ALPSA LESIONS
The Medialized Bankart
Neviaser Classification
Critical Must-Knows
- ALPSA = Anterior Labroligamentous Periosteal Sleeve Avulsion.
- Unlike Bankart, the anterior scapular periosteum remains INTACT.
- The labrum falls medially down the glenoid neck and heals there.
- This eliminates the 'bumper' effect of the labrum.
- Surgical repair requires aggressive mobilization (takedown) before fixation.
Examiner's Pearls
- "Simply fixing it where it lies (in situ) leads to failure.
- "Think of it as a 'healed' dislocation in a bad position.
- "Common in chronic instability or younger patients.
The Surgical Trap
The Problem
"In Situ Repair" Because the ALPSA lesion heals, it can look like a normal (but small) labrum. If you just put anchors in and stitch it, you validatethe malposition. You have NOT restored the bumper. Recurrence is guaranteed.
The Solution
"Mobilize and Elevate" You must incise the healed tissue. Elevate the labrum off the glenoid neck. Bring it up to the articular margin (the 'face'). Only then do you fix it.
| Feature | Bankart Lesion | ALPSA Lesion |
|---|---|---|
| Periosteum | Torn (Detached) | Intact (Stripped) |
| Position | Floating | Medialized on Neck |
| Healing | Does not heal (Gap) | Heals in bad position |
| Recurrence | Standard risk | Higher risk (if not mobilized) |
GAPVariants of Instability
Memory Hook:Mind the GAP in the labrum.
TRIPSurgical Steps
Memory Hook:Take a TRIP to the glenoid rim.
MINTPathology
Memory Hook:The labrum is stuck in a MINT condition (not really).
Overview
An ALPSA lesion (Anterior Labroligamentous Periosteal Sleeve Avulsion) involves the avulsion of the anterior labrum and IGHL from the glenoid rim. Critically, the anterior scapular periosteum remains intact but stripped, allowing the labrum to displace medially and heal inferiorly on the glenoid neck.
This "medialization" effectively shortens the anterior restraints and eliminates the glenoid depth, predisposing the joint to recurrent anterior instability even with minor trauma.
This contrasts with a Bankart lesion, where the periosteum tears, creating a distinct separation. The "sleeve" nature of the ALPSA allows the tissue to essentially slide down the neck and scar in a non-functional position.
Pathophysiology and Mechanisms
The Labroligamentous Complex
- Labrum: Fibrocartilaginous bumper. Increases glenoid depth by 50%.
- IGHL: Inferior Glenohumeral Ligament. Main static restraint.
- Periosteum: Thick anterior scapular periosteum.
In ALPSA, the forceful anterior dislocation strips the periosteum without rupturing it.
This creates a "sleeve" that slides medially.
Classification Systems
Neviaser Classification
- Acute (Type I): Less than 3 months. Tissue is mobile. Can be reduced easily.
- Chronic (Type II): Greater than 3 months. Tissue is scarred and fixed. Requires sharp dissection/release.
This distinction is crucial for surgical planning.
Chronic lesions often require a capsular release (anterior and inferior) to gain excursion.
History
Presentation
- Recurrent Instability: Often reports "loose" shoulder.
- Clicking/Catching: Can occur.
- Mechanism: Often a history of multiple dislocations.
- Age: Common in young patients (under 25).
- Symptom Duration: Chronic symptoms suggest ALPSA vs acute Bankart.
- Provoking Positions: Abduction and external rotation (ABER position).
The history is identical to Bankart instability.
Red Flags
- First-time dislocation over age 40 (rotator cuff tear risk).
- Neurological symptoms (axillary nerve injury).
- Unable to reduce (locked posterior dislocation).
These red flags warrant urgent imaging and specialist referral.
Examination
Physical Exam
- Apprehension: Positive.
- Relocation: Positive.
- Load and Shift: Increased translation (Grade 2-3).
- Sulcus Sign: May be present (multidirectional component).
- Jobe Relocation Test: Relief with posterior force.
- Gagey Hyperabduction Test: Assesses inferior capsular laxity.
