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Flexor Tendon Rehabilitation

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Flexor Tendon Rehabilitation

Evidence-based rehabilitation protocols for flexor tendon repairs across all zones, including early active mobilization, passive motion, and place-and-hold techniques

complete
Updated: 2025-12-24
High Yield Overview

FLEXOR TENDON REHABILITATION

Early Active Motion | Zone-Specific Protocols | Preventing Rupture vs Adhesions

4-6 weeksCritical healing period
10-15%Rupture risk with EAM
Zone 2Most challenging rehabilitation
6 weeksProtected motion period

REHABILITATION PROTOCOLS

Passive Motion
PatternKleinert/Duran - Lower rupture risk
TreatmentZones 1-2, low compliance
Place-and-Hold
PatternIntermediate tension
TreatmentBalanced approach
Early Active Motion
PatternBest outcomes, higher rupture risk
TreatmentCompliant patients, strong repair

Critical Must-Knows

  • Early mobilization superior to immobilization - reduces adhesions without increasing rupture
  • Zone 2 (no man's land) requires most careful rehabilitation - FDS and FDP in flexor sheath
  • Kleinert protocol: rubber band traction with passive extension, active flexion blocked
  • EAM protocols: Controlled active flexion from day 3-5, superior functional outcomes
  • Rupture vs adhesion balance: Too conservative = stiffness, too aggressive = rupture

Examiner's Pearls

  • "
    Duran protocol uses passive flexion/extension exercises without rubber band traction
  • "
    Place-and-hold allows passive positioning, then patient actively holds position
  • "
    Strickland criteria: Good result = greater than 70% TAM compared to opposite hand
  • "
    Week 6-8: Transition to unrestricted active motion and light resistance

Clinical Imaging

Imaging Gallery

flexor-tendon-rehabilitation imaging 1
Click to expand
Clinical imaging for flexor-tendon-rehabilitationCredit: Wirtz C, Leclère FM, Oberfeld E, Unglaub F, Vögelin E. A retrospective analysis of controlled active motion (CAM) versus modified Kleinert/Duran (modKD) rehabilitation protocol in flexor tendon repair (zones I and II) in a single center. Arch Orthop Trauma Surg. 2022 Aug 17;143(2):1133-1141. PMC9925601. via web-sourced (CC-BY)
flexor-tendon-rehabilitation imaging 2
Click to expand
Clinical imaging for flexor-tendon-rehabilitationCredit: Wirtz C, Leclère FM, Oberfeld E, Unglaub F, Vögelin E. A retrospective analysis of controlled active motion (CAM) versus modified Kleinert/Duran (modKD) rehabilitation protocol in flexor tendon repair (zones I and II) in a single center. Arch Orthop Trauma Surg. 2022 Aug 17;143(2):1133-1141. PMC9925601. via web-sourced (CC-BY)

Critical Flexor Tendon Rehabilitation Exam Points

Zone 2 Challenge

No man's land complexity. Both FDS and FDP in restrictive fibro-osseous sheath. High adhesion risk without early motion, rupture risk with aggressive mobilization.

Timing Principles

0-6 weeks = protected motion. Weeks 6-8 = transition. Week 8+ = strengthening. Critical collagen remodeling occurs weeks 3-8.

Protocol Selection

Patient compliance determines protocol. Non-compliant = passive motion only. Reliable patient + strong repair = early active motion for superior outcomes.

Outcome Measurement

Total Active Motion (TAM). Excellent = greater than 85%, Good = 70-84%, Fair = 50-69%, Poor = under 50% of normal. Strickland criteria.

At a Glance

Flexor tendon rehabilitation balances the competing risks of adhesion formation (too conservative) versus rupture (too aggressive) during the critical 4-6 week healing period. Zone 2 (no man's land) presents the greatest challenge because both FDS and FDP traverse the restrictive fibro-osseous sheath, creating high adhesion risk. Three main protocols exist: passive motion (Kleinert/Duran) with lowest rupture risk for non-compliant patients; place-and-hold providing intermediate tension; and early active motion (EAM) offering superior functional outcomes with 10-15% rupture risk in compliant patients with strong repairs. EAM begins days 3-5 post-repair using a dorsal blocking splint (wrist 20-30° flexion, MPs 50-70°), differential FDS/FDP gliding exercises, and tenodesis movements. Outcomes are measured using Total Active Motion (TAM) via Strickland criteria: Excellent over 85%, Good 70-84%, Fair 50-69%, Poor under 50% of normal. Week 6-8 transitions to unrestricted active motion, with strengthening commencing after week 8 once collagen remodeling provides adequate repair strength.

