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HAGL Lesions

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HAGL Lesions

Comprehensive guide to Humeral Avulsion of the Glenohumeral Ligament (HAGL), including the MRI 'J Sign', West Point Classification, and surgical management.

complete
Updated: 2025-12-20
High Yield Overview

HAGL LESIONS

The Other Side of Instability

Incidence9% of Instability
SignJ Sign
RecurrenceHigh if Missed
RepairOpen/Scope

West Point Classification

Anterior
PatternAvulsion of Anterior Band IGHL (93%)
Treatment
Posterior
PatternAvulsion of Posterior Band IGHL (7%)
Treatment
Bony (BHAGL)
PatternAvulsion with Bone Fragment (20%)
Treatment
Floating
PatternBipolar avulsion (Glenoid + Humerus)
Treatment

Critical Must-Knows

  • HAGL stands for Humeral Avulsion of the Glenohumeral Ligament.
  • It is a cause of recurrent instability often missed on standard MRI.
  • The 'J Sign' on coronal oblique MRI is pathognomonic.
  • It is frequently associated with subscapularis tears (Open repair addresses both).
  • Failure to recognize HAGL leads to failed Bankart repairs.

Examiner's Pearls

  • "
    In a patient with instability but NO Bankart lesion, look for the HAGL.
  • "
    Arthroscopic repair is technically demanding (70-degree scope helps).
  • "
    Open repair is the historical gold standard.

Clinical Imaging

Imaging Gallery

The Missed Diagnosis

The Scenario

"Bankart Negative Instability" A patient presents with recurrent anterior instability. MRI report says "No labral tear". DO NOT dismiss as "functional" or "MDI". Review the MRI specifically for the J Sign. The IGHL may be stripped from the humerus.

The Consequences

Failed Stabilization. Performing a Bankart repair on a HAGL patient will FAIL. You are tightening the wrong side of the hammock. The instability persists.

FeatureBankart LesionHAGL Lesion
LocationGlenoid (Labrum)Humerus (Neck)
PathologyLabral detachmentLigament avulsion
Incidence80-90%2-9%
ApproachAnterior ScopeAnterior Open / 5 O'clock Scope
Mnemonic

APEWest Point Classification

A
Anterior
Anterior HAGL (Most common)
P
Posterior
Posterior HAGL (Reverse HAGL usually glenoid, but PHAGL exists)
E
Extra
Extra-ligamentous (Bony HAGL involved)

Memory Hook:The APE hung from the humerus.

Mnemonic

JUMPMRI Signs

J
J Sign
Inferior droop of IGHL
U
U Shape
Instead of U shape axillary pouch, it looks like J
M
Medial
Medial extravasation of contrast
P
Pouch
Double Axillary Pouch Sign

Memory Hook:JUMP to the diagnosis.

Mnemonic

SHAGAssociations

S
Subscapularis
Subscapularis tears (Anterior HAGL)
H
Hill-Sachs
Hill-Sachs Lesion (Common)
A
Axillary
Axillary Nerve (Risk in repair)
G
Glenoid
Glenoid bone loss (Less common than Bankart)

Memory Hook:SHAG lesions (Shoulder HAGL).

Overview

Definition

A HAGL lesion (Humeral Avulsion of the Glenohumeral Ligament) is a traumatic detachment of the inferior glenohumeral ligament (IGHL) complex from its insertion on the anatomical neck of the humerus.

This lesion was once considered rare or a "lesion of exclusion," but modern MRI techniques and arthroscopic vigilance have revealed it to be a significant contributor to recurrent instability, particularly in contact athletes. It represents the "humeral side" of the capsular failure spectrum.

While the classic Bankart lesion involves the glenoid side of the IGHL complex, the HAGL lesion represents failure at the humeral insertion. It is a potent cause of anterior instability because the "hammock" of the IGHL is disabled.

Pathophysiology and Mechanisms

IGHL Complex

  • Anterior Band: The primary restraint to anterior translation in abduction and external rotation (ABER). Requires restoration.
  • Posterior Band: Restraint to posterior translation in flexion/internal rotation.
  • Axillary Pouch: The hammock between the bands.

The IGHL inserts onto the humeral neck, just below the articular surface.