You cannot clinically distinguish ALPSA from Bankart.
Imaging is the only differentiator.
Differential Diagnosis
Differential Diagnosis
- Bankart Lesion: True labral detachment, floating labrum.
- Perthes Lesion: Non-displaced periosteal sleeve avulsion.
- GLAD Lesion: Glenolabral articular disruption (cartilage focus).
- HAGL Lesion: Humeral avulsion of glenohumeral ligament.
- Bony Bankart: Labrum with attached bone fragment.
- Multidirectional Instability: Global capsular laxity, atraumatic.
Key Distinguishing Features
| Lesion | Periosteum | Labrum Position | Stability |
|---|---|---|---|
| Bankart | Torn | Floating/Detached | Unstable |
| ALPSA | Intact | Medialized | Unstable |
| Perthes | Stripped | In situ | Unstable |
| GLAD | Intact | Intact | Stable |
Imaging
Axial T2 / PD Fat Sat. Look for the labrum ("black triangle"). Normal: Sitting on the rim. ALPSA: Rounded, scarred bundle sitting medial to the rim on the glenoid neck. The periosteum may be visualized as a low signal line connecting the labrum to the scapula (the sleeve).
MRI Signs
- Medialization: The key sign.
- Synovial Stripping: Contrast tracking under the sleeve.
- Bone Loss: Assess for concomitant Bony Bankart (often remodelled in chronic cases).
- Hill-Sachs: Usually present due to recurrence.
Look for the "Cul-de-sac" deep to the capsule.
Management Algorithm

Decision Making
- Non-Operative: High failure rate in young patients. (Not recommended for athletes).
- Operative: Indicated for recurrent instability.
- Approach: Arthroscopic Repair is Gold Standard. Open repair rarely needed unless massive bone loss.
The key is the technical execution of the repair.
Surgical Considerations
Arthroscopic Repair Steps
- Diagnostic Scope: Confirm diagnosis (Probe the labrum - it may feel "healed" but lacks bumper function).
- Mobilization (CRITICAL):
- Use an elevator or shaver to detach the scarred labrum from the glenoid neck.
- Release anteriorly and inferiorly (around the corner).
- "Float the Labrum": It must rise to the rim effortlessly.
- Preparation: Decorticate the glenoid rim (create bleeding bed).
- Fixation: Suture anchors on the face (articular margin).
- Shift: Tension the tissue superiorly and laterally to restore the bumper.
Failure to mobilize turns this into a non-anatomical plication.
Complications
- Recurrence: The most common complication. Often due to failure to mobilize the lesion fully.
- Stiffness: Excessive tightening (Overtensioning).
- Hardware Issues: Loose anchors, chondrolysis (if placed on face too proud).
- Nerve Injury: Axillary nerve (during inferior dissection).
- Infection: Rare.
Chondrolysis Risk Placing anchors on the articular face is necessary for ALPSA repair, BUT they must not be prominent. Prominent metal or hard PEEK anchors can destroy the humeral head cartilage (Chondrolysis). Use soft anchors or ensure knotless anchors are countersunk.
Rehabilitation
-
Phase 1 (0-6 weeks):
- Sling immobilization.
- Protect ER (usually restricted to 0 or 30 degrees).
- Pendulums.
-
Phase 2 (6-12 weeks):
- Regain ROM.
- Active assistive to Active.
- Scapular control.
-
Phase 3 (3-6 months):
- Strengthening.
- Proprioception.
- Return to sport at 6 months (contact sports).
Protocol is identical to Bankart Repair.
Return to Sport Criteria
- Full, pain-free Range of Motion.
- Symmetrical strength (ER/IR/Abduction).
- Psychological readiness (ACL-RSI or similar scale).
- Completion of sport-specific training drills.
- No apprehension in apprehension position.
Sling Removal Criteria
- No pain at rest.
- Control of scapula.
- Ability to perform ADLs waist level.
Contact Progression
- 4 months: Non-contact drills (passing, catching).
- 5 months: Controlled contact (pad work).
- 6 months: Unrestricted contact (tackling).