Mnemonic

FIRMFlexor Tendon Healing Phases

F
Fibroplasia
Days 5-21: Fibroblasts proliferate, collagen deposition
I
Inflammatory
Days 0-5: Hematoma, inflammatory cells, weak repair
R
Remodeling
Weeks 6-12: Collagen reorganization, strength increases
M
Maturation
Months 3-12: Final collagen alignment, maximum strength

Memory Hook:FIRM grip requires all 4 healing phases - Inflammatory foundation, Fibroplasia builds strength, Remodeling refines, Maturation maintains!

Mnemonic

PLATEEarly Active Motion Protocol Components

P
Protected flexion
Dorsal blocking splint 20-30° wrist flexion, MPs 50-70°
L
Limited extension
IP joints extended only to neutral, no hyperextension
A
Active differential gliding
FDS vs FDP isolated exercises
T
Tenodesis exercise
Passive wrist motion with finger flexion/extension
E
Early motion
Start day 3-5 post-repair, 10-12 repetitions hourly

Memory Hook:Put rehabilitation on a PLATE - Protected position, Limited extension, Active gliding, Tenodesis, Early start!

Mnemonic

RAFTComplications Recognition

R
Rupture
Flexion lag, sudden loss active flexion, palpable gap, 10-15% EAM
A
Adhesions
Limited passive ROM, plateau after week 8, needs tenolysis
F
Flexion contracture
Cannot extend passively, static progressive splinting
T
Tenolysis timing
3-6 months optimal, local anesthesia for active motion check

Memory Hook:Build a RAFT to rescue failed rehab - Rupture needs re-repair, Adhesions need lysis, Flexion contracture needs splinting, Timing is 3-6 months!

Overview and Epidemiology

Flexor tendon rehabilitation is one of the most demanding challenges in hand therapy. The balance between preventing adhesions (requiring early motion) and avoiding rupture (requiring protection) defines successful outcomes. Zone 2 injuries, where both FDS and FDP tendons run through the restrictive fibro-osseous sheath, present the greatest rehabilitation challenge.

Epidemiology of Flexor Tendon Injuries

Incidence: Flexor tendon lacerations represent approximately 15-20% of all hand injuries requiring surgical intervention. Annual incidence is estimated at 15-20 cases per 100,000 population in developed countries.

Demographics: Predominantly affects working-age males (18-45 years, 75% of cases) engaged in manual occupations including construction, manufacturing, food service, and agriculture. Power tools, knives, and glass are the most common mechanisms.

Zone Distribution: Zone 2 (no man's land) accounts for 40-45% of flexor tendon injuries, Zone 1 represents 15-20%, Zone 3 approximately 15%, Zone 4 is 10-15%, and Zone 5 accounts for 15-20%.

Socioeconomic Impact: Flexor tendon injuries result in average work loss of 3-6 months for manual laborers. Direct medical costs range from $15,000-$30,000 per injury including surgery, therapy, and rehabilitation. Indirect costs from lost productivity exceed $40,000 per case in working-age individuals.

Historical Context and Evolution

Traditional Immobilization (Pre-1970s): Complete immobilization for 3-4 weeks resulted in severe adhesions and poor functional outcomes. Verdan's classic studies demonstrated stiffness rates exceeding 60%, leading to abandonment of this approach.

Passive Motion Era (1970s-1980s): Kleinert (1967) and Duran (1975) protocols introduced controlled passive motion, dramatically improving outcomes (60-70% good-excellent results) while minimizing rupture risk to 3-5%. This represented a paradigm shift in flexor tendon rehabilitation philosophy.

Early Active Motion (1990s-Present): Advances in core suture techniques, particularly multi-strand repairs (4-6 strands), provided biomechanical strength enabling earlier active motion. Strickland, Trumble, and others demonstrated superior gliding and functional outcomes (75-85% good-excellent results) despite slightly higher rupture rates (10-15%).

Anatomy

Flexor Tendon Anatomy

Zone Classification (Verdan): The flexor tendon system is divided into five anatomical zones based on structural and functional considerations. Zone 1 extends from the FDS insertion to the fingertip (FDP only). Zone 2 (no man's land) runs from the A1 pulley to the FDS insertion - both FDS and FDP within the restrictive fibro-osseous sheath with five annular pulleys (A1-A5). Zone 3 encompasses the lumbrical origin area in the palm. Zone 4 is the carpal tunnel region. Zone 5 represents the forearm muscle-tendon junction.

Pulley System: The flexor sheath contains annular pulleys (A1-A5) and cruciate pulleys (C1-C3). The A2 (proximal phalanx) and A4 (middle phalanx) pulleys are biomechanically critical, preventing bowstringing during finger flexion. Loss of either pulley results in significant mechanical disadvantage and reduced grip strength.