This insertion is the "weak link" in HAGL lesions.

Humeral Insertion Anatomy

  • Location: Anatomical neck, approximately 1-2 cm from articular margin.
  • Width: The IGHL insertion spans 2-3 cm along the humeral neck.
  • Blood Supply: Branches of anterior circumflex humeral artery.
  • Surgical Relevance: Anchors must be placed at the anatomical footprint for restoration.

Understanding this anatomy is critical for proper anchor placement during repair.

Mechanism of Injury

  • Hyper-abduction and External Rotation.
  • Similar mechanism to Bankart, but the failure point is humeral.
  • Studies suggest HAGL may occur when the rotator cuff is intact (stiff humerus) or with concurrent subscapularis strain.
  • Biomechanical Consequence: Removes the anterior check-rein. The head translates anteriorly without resistance.

The axillary pouch loses its tension, failing to cradle the head in abduction.

Biomechanical Consequences

  • Hammock Failure: The IGHL functions as a hammock supporting the humeral head in ABER. HAGL removes this support.
  • Stability Ratio: The force required to cause dislocation is reduced by up to 50%.
  • Associated Injuries: Often occurs with subscapularis strain due to shared anterior structures.
  • Healing Potential: Unlike Bankart, HAGL lesions have limited spontaneous healing due to retraction and synovial environment.

Spectrum of Capsular Failure

Understanding the continuum is essential for diagnosis:

  • Bankart: Glenoid-side labral avulsion.
  • ALPSA: Medialized glenoid-side failure with intact periosteum.
  • HAGL: Humeral-side avulsion of the ligament proper.
  • Floating IGHL: Bipolar failure - both ends detached (most severe).

Proper identification of the specific failure pattern directs appropriate surgical management.

Classification Systems

West Point Classification (Bui-Mansfield)

  • Type A: Anterior HAGL (93%) - Avulsion of anterior band from humeral neck.
  • Type B: Posterior HAGL (7%) - Avulsion of posterior band (rare).
  • Type C: Floating AIGHL (Bipolar - Glenoid and Humeral) - Both ends detached.
  • Bony HAGL (BHAGL): Any of the above with a bone fragment from the humerus (20%).

BHAGL is often easier to heal (bone-to-bone) if fixed.

The floating type represents a devastating loss of all inferior restraints.

HAGL Variants

  • Anterior HAGL: Most common (93%). Affects anterior band of IGHL.
  • Posterior HAGL (PHAGL): Rare (7%). Associated with posterior instability.
  • Bony HAGL (BHAGL): Avulsion fracture from humeral neck. Visible on X-ray.
  • Floating IGHL: Complete detachment from both glenoid and humerus.
  • Combined HAGL-Bankart: Dual pathology - both sides affected.

Prognostic Implications

TypeHealing PotentialRepair Difficulty
Soft tissue HAGLModerateStandard
BHAGLGood (bone-bone)May be easier
FloatingPoorMost complex
CombinedVariableDual repair needed

History

Presentation

  • Event: Traumatic dislocation (often high energy).
  • Recurrence: Instability with overhead activity.
  • Pop: Patient may report a pop deep in the axilla.
  • Failed Surgery: Previously had a Bankart repair but instabilty persists.

Always question the diagnosis in failed instability repairs.

Examination

Physical Exam

  • Apprehension: Positive in ABER.
  • Relocation: Positive (Pain/Apprehension relief).
  • Subscapularis: Check Lift-off / Belly-press (often associated tear).
  • Axillary Nerve: ALWAYS verify sensation/deltoid function.

Clinically indistinguishable from Bankart instability without imaging.

The Apprehension test is sensitive but not specific for the type of lesion.

Imaging: MRI

The J Sign

Coronal Oblique T2. Normally, the axillary pouch forms a "U" shape under the humeral head. In a HAGL, the lateral attachment is gone. The IGHL falls inferiorly against the chest wall/glenoid, forming a deep "J" shape.

MRI Signs

  • J Sign: Retraction of the IGHL.
  • Double Axillary Pouch: Can mimic a double pouch if fluid extravasates.
  • Extravasation: Contrast (Arthrogram) leaking laterally down the humerus (not typical, usually medial leak for Bankart - wait, HAGL leaks medially too? No, HAGL leaks from humeral side, so extravasation extends laterally/inferiorly along the shaft).
  • Edema: Bone edema at the humeral neck (acute).

