton test performed pre-operatively (elevation of first ray with hindfoot alignment observed)
- Positive Cotton test (hindfoot valgus persists with first ray elevated): 28 patients (45%)
- All positive Cotton test patients underwent medial cuneiform osteotomy in addition to MCO + FDL
- Negative Cotton test patients: MCO + FDL only
- At 2-year follow-up: Positive Cotton group with cuneiform osteotomy had 8% recurrence. Negative Cotton group had 18% recurrence.
- Importantly: 4 patients had false-negative Cotton test (hindfoot seemed to correct but still had recurrence) - likely examiner technique variation
Australian Orthopaedic Foot and Ankle Registry Data on AAFD Reconstruction Outcomes
- Registry data from 456 AAFD reconstruction procedures (2015-2023) across Australian centers
- Stage II reconstruction (MCO + FDL ± LCL): 68% of procedures, 3-year revision rate 8.5%
- Triple arthrodesis: 24% of procedures, 3-year revision rate 12% (mostly for nonunion)
- Infection rate: 4.2% overall (higher in diabetics 9.8% vs non-diabetics 2.1%)
- Most common reason for revision: Undercorrection / recurrent deformity (42%), nonunion (28%), infection (18%)
- Smoking significantly increased revision risk (HR 2.3, p less than 0.01) and infection risk (OR 3.1, p less than 0.01)
Exam Viva Scenarios
Practice these scenarios to excel in your viva examination
Scenario 1: Stage II Classification and Management Decision (2-3 minutes)
"A 52-year-old female presents with progressive medial foot pain and flattening of her arch over 2 years. She has type 2 diabetes (HbA1c 7.2%) and BMI 32. On examination, you observe a too many toes sign, and she is unable to perform a single heel rise on the affected side. Her hindfoot corrects to neutral when you passively stress it into varus (seated examination). You perform a Cotton test: when you elevate her first ray (plantarflex the first metatarsal), her hindfoot valgus DOES correct. Weight-bearing radiographs show hindfoot valgus with talonavicular uncovering of 25 degrees and decreased calcaneal pitch. What is your assessment and management plan?"
Scenario 2: Surgical Technique - Medializing Calcaneal Osteotomy (3-4 minutes)
"Walk me through your technique for performing a medializing calcaneal osteotomy as part of a Stage II AAFD reconstruction. Specifically, describe your approach, osteotomy location and orientation, how much translation you aim for, and your fixation method. What nerves are at risk and how do you protect them?"
Scenario 3: Complication Management - Recurrent Deformity (2-3 minutes)
"A 58-year-old patient returns 18 months after Stage IIA reconstruction (MCO + FDL transfer, no lateral column lengthening) with recurrent medial foot pain and visible return of arch collapse. Radiographs show hindfoot valgus has recurred (calcaneal osteotomy healed in translated position) and talonavicular uncovering has worsened from 20 degrees pre-op to 15 degrees immediately post-op to now 30 degrees. She is obese (BMI 36) and non-compliant with orthotic use (admits she rarely wears it). How do you approach this patient?"
MCQ Practice Points
Anatomy Question
Q: What is the PRIMARY insertion of the posterior tibial tendon and why is it critical for arch support? A: The primary insertion is the navicular tuberosity. This is critical because the navicular is the keystone of the medial longitudinal arch. The PTT's pull on the navicular (via its insertion) lifts the arch during stance phase. When the PTT fails, the navicular sags inferiorly and the arch collapses. The PTT has secondary insertions to all 3 cuneiforms and metatarsal bases 2-4, providing distributed midfoot stability, but the navicular insertion is the main arch supporter.
Classification Question
Q: What distinguishes Stage IIA from Stage IIB in the Johnson and Strom classification, and why does this matter for surgical planning? A: Both are flexible deformities, but the key difference is the forefoot. Stage IIA has a flexible forefoot - the Cotton test is negative (hindfoot valgus corrects when first ray is elevated). Stage IIB has a fixed forefoot varus - the Cotton test is positive (hindfoot valgus persists despite first ray elevation because the forefoot varus is DRIVING the hindfoot valgus compensatorily). This matters because Stage IIB requires additional surgery: medial cuneiform plantar flexion osteotomy (Cotton osteotomy) to correct the forefoot varus. Without this, the reconstruction WILL fail because the uncorrected forefoot drives recurrent hindfoot valgus.