Blood Supply: Flexor tendons receive nutrition through two mechanisms: intrinsic vascular supply (longitudinal vincular vessels) and synovial fluid diffusion. Zone 2 has relatively poor vascularity, contributing to healing challenges.

Pathophysiology

Tendon Healing Biology

Intrinsic vs Extrinsic Healing: Tendon healing occurs through two competing processes. Intrinsic healing involves tenocytes from the tendon ends proliferating and producing organized collagen fibers that maintain tendon gliding. Extrinsic healing recruits fibroblasts from surrounding synovial sheath and peritendinous tissues, producing disorganized scar tissue that creates adhesions. Early controlled motion promotes intrinsic healing while suppressing excessive extrinsic healing.

Healing Phases:

  • Inflammatory Phase (Days 0-5): Hematoma formation, inflammatory cell infiltration, early fibroblast migration. Repair site is weakest, dependent entirely on suture strength.
  • Fibroplastic Phase (Days 5-21): Fibroblast proliferation, collagen type III deposition, neovascularization. Tensile strength increases but remains low.
  • Remodeling Phase (Weeks 6-12): Collagen type I replaces type III, fiber alignment along stress lines, strength increases significantly. Collagen cross-linking matures.
  • Maturation Phase (Months 3-12): Final collagen reorganization, maximum tensile strength achieved (approximately 70-80% of normal tendon).

Biomechanical Considerations: The repair construct strength depends on core suture technique (90% of strength) and epitenon suture (10-20% additional strength). Multi-strand repairs (4-6 strands) provide 60% greater strength than 2-strand techniques, enabling early active motion protocols. Gap formation at the repair site occurs most commonly during weeks 1-3 when tension exceeds healing tissue strength.

Biological Healing

  • Intrinsic healing: Tenocytes from tendon ends (desired)
  • Extrinsic healing: Fibroblasts from synovial sheath (adhesions)
  • Motion benefits: Promotes intrinsic over extrinsic healing
  • Collagen alignment: Stress improves fiber organization

Biomechanical Factors

  • Repair strength: Increases weeks 3-12
  • Gap formation: Greatest risk weeks 1-3
  • Core suture: Provides 90% of repair strength
  • Epitenon suture: Adds 10-20% strength, smooths surface

Classification

Rehabilitation Protocol Classification

Flexor Tendon Rehabilitation Protocols

ProtocolMechanismRupture RiskIndications
ImmobilizationNo motion for 3-4 weeksLow (2%)Historical only - poor outcomes
Passive Motion (Kleinert)Rubber band traction, active extensionLow (3-5%)Non-compliant patients, 2-strand repairs
Passive Motion (Duran)Therapist-guided passive ROMLow (3-5%)Supervised therapy available
Place-and-HoldPassive placement, active holdModerate (6-8%)Moderate compliance, 3-strand repairs
Early Active Motion (EAM)Controlled active flexion from day 3-5Higher (10-15%)Compliant patients, 4+ strand repairs

Splinting Techniques

Controlled active motion (CAM) protocol splint configuration
Click to expand
Controlled active motion (CAM) protocol splint configuration. Left panel: lateral view showing white dorsal blocking splint with blue velcro straps securing wrist in slight extension (20°). Right panel: side view demonstrating MCP joints positioned in 30° flexion while allowing full IP joint extension. This splint configuration permits active flexion with controlled extension within safe limits.
Modified Kleinert/Duran protocol splint configuration
Click to expand
Modified Kleinert/Duran protocol splint configuration. Lateral view showing dorsal blocking splint with wrist maintained in flexion and elastic band/traction system. The elastic band provides passive flexion while allowing active extension within the protective arc of the dorsal blocking splint. This classic passive mobilization approach reduces tendon rupture risk while preventing adhesion formation.

Zone Classification (Verdan)

Flexor Tendon Zones

ZoneLocationStructuresRehabilitation Challenge
Zone 1Distal to FDS insertionFDP onlyGood prognosis
Zone 2A1 pulley to FDS insertionFDS and FDP in sheathHighest - 'no man's land'
Zone 3Lumbrical originFDS/FDP, lumbricalsGood gliding space
Zone 4Carpal tunnel8 tendons, median nerveAdd nerve gliding
Zone 5ForearmMuscle-tendon junctionBest healing

Protocol Progression

Evolution of rehabilitation: Immobilization → Passive motion → Place-and-hold → Early active motion. Each advance in surgical technique (stronger repairs with more core sutures) enabled more aggressive rehabilitation protocols with superior functional outcomes.