Prognosis
- Recurrence Rate: With proper mobilization, results equal Bankart repair (5-10% recurrence).
- Without Mobilization: Recurrence rates are significantly higher.
- Return to Sport: High (greater than 85%).
- Arthritis: Long term risk exists due to initial cartilage damage (GLAD component often co-exists).
Prognostic Factors
- Age: Younger patients have higher recurrence risk.
- Bone Loss: Significant glenoid bone loss (greater than 20%) requires Latarjet.
- Hyperlaxity: Beighton score greater than 5 increases failure risk.
- Sport: Collision athletes have higher recurrence.
Evidence Base
Original Description
- Described the ALPSA lesion
- Highlighted the intact periosteal sleeve
- Noted medial healing and loss of bumper
Recurrence Rates
- Compared Bankart vs ALPSA repair outcomes
- Found no significant difference in recurrence IF ALPSA is mobilized
- Emphasized technical factors
Biomechanics
- Cadaver study of capsular failure
- IGHL strain properties
- Showed that medialization significantly reduces restraint to anterior translation
Chronic ALPSA
- Evolution of the lesion over time
- Described the 'single anterior instability lesion' becoming ALPSA with recurrence
- Associated with bone remodelling
Arthroscopic Technique
- Famous paper on instability failures
- Identified Bone Loss and ALPSA as risk factors
- Advocated for 'Address the Tissues and the Bone'
Viva Scenarios
Practice these scenarios to excel in your viva examination
Scenario 1: MRI Interpretation
"You are shown an MRI of a 19-year-old rugby player. There is a dark lump of tissue sitting on the anterior glenoid neck, medial to the rim. What is this and how does it differ from a Bankart?"
Scenario 2: Intra-operative Surprise
"You are scoping a 'Bankart'. You enter the joint and the labrum looks 'healed' but the shoulder is loose (Drive-through sign positive). Probe shows it is firmly attached but medial. What do you do?"
Scenario 3: The Revision
"A patient had a stabilisation elsewhere and re-dislocated. MRI shows the anchors are in the glenoid neck, not the face. The labrum is still medial. Why did it fail?"
Scenario 4: CT Arthrography Interpretation
"You are shown a CT arthrography of a 26-year-old footballer with chronic anterior instability. Contrast tracks medially along the glenoid neck with the labrum. What is the diagnosis and how does this affect your surgical planning?"
MCQ Practice Points
Pathology
Q: What structure remains intact in an ALPSA lesion that is torn in a Bankart? A: The anterior scapular periosteum.
Terminology
Q: What does ALPSA stand for? A: Anterior Labroligamentous Periosteal Sleeve Avulsion.
Surgical Technique
Q: What is the most critical step in ALPSA repair? A: Mobilization (Takedown) of the labrum.
Classification
Q: A chronic, fixed ALPSA lesion is classified as: A: Neviaser Type II.
MRI Diagnosis
Q: What MRI finding differentiates ALPSA from Bankart lesion? A: Medially displaced labrum with intact periosteal sleeve (labrum lies against glenoid neck rather than at rim).
Recurrence Risk
Q: Why do ALPSA lesions have higher recurrence rates if not properly mobilized? A: The medialized labrum heals in a non-anatomic position, failing to restore the labral bumper effect and capsular tension.
Australian Context
- Prevalence: Highly common in the Australian contact sport population (Rugby, AFL).
- Practice: ALPSA recognition is a standard of care expectation for Orthopaedic candidates. "In situ" repair is considered a technical error.
High-Yield Exam Summary
Diagnosis
- •History: Recurrent Instability
- •MRI: Medialized Labrum
- •MRI: Intact Periosteal Sleeve
- •Sign: Drive-through positive
Management
- •Arthroscopic Repair is Gold Standard
- •CRITICAL: Mobilize/Takedown lesion
- •Fix to Glenoid Face (Not neck)
- •Restore Bumper Effect
Key Differentiators
- •Bankart: Torn Periosteum, Floating
- •ALPSA: Intact Periosteum, Medialized
- •GLAD: Cartilage defect
- •Perthes: Undisplaced sleeve