Clinical Test Question
Q: Describe the single heel rise test and what a positive (abnormal) test indicates. A: The patient stands on one leg (the affected side) and attempts to rise up on tiptoes (heel rise off ground). The examiner observes the hindfoot from behind. Normal: As the patient rises, the hindfoot inverts (shifts from valgus to varus) due to tibialis posterior contraction. Abnormal (positive test): The hindfoot remains in valgus or everts further, OR the patient is completely unable to perform a heel rise. This indicates posterior tibial tendon dysfunction - the PTT is incompetent and cannot generate the inversion moment. A positive test signifies Stage II or greater AAFD (Stage I patients can usually still perform heel rise, though may have pain).
Surgical Technique Question
Q: Why is FDL preferred over FHL for tendon transfer in AAFD reconstruction? A: FDL advantages: (1) Better excursion - longer muscle belly and arc of contraction provides greater active inversion force. (2) Lower donor morbidity - FHL is the stronger hallux flexor; transferring FHL causes more noticeable hallux weakness and potential IP joint stiffness. FDL loss to lesser toes is rarely symptomatic (FHL compensates). (3) Easier harvest - medial approach (same as navicular exposure) vs deep posterior approach for FHL. (4) Equivalent strength - biomechanical studies show FDL provides sufficient force for arch support. Both FDL and FHL have similar clinical outcomes in terms of pain relief and deformity correction, but FDL has better risk-benefit profile.
Complication Question
Q: What is the most common cause of failure after Stage II AAFD reconstruction, and how can it be prevented? A: The most common cause is undercorrection / recurrent deformity (occurs in 20-30% of patients). Causes include: (1) Missing Stage IIB - failing to perform medial cuneiform osteotomy when forefoot varus is present (Cotton test positive). (2) Inadequate calcaneal translation - not medializing enough (should aim for 8-12mm, confirmed on hindfoot alignment view). (3) Missing lateral column lengthening - when significant forefoot abduction present (talonavicular uncovering greater than 30 degrees, too many toes sign) but LCL not performed. (4) Patient factors - obesity (high BMI increases mechanical load), diabetes, non-compliance with orthotic use post-op. Prevention: Meticulous pre-operative planning (Cotton test, assess forefoot abduction, measure angles), adequate surgical correction (confirm with intraoperative fluoroscopy), address patient risk factors (weight loss, diabetes control), enforce orthotic compliance post-operatively.
Evidence Question
Q: What does the Australian Orthopaedic Foot and Ankle Registry data tell us about revision rates and risk factors for AAFD reconstruction? A: AOANJRR 2023 data (456 AAFD procedures): Stage II reconstruction has a 3-year revision rate of 8.5%. Triple arthrodesis has a 3-year revision rate of 12% (mostly for nonunion). The most common reasons for revision are undercorrection/recurrent deformity (42% of revisions), nonunion (28%), and infection (18%). Smoking is a significant risk factor - increases revision risk (hazard ratio 2.3) and infection risk (odds ratio 3.1). Diabetes increases infection rate (9.8% in diabetics vs 2.1% in non-diabetics). This Australian data confirms international findings and emphasizes the importance of smoking cessation and diabetic optimization pre-operatively.