Clinical Presentation

Patient Assessment for Rehabilitation

Post-operative Factors:

  • Repair quality: Multi-strand core sutures (4-6 strands) vs 2-strand techniques determine protocol safety
  • Associated injuries: Nerve injuries, fractures, or vascular compromise affect rehabilitation approach
  • Zone of injury: Zone 2 requires strictest protocols; Zone 1, 3, 5 permit faster progression
  • Tendon involvement: Single tendon (FDP or FDS) vs both tendons affects complexity

Patient Factors for Protocol Selection:

  • Compliance: Ability to follow complex instructions determines passive vs active protocol choice
  • Cognitive function: Elderly or cognitively impaired patients require simpler passive protocols
  • Motivation: Return-to-work timeline and functional goals influence rehabilitation intensity
  • Manual dexterity: Ability to don/doff splints and perform exercises independently

Signs of Successful Rehabilitation vs Complications

Normal Progression Indicators:

  • Progressive increase in active flexion range (5-10° improvement weekly in early phase)
  • Maintained passive extension to neutral without excessive force
  • Minimal pain with exercises (2-3/10 maximum)
  • No signs of infection or inflammation at repair site

Warning Signs Requiring Immediate Assessment:

  • Rupture: Sudden loss of active flexion, flexion lag appears (passive range intact, active flexion lost)
  • Excessive adhesions: Plateau in passive range before week 8, limited gliding
  • Flexion contracture: Progressive loss of passive extension capability
  • Complex regional pain syndrome: Disproportionate pain, edema, skin changes, temperature asymmetry

Investigations and Monitoring

Clinical Assessment Tools

Total Active Motion (TAM) Measurement:

  • Technique: Measure active PIPJ flexion plus active DIPJ flexion, subtract extension lag at both joints
  • Frequency: Weekly during weeks 0-6, biweekly weeks 6-12, monthly thereafter
  • Normal values: 260° for finger composite motion, compare to contralateral hand
  • Documentation: Photograph hand positions at each assessment for medicolegal records

Passive Range of Motion (PROM):

  • Therapist-measured maximum passive flexion and extension at each joint
  • Discrepancy between PROM and active ROM indicates either adhesions (both limited) or weakness (only active limited)
  • Isolated limitation suggests specific anatomical restriction (e.g., A2 pulley adhesion)

Differential Gliding Assessment:

  • FDS isolation: Block proximal phalanx, measure isolated PIPJ flexion
  • FDP isolation: Extend PIPJ fully, measure isolated DIPJ flexion
  • Reduced differential gliding indicates adhesions between FDS and FDP tendons

Imaging (Rarely Required)

Ultrasound: Dynamic assessment can demonstrate tendon gliding, gap formation, or adhesions. Useful when clinical examination is equivocal regarding rupture vs adhesions.

MRI: Reserved for complex cases with uncertain diagnosis. Can identify tendon discontinuity, extent of adhesions, or associated pathology (ligament injury, occult fracture).

Radiographs: Obtained if associated fracture suspected or to assess joint alignment if contracture develops.

Management

Protocol Selection Framework

The fundamental management decision is selecting the appropriate rehabilitation protocol based on repair strength, patient compliance, and functional goals. This choice balances functional outcomes against rupture risk.

Decision Algorithm:

  1. Assess repair strength: Multi-strand (4-6 core sutures) permits aggressive protocols; 2-strand requires conservative approach
  2. Evaluate patient compliance: Reliable patients with strong repairs are candidates for early active motion
  3. Consider zone of injury: Zone 2 requires strictest adherence; other zones permit faster progression
  4. Account for associated injuries: Nerve injuries, fractures, or multiple-digit involvement may necessitate modified protocols
📊 Management Algorithm
Hand-drawn medical sketchnote showing decision tree flowchart for flexor tendon rehabilitation protocol selection
Click to expand
Protocol selection algorithm based on repair strength, patient compliance, and injury zone. Balances functional outcomes against rupture risk.

Management Protocol Selection Matrix

Clinical ScenarioRecommended ProtocolExpected TAM OutcomeRupture Risk
Strong repair (4+ strands), compliant patient, Zone 2Early Active Motion (EAM)75-85% good-excellent (TAM over 180°)10-15%
Moderate repair (3 strands), moderate compliancePlace-and-Hold70-80% good-excellent (TAM 165-200°)5-8%
Weak repair (2 strands), poor compliance, elderlyPassive Motion (Kleinert/Duran)60-70% good-excellent (TAM 150-180°)3-5%

Primary Rehabilitation Approach

All flexor tendon repairs require structured rehabilitation programs. The specific protocol varies, but all share common principles:

Universal Principles:

  • Edema control: Elevation, compression wrapping, retrograde massage in first 2 weeks
  • Splint positioning: Dorsal blocking splint maintains wrist flexion (20-30°), MP flexion (50-70°), IP extension to neutral
  • Progressive loading: Gradual increase in tendon stress from passive to active to resistive
  • Therapist supervision: Weekly minimum during protected phase (weeks 0-6)
  • Patient education: Understanding rupture signs, importance of compliance, realistic outcome expectations

Monitoring Parameters:

  • Active and passive range of motion at each joint (PIPJ, DIPJ)
  • Pain scores (should remain under 3/10 during exercises)
  • Edema measurement (volumeter or circumference)
  • Functional grip strength (after week 8)
  • TAM calculation and comparison to normal side

Return to Activity Timeline:

  • Week 6-8: Light activities of daily living (ADLs) - eating, writing, grooming
  • Week 8-12: Unrestricted ADLs, light work activities
  • Week 12-16: Progressive strengthening, return to light manual work
  • Month 4-6: Full return to manual labor, contact sports

All patients require individualized protocol selection and close monitoring throughout the rehabilitation process to optimize functional outcomes while minimizing complication risk.

Tenolysis for Adhesions

Indications: Reserved for patients with persistent adhesions limiting function despite 3-6 months of intensive therapy. Optimal timing is 3-6 months post-repair (tendon healed but adhesions not yet matured).

Preoperative Assessment:

  • TAM typically less than 50% of normal despite therapy
  • Limited passive range of motion (confirms adhesions vs weakness)
  • Plateau in progress for 2-3 months minimum
  • Patient motivated for intensive post-tenolysis rehabilitation

Surgical Technique:

  • Performed under local anesthesia with sedation (permits intraoperative active motion)
  • Reopen original incision, identify tendon under flexor sheath
  • Complete release of adhesions from tendon to sheath
  • Preserve A2 and A4 pulleys (critical for biomechanics)
  • Patient actively flexes finger on operating table to confirm adequate release
  • Aim for full active flexion intraoperatively

Post-Tenolysis Rehabilitation:

  • Immediate early active motion protocol starting day 1
  • Aggressive therapy to prevent re-adhesion
  • Expected improvement: 30-50° TAM increase
  • Best results in young, motivated patients with isolated adhesions

Reconstruction for Rupture

Re-Repair (Under 2 weeks):

  • Feasible if recognized early
  • Debride frayed tendon ends
  • Perform revised 4-strand core suture
  • Use more conservative passive protocol post-operatively
  • Outcomes approach primary repair if tissue quality good

Two-Stage Reconstruction (After 6 weeks):

  • Stage 1: Silicone rod (Hunter rod) insertion to maintain tendon bed
  • Wait 3-6 months for passive tendon bed formation
  • Stage 2: Rod removal, tendon graft insertion (palmaris longus, plantaris, or toe extensor)
  • Outcomes variable - TAM typically 60-70% of normal
  • Reserved for ruptures with significant tendon retraction or poor tissue quality

Surgical intervention for complications requires careful patient selection and realistic expectation counseling, as outcomes rarely match those of uncomplicated primary repairs.

Rehabilitation Protocols

Quick Protocol Selection Guide

Patient ProfileProtocolKey FeatureRupture Risk
Non-compliant, elderly, poor cognitionPassive Motion (Kleinert/Duran)Rubber band traction or therapist-guided2-5%
Moderate compliance, concern for repairPlace-and-HoldPassive positioning, active hold5-8%
Compliant, strong repair, motivatedEarly Active MotionControlled active flexion day 3-510-15%

Early Active Motion (EAM) Protocol

Best functional outcomes - highest rupture risk

EAM Timeline

Post-opDays 0-2

Dorsal blocking splint: Wrist 20-30° flexion, MPs 50-70° flexion, IPs extended Immediate: Elevation, ice, edema control No exercises: Allow initial healing

Begin EAMDays 3-5

Active differential gliding:

  • FDS blocking: Hold proximal phalanx, flex PIPJ
  • FDP isolated: Flex DIPJ while PIPJ extended
  • Composite fist: All joints flexed together

Frequency: 10-12 repetitions every waking hour Splint: Remove for exercises, replace between sessions

ProgressiveWeeks 2-4

Continue active flexion: Increase hold time to 5 seconds Add passive extension: Therapist extends fingers to neutral Tenodesis exercises: Passive wrist motion with finger flexion/extension Monitor: Check for flexion lag (sign of rupture)

TransitionWeeks 5-6

Increase IP extension: Allow gentle passive extension beyond neutral Wean splint: Daytime only, continue night splinting Light ADLs: Eating, writing, grooming

Active MotionWeeks 6-8

Full active ROM: All joints, no restrictions on extension Gentle blocking exercises: Isolate FDS, FDP Begin light resistance: Therapy putty, light grip Discontinue splint: Week 8

StrengtheningWeeks 8-12

Progressive resistance: Hand grippers, weighted exercises Return to work: Light duty first, progress to full duty Sports: Non-contact first, contact sports month 3-4

EAM Evidence Base

Trumble et al. (2010): RCT comparing early active vs passive motion. EAM group had significantly better TAM (241° vs 220°), grip strength (38 kg vs 31 kg), but higher rupture rate (12% vs 4%). Most surgeons accept higher rupture risk for superior function in compliant patients.

