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ALPSA Lesions

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ALPSA Lesions

Comprehensive guide to Anterior Labroligamentous Periosteal Sleeve Avulsion (ALPSA), a variant of Bankart lesion characterised by medialized healing.

complete
Updated: 2025-12-20
High Yield Overview

ALPSA LESIONS

The Medialized Bankart

Incidence10-20% of Instability
HealingMedialized
PeriosteumIntact
RecurrenceHigher than Bankart

Neviaser Classification

Acute (Type I)
PatternMobile, easy to reduce (less than 3 months)
Treatment
Chronic (Type II)
PatternFixed, scarred, needs release (greater than 3 months)
Treatment
Perthes
PatternVariation: Non-displaced sleeve avulsion
Treatment
GLAD
PatternVariation: Cartilage defect, stable labrum
Treatment

Critical Must-Knows

  • ALPSA = Anterior Labroligamentous Periosteal Sleeve Avulsion.
  • Unlike Bankart, the anterior scapular periosteum remains INTACT.
  • The labrum falls medially down the glenoid neck and heals there.
  • This eliminates the 'bumper' effect of the labrum.
  • Surgical repair requires aggressive mobilization (takedown) before fixation.

Examiner's Pearls

  • "
    Simply fixing it where it lies (in situ) leads to failure.
  • "
    Think of it as a 'healed' dislocation in a bad position.
  • "
    Common in chronic instability or younger patients.

The Surgical Trap

The Problem

"In Situ Repair" Because the ALPSA lesion heals, it can look like a normal (but small) labrum. If you just put anchors in and stitch it, you validatethe malposition. You have NOT restored the bumper. Recurrence is guaranteed.

The Solution

"Mobilize and Elevate" You must incise the healed tissue. Elevate the labrum off the glenoid neck. Bring it up to the articular margin (the 'face'). Only then do you fix it.

FeatureBankart LesionALPSA Lesion
PeriosteumTorn (Detached)Intact (Stripped)
PositionFloatingMedialized on Neck
HealingDoes not heal (Gap)Heals in bad position
RecurrenceStandard riskHigher risk (if not mobilized)
Mnemonic

GAPVariants of Instability

G
GLAD
Glenolabral Articular Disruption (Cartilage damage, stable)
A
ALPSA
Anterior Labroligamentous Periosteal Sleeve Avulsion (Medialized)
P
Perthes
Periosteal sleeve detached but labrum NOT displaced (undisplaced ALPSA)

Memory Hook:Mind the GAP in the labrum.

Mnemonic

TRIPSurgical Steps

T
Takedown
Release healed tissue from neck
R
Release
Mobilize completely
I
Inspect
Check for bone loss
P
Plicate
Repair with tension (shift)

Memory Hook:Take a TRIP to the glenoid rim.

Mnemonic

MINTPathology

M
Medialized
Labrum moves medially
I
Intact
Intact periosteum
N
Neck
Heals on the glenoid neck
T
Tethered
Becomes tethered/scarred

Memory Hook:The labrum is stuck in a MINT condition (not really).

Overview

Definition

An ALPSA lesion (Anterior Labroligamentous Periosteal Sleeve Avulsion) involves the avulsion of the anterior labrum and IGHL from the glenoid rim. Critically, the anterior scapular periosteum remains intact but stripped, allowing the labrum to displace medially and heal inferiorly on the glenoid neck.

This "medialization" effectively shortens the anterior restraints and eliminates the glenoid depth, predisposing the joint to recurrent anterior instability even with minor trauma.

This contrasts with a Bankart lesion, where the periosteum tears, creating a distinct separation. The "sleeve" nature of the ALPSA allows the tissue to essentially slide down the neck and scar in a non-functional position.

Pathophysiology and Mechanisms

The Labroligamentous Complex

  • Labrum: Fibrocartilaginous bumper. Increases glenoid depth by 50%.
  • IGHL: Inferior Glenohumeral Ligament. Main static restraint.
  • Periosteum: Thick anterior scapular periosteum.

In ALPSA, the forceful anterior dislocation strips the periosteum without rupturing it.