Australian Context and Medicolegal Considerations
AOANJRR Registry Data
- 3-year revision rate: 8.5% (Stage II reconstruction), 12% (triple arthrodesis)
- Undercorrection is most common revision reason (42% of revisions)
- Smoking increases revision risk (HR 2.3) - counsel cessation pre-operatively
- Diabetes increases infection (9.8% vs 2.1%) - optimize HbA1c to below 7.5%
- Registry participation: Mandatory reporting for quality improvement and benchmarking
Australian Guidelines and Standards
- ACSQHC Surgical Site Infection Prevention: Pre-operative skin antisepsis (chlorhexidine alcohol), antibiotic prophylaxis (cefazolin 2g within 60 min of incision), normothermia, glycemic control (target glucose 6-10 mmol/L peri-operatively)
- National Safety and Quality Health Service Standards: Informed consent documentation (risks, benefits, alternatives), surgical safety checklist (WHO checklist adapted for Australian use)
- DVT prophylaxis: Australian and New Zealand Working Party guidelines - mechanical prophylaxis (TED stockings, foot pumps) PLUS pharmacological (enoxaparin or aspirin) for foot/ankle surgery with prolonged immobilization
Medicolegal Considerations Specific to AAFD Reconstruction
Key documentation requirements:
-
Informed consent must include:
- Realistic success rate (75-85% good outcomes, but 20-30% recurrence risk)
- Prolonged recovery (3-6 months to full function, 12+ months to final outcome)
- Complications: Undercorrection (20-30%), overcorrection (5-10%), nerve injury (sural nerve 5-15%), infection (superficial 5-10%, deep 2-5%), nonunion (5-15%), progression to arthrodesis (10-15% long-term)
- Alternative treatments: Conservative (orthotic), arthrodesis (if rigid deformity)
- Expected functional outcomes: Improved pain and gait, but NOT return to high-impact sports
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Common litigation issues:
- Failure to diagnose Stage IIB (missing Cotton test) leading to recurrence - document Cotton test result in notes
- Nerve injury (sural nerve, tibial nerve branches) without pre-operative counseling - must consent for numbness risk
- Infection in diabetic patients without adequate optimization - document HbA1c, pre-operative counseling, glucose control peri-op
- Undercorrection perceived as surgical failure - set realistic expectations (not all patients achieve perfect correction), document degree of deformity pre-op and plan for multi-stage if severe
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Documentation best practices:
- Pre-operative: Clinical photos (too many toes sign, arch height), weight-bearing radiographs with measurements (angles documented), Cotton test result, Silfverskiöld test, single heel rise test result
- Intraoperative: Fluoroscopy images saved (hindfoot alignment view showing correction), degree of calcaneal translation measured and documented, FDL transfer tension confirmed
- Post-operative: Serial radiographs to monitor healing and alignment, document orthotic prescription and compliance counseling
Adult Acquired Flatfoot Deformity
High-Yield Exam Summary
Key Anatomy
- •PTT = primary dynamic arch stabilizer (over 50% of support)
- •Navicular tuberosity = primary PTT insertion (arch keystone)
- •Hypovascular zone 2-4cm proximal to insertion = site of rupture
- •Spring ligament = static stabilizer (fails secondary to PTT loss)
- •3-plane deformity: hindfoot valgus (coronal) + arch collapse (sagittal) + forefoot abduction (axial)
Johnson and Strom Classification
- •Stage I = PTT tenosynovitis, no deformity, conservative treatment
- •Stage IIA = Flexible hindfoot, negative Cotton test, MCO + FDL ± LCL
- •Stage IIB = Flexible hindfoot + fixed forefoot varus (positive Cotton), add medial cuneiform osteotomy
- •Stage III = Rigid deformity, subtalar/triple arthrodesis
- •Stage IV = Ankle valgus tilt (deltoid insufficiency), TTC fusion
Clinical Assessment Algorithm
- •Standing: Too many toes sign (forefoot abduction + hindfoot valgus)
- •Functional: Single heel rise test (PTT function - negative = incompetent)
- •Seated: Hindfoot flexibility test (Stage II flexible, Stage III rigid)
- •Cotton test: Elevate first ray - if valgus persists = Stage IIB (forefoot-driven)
- •Silfverskiöld test: Gastrocnemius contracture (add recession if positive)
Surgical Decision Points
- •Flexible (Stage II) = osteotomy + tendon transfer, Rigid (Stage III) = arthrodesis
- •FDL over FHL: Better excursion, lower donor morbidity, easier harvest
- •LCL indications: Forefoot abduction greater than 30 degrees (talonavicular uncovering)
- •MCO translation: 8-12mm medially (confirm with hindfoot alignment view)
- •Post-op: 8-12 weeks strict non-weight-bearing (multiple osteotomies to heal)
Complications and Prevention
- •Undercorrection 20-30% = most common, prevent with adequate MCO + LCL if needed + Cotton test
- •Overcorrection 5-10% = hindfoot varus (painful), avoid greater than 12mm translation/LCL
- •Sural nerve injury 5-15% = identify and protect during lateral approach
- •Nonunion 5-15% = smoking cessation mandatory, avoid NSAIDs during healing
- •Recurrence risk factors: obesity, diabetes, orthotic non-compliance, smoking