Kleinert Protocol (Most Common Passive)

Rubber band traction maintains flexion

Kleinert Timeline

Day 0Post-op

Dorsal blocking splint: Wrist 20° flexion, MPs 60° flexion Rubber band attachment: Fingernail to volar forearm Mechanism: Band pulls finger into flexion, patient actively extends against band

Protected passiveWeeks 0-3

Patient exercises:

  • Active extension to neutral (against rubber band)
  • Passive flexion (rubber band pulls finger)
  • 10 repetitions every 1-2 hours

Therapist check: Weekly to adjust tension, monitor healing

TransitionWeeks 3-5

Remove rubber band: Week 3-4 Begin active flexion: Gentle composite fist Continue splint protection: Between exercises

Progressive activeWeeks 5-8

Increase active motion: Full active flexion/extension Wean splint: Discontinue week 6-7 Light strengthening: Week 8

Duran Protocol (Controlled Passive Motion)

Therapist-guided passive exercises, no rubber band

Duran Exercise Technique

Passive composite flexion: Therapist flexes all joints simultaneously to 3-5 mm from palm Passive differential: Isolate FDS (PIPJ flex, DIPJ extended), then FDP (DIPJ flex, PIPJ extended) Frequency: 5 sets of 10 repetitions, 4-6 times daily Patient role: Completely passive, therapist controls all motion

Duran protocol is beneficial when therapist supervision is available, eliminates rubber band compliance issues, but requires multiple weekly therapy sessions.

Place-and-Hold Protocol

Intermediate between passive and full active

Concept: Therapist passively positions finger in flexion, patient actively maintains position while therapist removes hand. This generates less tendon tension than full active flexion from extension.

Place-and-Hold Progression

Passive placementWeeks 0-3

Therapist: Passively flex finger to full fist position Patient: Actively hold position 5-10 seconds, then relax Frequency: 10 repetitions hourly Splint: Dorsal blocking between exercises

Add active returnWeeks 3-5

Continue place-and-hold: From full flexion Add: Active return to extension (still within splint) Increase hold time: 10-15 seconds

Transition to full activeWeeks 5-8

Full active flexion: Initiate from extended position Progress: Same as EAM protocol from week 5 onward

Place-and-hold is useful for patients with moderate compliance concerns or repairs with moderate strength. Generates intermediate tendon excursion and stress.

Zone-Specific Considerations

ZoneAnatomyRehabilitation ChallengeProtocol Modification
Zone 1 (FDP only)Distal to FDS insertionSingle tendon, usually good outcomesStandard EAM or passive, simpler than Zone 2
Zone 2 (No man's land)FDS and FDP in fibro-osseous sheathBoth tendons, high adhesion risk, most challengingStrict protocol adherence critical, consider passive if non-compliant
Zone 3 (Lumbrical origin)Proximal to A1 pulleyGood gliding, larger spaceEAM typically safe, faster progression
Zone 4 (Carpal tunnel)Median nerve, 8 tendonsNerve injury concern, adhesions to transverse ligamentNerve gliding exercises added, standard tendon protocol
Zone 5 (Forearm)Muscle-tendon junctionGood healing, ample spaceFaster progression, strengthening earlier (week 6)

Zone 2 Special Precautions

Why Zone 2 is different:

  • Both FDS and FDP tendons in restrictive sheath
  • Five annular pulleys (A1-A5) create friction
  • Synovial sheath from A1 to C3 - adhesion risk
  • FDS decussation at A2 pulley level - complex anatomy

Rehabilitation modifications:

  • Strict splint compliance mandatory
  • Consider passive protocols if any compliance doubt
  • Weekly therapist supervision minimum
  • Monitor for flexion lag (rupture sign) at every session

Complications and Problem-Solving

ComplicationRecognitionPreventionManagement
Rupture (10-15% EAM)Sudden loss of active flexion, flexion lag, palpable gapProtocol adherence, patient education, appropriate protocol selectionImmediate referral to surgeon, consider re-repair vs reconstruction
Adhesions (20-30%)Limited passive ROM, lacks final 10-20° flexionEarly motion protocols, differential gliding exercisesWeeks 8-12: Aggressive therapy, consider tenolysis if plateau after 3 months
Flexion contracturePIPJ cannot extend to neutral passivelySplint compliance, extension exercisesStatic progressive extension splinting, night extension splints
Quadriga effectLoss of independent finger flexion, all fingers flex togetherProper FDP tendon tensioning at surgerySurgical revision if severe, therapy for mild cases