This creates a "sleeve" that slides medially.

The Healing Trap

  • Because the tissue remains vascularized (via periosteum) and in contact with bone, it heals.
  • BUT: It heals on the glenoid neck, medial to the rim.
  • Consequence: The glenoid concavity is lost ("Flat glenoid"). The "chock block" effect is gone.
  • The active restraint (IGHL) is also effectively lengthened/lax.

This explains high recurrence rates in untreated ALPSA.

Biomechanical Consequences

  • Glenoid Depth Reduction: Loss of labral height reduces effective glenoid concavity by up to 50%.
  • Capsular Laxity: Medialized healing elongates the IGHL complex, reducing tension.
  • Force Couples: Disrupted force distribution across the glenohumeral joint.
  • Stability Ratio: The force required to cause dislocation is significantly reduced.

The Compression-Concavity Mechanism

The labrum normally creates a "chock block" that resists humeral head translation. When medialized, this effect is lost even though tissue is present. The joint becomes functionally equivalent to a glenoid with bone loss.

This is why repair WITHOUT mobilization fails - the bumper never returns.

Classification Systems

Neviaser Classification

  • Acute (Type I): Less than 3 months. Tissue is mobile. Can be reduced easily.
  • Chronic (Type II): Greater than 3 months. Tissue is scarred and fixed. Requires sharp dissection/release.

This distinction is crucial for surgical planning.

Chronic lesions often require a capsular release (anterior and inferior) to gain excursion.

History

Presentation

  • Recurrent Instability: Often reports "loose" shoulder.
  • Clicking/Catching: Can occur.
  • Mechanism: Often a history of multiple dislocations.
  • Age: Common in young patients (under 25).
  • Symptom Duration: Chronic symptoms suggest ALPSA vs acute Bankart.
  • Provoking Positions: Abduction and external rotation (ABER position).

The history is identical to Bankart instability.

Red Flags

  • First-time dislocation over age 40 (rotator cuff tear risk).
  • Neurological symptoms (axillary nerve injury).
  • Unable to reduce (locked posterior dislocation).

These red flags warrant urgent imaging and specialist referral.

Examination

Physical Exam

  • Apprehension: Positive.
  • Relocation: Positive.
  • Load and Shift: Increased translation (Grade 2-3).
  • Sulcus Sign: May be present (multidirectional component).
  • Jobe Relocation Test: Relief with posterior force.
  • Gagey Hyperabduction Test: Assesses inferior capsular laxity.

You cannot clinically distinguish ALPSA from Bankart.

Imaging is the only differentiator.

Differential Diagnosis

Differential Diagnosis

  • Bankart Lesion: True labral detachment, floating labrum.
  • Perthes Lesion: Non-displaced periosteal sleeve avulsion.
  • GLAD Lesion: Glenolabral articular disruption (cartilage focus).
  • HAGL Lesion: Humeral avulsion of glenohumeral ligament.
  • Bony Bankart: Labrum with attached bone fragment.
  • Multidirectional Instability: Global capsular laxity, atraumatic.

Key Distinguishing Features

LesionPeriosteumLabrum PositionStability
BankartTornFloating/DetachedUnstable
ALPSAIntactMedializedUnstable
PerthesStrippedIn situUnstable
GLADIntactIntactStable

Imaging

MRI Features

Axial T2 / PD Fat Sat. Look for the labrum ("black triangle"). Normal: Sitting on the rim. ALPSA: Rounded, scarred bundle sitting medial to the rim on the glenoid neck. The periosteum may be visualized as a low signal line connecting the labrum to the scapula (the sleeve).

MRI Signs

  • Medialization: The key sign.
  • Synovial Stripping: Contrast tracking under the sleeve.
  • Bone Loss: Assess for concomitant Bony Bankart (often remodelled in chronic cases).
  • Hill-Sachs: Usually present due to recurrence.

Look for the "Cul-de-sac" deep to the capsule.

CT Arthrography

CT arthrography (CTA) is particularly useful for:

  • Bone Loss Quantification: Precise measurement of glenoid bone loss.
  • Chronic Cases: When MRI interpretation is difficult due to scarring.
  • Preoperative Planning: 3D reconstructions for surgical approach.