Always evaluate the integrity of the capsule on the humeral side.

The "Fake" HAGL

  • The IGHL is lax in adduction.
  • Folds can look like tears.
  • Look for fluid extending below the teres minor or frank detachment.
  • MR Arthrography (MRA) significantly increases sensitivity.

A high index of suspicion is required to differentiate artifact from pathology.

Management Strategy

Decision Making

  • Non-Operative: Low demand, elderly. (Immobilization in internal rotation? No, ER brace logic applies to Bankart, unlikely to help HAGL retraction).
  • Operative: Young, Athletes, Recurrent Instability.
  • Choice of Approach:
    • Open: Gold Standard. Associated Subscap tear. Easier to find and fix.
    • Arthroscopic: Technically difficult. Needs 70-degree scope and accessory portals (5 o'clock).

The choice depends largely on surgeon experience and presence of subscapularis pathology.

Surgical Pearl

The 5 O'Clock Portal For arthroscopic repair, a standard anterior portal is too high. Establish a low anterior portal (5 o'clock) to access the inferior pouch. Use localized cutaneous nerve protection.

Timing

  • Acute repair (less than 3 weeks) allows mobilization of tissue.
  • Chronic HAGL may be scarred and retracted, making arthroscopic repair impossible.

Chronic cases often require open release of the capsule to mobilize it.

Surgical Considerations

Open Repair (L-shaped Incision)

  • Approach: Deltopectoral.
  • Subscapularis: L-shaped tenotomy or Takedown (lower half) to access the deep capsule.
  • Identification: Humerus is externally rotated to reveal the avulsed ligament.
  • Fixation: Suture anchors (double loaded) placed in the humeral neck.
  • Closure: Repair Subscapularis robustly.
  • Pros: Visualization, protect Axillary nerve.
  • Cons: Subscapularis morbidity.

The L-shaped tenotomy provides excellent exposure of the inferior pouch.

Arthroscopic Repair

  • Portals: Standard post/ant. Accessory low anterior (5 o'clock) or trans-subscapularis portal.
  • Technique:
    1. View from Anterior portal (or high anterolateral).
    2. Prepare bone bed on humerus (difficult angle).
    3. Pass sutures through ligament first (outside-in or penetrator).
    4. Fix to humerus.
  • Risks: Axillary nerve (at 6 o'clock) is dangerously close to the 5 o'clock portal.

Visualization is key; switch portals frequently.

Surgical Pearl

The Axillary Nerve During both open and arthroscopic repair, the axillary nerve is at risk. It runs approx 1-2cm inferior to the glenoid at the 6 o'clock position. In open surgery, bluntly dissect and visualize it if unsure. In scopy, keep portals high (above equator) or use safe zones (5 o'clock needs care).

Complications

Intraoperative Complications

  • Axillary Nerve Injury: The most feared complication. The nerve runs approximately 1-2 cm inferior to the capsule at the surgical neck. Both open and arthroscopic approaches place it at risk.
  • Subscapularis Damage: During open approach, aggressive retraction or inadequate repair can lead to subscapularis insufficiency.
  • Anchor Malposition: Anchors placed too high (cartilage) or too low (shaft) fail to restore anatomy.
  • Inadequate Mobilization: Failure to release scarred tissue in chronic cases leads to insufficient repair.

Early Postoperative Complications

  • Recurrent Instability: If repair fails or associated Bankart/bone loss missed.
  • Stiffness: Specifically loss of External Rotation (capsular tightening).
  • Infection: Standard surgical risk.
  • Hematoma: Risk increased with subscapularis dissection.

Late Complications

  • Chronic Instability: Often due to missed pathology or inadequate repair.
  • Subscapularis Failure: Weakness in internal rotation and lift-off tests.
  • Glenohumeral Arthritis: Long-term sequela of recurrent instability.
  • Anchor Migration: Bioabsorbable anchor osteolysis or metal anchor loosening.
Complication Prevention

Axillary Nerve Protection:

  • In open surgery, identify and protect the nerve before capsular repair.
  • In arthroscopy, keep instruments above the equator when possible.
  • The 5 o'clock portal should be established with care using blunt trocar.
  • Never pass needles blindly in the inferior pouch.