Recognizing Tendon Rupture

Clinical signs:

  • Sudden loss of active flexion: Patient unable to flex DIPJ (FDP) or PIPJ (FDS)
  • Flexion lag: Passive flexion possible, active flexion cannot maintain position
  • Palpable gap: Tendon discontinuity on palpation (not always present)
  • Pain: Often minimal pain despite rupture

Management timeline:

  • First 2 weeks: Re-repair usually possible
  • Weeks 2-6: Re-repair difficult, consider two-stage reconstruction
  • After 6 weeks: Usually requires tendon graft or reconstruction

Outcome Measurement

Total Active Motion (TAM) - Strickland Criteria

Calculation: (Active PIPJ flexion + Active DIPJ flexion) - (Extension lag PIPJ + Extension lag DIPJ)

Grading:

  • Excellent: Greater than 85% of normal (greater than 220°)
  • Good: 70-84% of normal (180-219°)
  • Fair: 50-69% of normal (130-179°)
  • Poor: Under 50% of normal (under 130°)

Clinical Application: Good or excellent outcomes (greater than 70% TAM) correlate with high patient satisfaction and functional independence. Fair or poor outcomes often require tenolysis or reconstruction.

Evidence Base and Key Trials

Early Active vs Passive Motion RCT

1
Trumble et al. • JBJS Am (2010)
Key Findings:
  • RCT of 66 Zone 2 flexor tendon repairs: EAM vs passive motion
  • EAM group: Better TAM (241° vs 220°), grip strength (38kg vs 31kg)
  • Rupture rate: 12% EAM vs 4% passive motion
  • Patient satisfaction higher in EAM group despite ruptures
Clinical Implication: EAM protocols produce superior functional outcomes but require strong repair, compliant patient, and acceptance of higher rupture risk.
Limitation: Single-center study, all repairs by experienced hand surgeons with 4-strand core sutures.

Systematic Review of Rehabilitation Protocols

1
Hung et al. • J Hand Surg Am (2017)
Key Findings:
  • Meta-analysis of 23 studies, 1815 flexor tendon repairs
  • EAM protocols: 75% good-excellent outcomes, 8% rupture rate
  • Passive protocols: 65% good-excellent outcomes, 4% rupture rate
  • Place-and-hold: Intermediate outcomes (70% good-excellent, 6% rupture)
Clinical Implication: Protocol choice should balance functional goals with rupture risk based on patient compliance and repair strength.
Limitation: Heterogeneous protocols, variable repair techniques, limited comparative RCTs.

Four-Strand vs Two-Strand Core Suture Repairs

2
Strickland et al. • J Hand Surg Am (2005)
Key Findings:
  • Biomechanical study: 4-strand repairs 60% stronger than 2-strand
  • Clinical series: 4-strand repairs enabled EAM protocols safely
  • Gap formation reduced with 4-strand technique
  • Rupture rates with EAM: 3-5% for 4-strand, 15-20% for 2-strand
Clinical Implication: Modern multi-strand repairs (4-6 strands) provide biomechanical foundation for early active motion protocols.
Limitation: Cadaveric biomechanics may not reflect in vivo healing.

Kleinert vs Duran Passive Motion Protocols

2
May et al. • J Hand Surg Am (1992)
Key Findings:
  • Prospective comparison of Kleinert (rubber band) vs Duran (controlled passive) in 80 Zone 2 repairs
  • Kleinert group: 65% good-excellent outcomes, 4% rupture rate, better patient compliance
  • Duran group: 68% good-excellent outcomes, 3% rupture rate, required frequent therapy visits
  • No statistically significant difference in final TAM between protocols
Clinical Implication: Both passive protocols produce similar functional outcomes. Protocol selection should consider therapy resource availability and patient compliance with rubber band traction.
Limitation: Pre-dates modern multi-strand repairs, outcomes may not reflect current surgical techniques.

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: Protocol Selection for Zone 2 Repair

EXAMINER

"A 35-year-old tradesman sustained a Zone 2 FDP and FDS laceration to his index finger. You performed a 4-strand core suture repair with running epitenon suture. He is motivated to return to work quickly. What rehabilitation protocol would you recommend and why?"