CTA Findings in ALPSA

  • Medialized Contrast: Contrast tracks medially with the labrum.
  • Intact Periosteal Sleeve: Visible as soft tissue connection to scapula.
  • Glenoid Morphology: Assess for erosive bone loss vs inverted pear glenoid.

CTA is superior to MRI for detecting subtle bone loss less than 10%.

Glenoid Bone Loss Assessment

Critical for surgical planning - significant bone loss may require Latarjet.

Measurement Methods

  • Best-Fit Circle: Measure defect as percentage of inferior glenoid circle.
  • Glenoid Index: Ratio of anterior to posterior glenoid width.
  • En-Face View: 3D CT reconstruction for direct visualization.

Thresholds

  • Less than 15%: Soft tissue repair adequate.
  • 15-25%: Consider augmentation (remplissage, bone graft).
  • Greater than 25%: Latarjet typically required.

These thresholds guide surgical decision-making for optimal outcomes.

Management Algorithm

📊 Management Algorithm
ALPSA Lesion Management Algorithm Flowchart
Click to expand
Diagnostic and management strategy for ALPSA lesions, emphasizing mobilization of the medialized labrum.

Decision Making

  • Non-Operative: High failure rate in young patients. (Not recommended for athletes).
  • Operative: Indicated for recurrent instability.
  • Approach: Arthroscopic Repair is Gold Standard. Open repair rarely needed unless massive bone loss.

The key is the technical execution of the repair.

Timing

  • Early repair (Acute) is easier as tissue is not scarred.
  • Chronic repair is equally successful IF adequately mobilized.

Do not delay if diagnosis is confirmed.

Surgical Considerations

Arthroscopic Repair Steps

  1. Diagnostic Scope: Confirm diagnosis (Probe the labrum - it may feel "healed" but lacks bumper function).
  2. Mobilization (CRITICAL):
    • Use an elevator or shaver to detach the scarred labrum from the glenoid neck.
    • Release anteriorly and inferiorly (around the corner).
    • "Float the Labrum": It must rise to the rim effortlessly.
  3. Preparation: Decorticate the glenoid rim (create bleeding bed).
  4. Fixation: Suture anchors on the face (articular margin).
  5. Shift: Tension the tissue superiorly and laterally to restore the bumper.

Failure to mobilize turns this into a non-anatomical plication.

Surgical Pearls

  • The 5 O'clock Portal: Essential for inferior release.
  • Viewing: Switch to Anterior Superior portal to view the "drive-through" sign reduction.
  • Anchors: Use 3 or more anchors for robust fixation.

If the tissue is poor, incorporate capsule (Bankart Repair + Capsular Shift).

Complications

  • Recurrence: The most common complication. Often due to failure to mobilize the lesion fully.
  • Stiffness: Excessive tightening (Overtensioning).
  • Hardware Issues: Loose anchors, chondrolysis (if placed on face too proud).
  • Nerve Injury: Axillary nerve (during inferior dissection).
  • Infection: Rare.
Complication Pearl

Chondrolysis Risk Placing anchors on the articular face is necessary for ALPSA repair, BUT they must not be prominent. Prominent metal or hard PEEK anchors can destroy the humeral head cartilage (Chondrolysis). Use soft anchors or ensure knotless anchors are countersunk.

Rehabilitation

  • Phase 1 (0-6 weeks):

    • Sling immobilization.
    • Protect ER (usually restricted to 0 or 30 degrees).
    • Pendulums.
  • Phase 2 (6-12 weeks):

    • Regain ROM.
    • Active assistive to Active.
    • Scapular control.
  • Phase 3 (3-6 months):

    • Strengthening.
    • Proprioception.
    • Return to sport at 6 months (contact sports).

    Protocol is identical to Bankart Repair.

    Return to Sport Criteria

    • Full, pain-free Range of Motion.
    • Symmetrical strength (ER/IR/Abduction).
    • Psychological readiness (ACL-RSI or similar scale).
    • Completion of sport-specific training drills.
    • No apprehension in apprehension position.