Rehabilitation

  • Phase 1 (0-6 weeks):

    • Sling immobilization.
    • Limit External Rotation (to protect Subscap and HAGL repair) - usually less than 30 degrees.
    • Pendulums.
  • Phase 2 (6-12 weeks):

    • Active Range of Motion.
    • Progress ER.
  • Phase 3 (3-6 months):

    • Strengthening.
    • Return to sport at 6 months.

    Note: Rehabilitation is often slower than Bankart due to Subscapularis precautions (if open).

Prognosis

Outcomes by Repair Type

  • Open Repair Success Rate: Greater than 90% stability restoration.
  • Arthroscopic Repair: Variable (75-90%), highly surgeon and case dependent.
  • BHAGL Repair: Excellent outcomes due to bone-to-bone healing.
  • Floating IGHL Repair: Most challenging; may require combined open approach.

Functional Outcomes

  • Return to Sport: 85-95% return to pre-injury level activity.
  • Time to Return: Typically 6-9 months (longer than isolated Bankart).
  • Range of Motion: External rotation may be slightly reduced (5-10 degrees).
  • Strength: Full recovery expected if subscapularis healed properly.

Prognostic Factors

FactorPositiveNegative
TimingAcute (less than 3 weeks)Chronic (greater than 6 months)
TypeBHAGLFloating IGHL
AssociatedIsolatedCombined Bankart/Bone loss
AgeYounger patientOlder with tissue degeneration

Long-Term Considerations

  • Missed HAGL: 100% failure rate of Bankart repair when HAGL is present.
  • Arthritis Risk: Ongoing instability leads to accelerated osteoarthritis.
  • Re-dislocation: Low (less than 5%) if properly repaired and rehabilitated.

Evidence Base

The HAGL Lesion

4
Wolf et al. • Arthroscopy (1995)
Key Findings:
  • Described the Humeral Avulsion of Glenohumeral Ligament
  • Identified incidence of 9% in instability patients
  • Coined the term HAGL
Clinical Implication: Foundation paper establishing HAGL as an entity.

Open vs Arthroscopic Repair

3
Rhee et al. • Am J Sports Med (2006)
Key Findings:
  • Comparison of open vs arthroscopic outcomes
  • Both groups had significant improvement
  • No significant difference in recurrence
  • Arthroscopic group had less postoperative pain
Clinical Implication: Arthroscopic repair is viable in expert hands.

The 6th Lesion

4
Bokor et al. • JBJS Br (1999)
Key Findings:
  • Analyzed failed Bankart repairs
  • Found unrecognized HAGL lesions in a subset of failures
  • Emphasized the need for diagnostic vigilance
Clinical Implication: Always verify the humeral attachment.

MRI Sensitivity

3
Bui-Mansfield et al. • Radiology (2002)
Key Findings:
  • Proposed West Point Classification
  • MRI Sensitivity for HAGL is approx 95% with arthrogram
  • Without arthrogram, sensitivity drops to 50%
Clinical Implication: Order an MRA if HAGL is suspected.

Biomechanical Analysis

5
Pouliart et al. • JSES (2006)
Key Findings:
  • Cadaveric creation of HAGL
  • Resulted in significant anterior translation
  • Repair restored kinematics to baseline
Clinical Implication: HAGL is a mechanical instability requiring mechanical repair.

Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: The Failed Bankart

EXAMINER

"A 22-year-old rugby player returns 1 year after 'successful' Bankart repair with recurrent instability. He says it never felt tight. What are your thoughts?"

EXCEPTIONAL ANSWER
Recurrent instability can be due to: Trauma (new injury), Technical failure (anchors loose), Bone Loss (Glenoid/Hill-Sachs), or Missed Pathology (HAGL, ALPSA). I would obtain a CT for bone loss and an MRI Arthrogram to check the repair and capsular integrity.
KEY POINTS TO SCORE
Differential of recurrence
Bone loss exclusion
Missed HAGL
COMMON TRAPS
✗Assuming just 'bad tissue'
✗Ignoring the humeral side
LIKELY FOLLOW-UPS
"You see a J-sign on MRI. How do you treat?"
"Open HAGL repair via deltopectoral approach."
VIVA SCENARIOStandard

Scenario 2: The Acute Dislocation

EXAMINER

"A 30-year-old male dislocates his shoulder. Reduced in ED. Post-reduction X-ray shows a small fleck of bone lateral to the humeral head. What is this?"