    Sling Removal Criteria

    • No pain at rest.
    • Control of scapula.
    • Ability to perform ADLs waist level.

    Contact Progression

    • 4 months: Non-contact drills (passing, catching).
    • 5 months: Controlled contact (pad work).
    • 6 months: Unrestricted contact (tackling).

Prognosis

  • Recurrence Rate: With proper mobilization, results equal Bankart repair (5-10% recurrence).
  • Without Mobilization: Recurrence rates are significantly higher.
  • Return to Sport: High (greater than 85%).
  • Arthritis: Long term risk exists due to initial cartilage damage (GLAD component often co-exists).

Prognostic Factors

  • Age: Younger patients have higher recurrence risk.
  • Bone Loss: Significant glenoid bone loss (greater than 20%) requires Latarjet.
  • Hyperlaxity: Beighton score greater than 5 increases failure risk.
  • Sport: Collision athletes have higher recurrence.

Evidence Base

Original Description

4
Neviaser et al. • Arthroscopy (1993)
Key Findings:
  • Described the ALPSA lesion
  • Highlighted the intact periosteal sleeve
  • Noted medial healing and loss of bumper
Clinical Implication: Defined the pathology distinct from Bankart.

Recurrence Rates

3
Ozbaydar et al. • Knee Surg Sports Traumatol Arthrosc (2008)
Key Findings:
  • Compared Bankart vs ALPSA repair outcomes
  • Found no significant difference in recurrence IF ALPSA is mobilized
  • Emphasized technical factors
Clinical Implication: It behaves like a Bankart if you turn it back into one.

Biomechanics

5
Bigliani et al. • Am J Sports Med (1998)
Key Findings:
  • Cadaver study of capsular failure
  • IGHL strain properties
  • Showed that medialization significantly reduces restraint to anterior translation
Clinical Implication: Anatomy must be restored.

Chronic ALPSA

4
Habermeyer et al. • JSES (1999)
Key Findings:
  • Evolution of the lesion over time
  • Described the 'single anterior instability lesion' becoming ALPSA with recurrence
  • Associated with bone remodelling
Clinical Implication: Chronic instability often equals ALPSA.

Arthroscopic Technique

4
Burkhart and De Beer • Arthroscopy (2000)
Key Findings:
  • Famous paper on instability failures
  • Identified Bone Loss and ALPSA as risk factors
  • Advocated for 'Address the Tissues and the Bone'
Clinical Implication: Identify the pathology or fail.

Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: MRI Interpretation

EXAMINER

"You are shown an MRI of a 19-year-old rugby player. There is a dark lump of tissue sitting on the anterior glenoid neck, medial to the rim. What is this and how does it differ from a Bankart?"

EXCEPTIONAL ANSWER
This is an ALPSA lesion. It differs from a Bankart in that the anterior periosteum is stripped but intact, tethering the labrum medially. A Bankart is a complete detachment. This is significant because surgically, I must mobilize and take down this ALPSA lesion proper to restoring the bumper.
KEY POINTS TO SCORE
Recognition of medialized labrum
Intact periosteum concept
Surgical implication (Mobilization)
COMMON TRAPS
✗Calling it a Bankart
✗Not mentioning mobilization
LIKELY FOLLOW-UPS
"What happens if you just put an anchor in without moving it?"
"I would repair it in a non-anatomical position, failing to restore stability. Recurrence is high."
VIVA SCENARIOStandard

Scenario 2: Intra-operative Surprise

EXAMINER

"You are scoping a 'Bankart'. You enter the joint and the labrum looks 'healed' but the shoulder is loose (Drive-through sign positive). Probe shows it is firmly attached but medial. What do you do?"