EXCEPTIONAL ANSWER
This is a Bony HAGL (BHAGL) lesion - an avulsion of the IGHL from its humeral attachment with a bone fragment. The fleck lateral to the humeral head on X-ray is pathognomonic for BHAGL. This represents significant anterior instability and warrants MRI to assess the full extent. Unlike soft tissue HAGL, BHAGL has better healing potential due to bone-to-bone union when anatomically reduced and fixed. Treatment typically involves open or arthroscopic repair with suture anchor fixation back to the humeral neck.
KEY POINTS TO SCORE
BHAGL identification
X-ray signs
Instability correlation
COMMON TRAPS
✗Calling it a loose body in the joint
✗Missing the significance
LIKELY FOLLOW-UPS
"Does this heal better than soft tissue HAGL?"
"Yes, bone-to-bone healing is generally more reliable."
VIVA SCENARIOStandard

Scenario 3: The Subscapularis Deficit

EXAMINER

"You perform an open Latarjet. During exposure, you notice the subscapularis tendon is partially torn inferiorly. What should you look for?"

EXCEPTIONAL ANSWER
You must look for an associated Anterior HAGL lesion. The 'L-shaped' rupture pattern of the subscapularis tendon often extends into the anterior-inferior capsule, creating a HAGL lesion. This is known as a 'Subscapularis-HAGL Complex' and is particularly common in high-energy anterior dislocations. During the Latarjet procedure, visualize the entire anterior-inferior capsule before proceeding with coracoid transfer. If HAGL is present, it must be repaired directly to the humeral neck using suture anchors before or after the bone block. Failure to address the HAGL leads to persistent instability despite adequate glenoid bone stock reconstruction.
KEY POINTS TO SCORE
Subscapularis/HAGL association
Intra-operative vigilance
Combined repair
COMMON TRAPS
✗Ignoring the capsule
✗Only fixing the bone block
LIKELY FOLLOW-UPS
"How does this change your rehab?"
"Slower ER progression to protect the subscapularis."

MCQ Practice Points

Pathognomonic Sign

Q: The 'J Sign' on coronal oblique MRI indicates: A: HAGL Lesion (Humeral Avulsion of Glenohumeral Ligament).

Incidence

Q: What percentage of anterior instability cases involve a HAGL lesion? A: Approximately 9%.

Association

Q: Which tendon tear is most commonly associated with an anterior HAGL? A: Subscapularis.

Classification

Q: A floating glenohumeral ligament (bipolar avulsion) corresponds to which West Point type? A: Type C.

Failed Bankart

Q: What is the most likely cause of a failed Bankart repair in a patient with persistent instability and no evidence of bone loss? A: Missed HAGL lesion - performing a glenoid-side repair when the lesion is on the humeral side leads to 100% failure.

Australian Context

  • Epidemiology: Seen frequently in contact sports (Rugby League, AFL) due to high-energy collisions.
  • Practice: Open repair remains popular among shoulder surgeons for reliability, though arthroscopic techniques are increasing.

High-Yield Exam Summary

Diagnosis

  • •History: Traumatic Dislocation
  • •Sign: Apprehension Positive
  • •MRI: J Sign (Inferior droop)
  • •MRI: Double Axillary Pouch
  • •X-ray: BHAGL Fleck

Classification (West Point)

  • •Type A: Anterior (93%)
  • •Type B: Posterior (7%)
  • •Type C: Floating (Bipolar)
  • •BHAGL: Bony Avulsion

Management

  • •Non-op: Elderly/Low demand
  • •Open: Gold Standard (Subscap approach)
  • •Scope: Technically demanding (5 o'clock portal)
  • •Rehab: Protect ER for 6 weeks
Quick Stats
Reading Time53 min
Related Topics

ALPSA Lesions

Axillary Nerve Anatomy

Quadrangular & Triangular Spaces

Subscapularis Anatomy