EXCEPTIONAL ANSWER
This is a healed ALPSA (Type II). Do not leave it. I would use an elevator or radiofrequency device to peel the labrum off the neck (takedown), mobilize it fully until it floats to the rim, and then repair it with suture anchors to the articular margin.
KEY POINTS TO SCORE
Recognition of chronic ALPSA
Step-by-step takedown
Restoration of bumper
COMMON TRAPS
✗Leaving it alone ('It's healed')
✗Plicating over the top (Insufficient)
LIKELY FOLLOW-UPS
"Is the rehabilitation different?"
"No, standard Bankart protocol."
VIVA SCENARIOStandard

Scenario 3: The Revision

EXAMINER

"A patient had a stabilisation elsewhere and re-dislocated. MRI shows the anchors are in the glenoid neck, not the face. The labrum is still medial. Why did it fail?"

EXCEPTIONAL ANSWER
This represents a failure to mobilize an ALPSA lesion. The previous surgeon likely fixed the labrum 'in situ' on the neck. The bumper effect was never restored. I would revise this by removing/ignoring old anchors, mobilizing the tissue fully, and placing new anchors on the face.
KEY POINTS TO SCORE
Mechanism of failure
Critique of anchor placement
Revision strategy
COMMON TRAPS
✗Blaming the patient or trauma
✗Missing the technical error
LIKELY FOLLOW-UPS
"What if the tissue is poor?"
"I would consider a Latarjet (Coracoid transfer) if soft tissue quality is insufficient."
VIVA SCENARIOStandard

Scenario 4: CT Arthrography Interpretation

EXAMINER

"You are shown a CT arthrography of a 26-year-old footballer with chronic anterior instability. Contrast tracks medially along the glenoid neck with the labrum. What is the diagnosis and how does this affect your surgical planning?"

EXCEPTIONAL ANSWER
This is an ALPSA lesion on CT arthrography. The contrast tracking medially with the labrum indicates an intact periosteal sleeve with medialized healing. Surgical planning must include: assessment of bone loss using best-fit circle method, planning for extensive soft tissue mobilization, and determining if bone augmentation is needed.
KEY POINTS TO SCORE
CT arthrography findings of ALPSA
Bone loss quantification
Surgical planning implications
COMMON TRAPS
✗Confusing with Bankart (no medial tracking)
✗Failing to assess bone loss
LIKELY FOLLOW-UPS
"The CT shows 18% glenoid bone loss. What now?"
"This is in the 'grey zone' (15-25%). I would consider soft tissue repair with remplissage for engaging Hill-Sachs, or bone graft augmentation depending on patient demands."

MCQ Practice Points

Pathology

Q: What structure remains intact in an ALPSA lesion that is torn in a Bankart? A: The anterior scapular periosteum.

Terminology

Q: What does ALPSA stand for? A: Anterior Labroligamentous Periosteal Sleeve Avulsion.

Surgical Technique

Q: What is the most critical step in ALPSA repair? A: Mobilization (Takedown) of the labrum.

Classification

Q: A chronic, fixed ALPSA lesion is classified as: A: Neviaser Type II.

MRI Diagnosis

Q: What MRI finding differentiates ALPSA from Bankart lesion? A: Medially displaced labrum with intact periosteal sleeve (labrum lies against glenoid neck rather than at rim).

Recurrence Risk

Q: Why do ALPSA lesions have higher recurrence rates if not properly mobilized? A: The medialized labrum heals in a non-anatomic position, failing to restore the labral bumper effect and capsular tension.

Australian Context

  • Prevalence: Highly common in the Australian contact sport population (Rugby, AFL).
  • Practice: ALPSA recognition is a standard of care expectation for Orthopaedic candidates. "In situ" repair is considered a technical error.

High-Yield Exam Summary

Diagnosis

  • •History: Recurrent Instability
  • •MRI: Medialized Labrum
  • •MRI: Intact Periosteal Sleeve
  • •Sign: Drive-through positive

Management

  • •Arthroscopic Repair is Gold Standard
  • •CRITICAL: Mobilize/Takedown lesion
  • •Fix to Glenoid Face (Not neck)
  • •Restore Bumper Effect

Key Differentiators

  • •Bankart: Torn Periosteum, Floating
  • •ALPSA: Intact Periosteum, Medialized
  • •GLAD: Cartilage defect
  • •Perthes: Undisplaced sleeve
Quick Stats
Reading Time52 min
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