EXCEPTIONAL ANSWER
For this compliant, motivated patient with a strong 4-strand repair, I would recommend an early active motion (EAM) protocol. This approach provides the best functional outcomes with superior tendon gliding and TAM scores, typically 70-85% of normal. I would counsel that EAM carries a 10-15% rupture risk compared to 3-5% with passive protocols, but the functional benefits typically outweigh this risk in motivated patients. The protocol involves dorsal blocking splint with wrist at 20-30° flexion, MPs at 50-70° flexion, and controlled active differential gliding exercises starting day 3-5. Critical phases include weeks 0-6 for protected motion, weeks 6-8 for transition to unrestricted motion, and weeks 8-12 for progressive strengthening. I would arrange weekly hand therapy with TAM measurements to monitor progress.
KEY POINTS TO SCORE
Protocol selection based on patient compliance, motivation, and repair strength
EAM superior outcomes but higher rupture risk - informed consent essential
Specific splint positioning: wrist 20-30° flex, MPs 50-70° flex
Timeline: Protected 0-6 weeks, transition 6-8 weeks, strengthen 8-12 weeks
COMMON TRAPS
✗Not mentioning rupture risk counseling - critical informed consent
✗Missing specific splint position angles
✗Failing to mention differential gliding exercises (FDS vs FDP isolation)
LIKELY FOLLOW-UPS
"What if the patient is elderly with poor compliance?"
"How would you modify the protocol for a Zone 1 injury?"
"What are the components of TAM measurement?"
VIVA SCENARIOChallenging

Scenario 2: Suspected Rupture at Week 3

EXAMINER

"A patient returns to clinic at 3 weeks post Zone 2 FDP repair on an EAM protocol. The therapist notes a new flexion lag at the DIPJ - the patient can passively flex to full range but cannot actively maintain the position. What is your assessment and management?"

EXCEPTIONAL ANSWER
This clinical presentation is highly concerning for tendon rupture. The key finding is flexion lag - passive motion intact but loss of active hold indicates tendon discontinuity. I would examine for palpable gap along the tendon course, though this is not always present. My management depends on timing: At 3 weeks post-repair, the repair site has minimal tensile strength and re-repair is technically feasible. I would counsel the patient that re-repair is indicated and arrange urgent surgery within 3-5 days. At surgery, I would explore the repair, debride frayed ends, and perform a revised 4-strand core suture if tendon quality permits. If significant tendon retraction has occurred or tissue quality is poor, I may need to consider two-stage reconstruction with silicone rod. Post-operatively, I would use a more conservative passive motion protocol given the re-repair. Prevention strategies for the future include careful patient selection for EAM protocols and considering passive protocols for less compliant patients.
KEY POINTS TO SCORE
Flexion lag = passive ROM intact, active flexion lost - pathognomonic for rupture
Timing matters: Under 2 weeks = re-repair possible, 2-6 weeks = difficult, over 6 weeks = reconstruction
Surgical decision-making: Assess tendon quality, retraction, and consider two-stage if needed
Post-rupture protocol more conservative - passive motion to prevent second rupture
COMMON TRAPS
✗Continuing therapy hoping it will improve - ruptures do not heal with therapy
✗Not examining for palpable gap
✗Missing the counseling about prevention and protocol selection
LIKELY FOLLOW-UPS
"When would you consider two-stage reconstruction over re-repair?"
"What is the timeline for silicone rod Hunter procedure?"
"How do outcomes of re-repair compare to primary repair?"
VIVA SCENARIOCritical

Scenario 3: Poor TAM at 12 Weeks

EXAMINER

"A patient is 12 weeks post Zone 2 flexor tendon repair with strict adherence to an EAM protocol. TAM is 120° (45% of normal) with significant limitations in both active and passive motion. No rupture occurred. What is the likely problem and management approach?"

EXCEPTIONAL ANSWER
A TAM of 120° at 12 weeks represents a poor outcome (under 50% of normal) despite protocol compliance and intact repair. This suggests significant adhesion formation between the repaired tendon and surrounding structures, particularly the flexor sheath and pulleys. The limitation in both active AND passive motion confirms this is an adhesion problem rather than weakness. My approach would be: First, ensure we are at least 12 weeks post-repair to allow tendon healing maturation. Second, intensify hand therapy with aggressive passive ROM, differential gliding exercises, and therapist-assisted stretching for 8-12 weeks. If there is no improvement after 3 months of intensive therapy (total 6 months post-injury), I would discuss tenolysis surgery. Tenolysis involves surgical release of adhesions under local anesthesia with sedation, allowing immediate intraoperative active motion to confirm adequate release. Post-tenolysis, early active motion is essential to prevent re-adhesion. I would counsel that tenolysis can improve TAM by 30-50°, but results rarely match primary repair outcomes.
KEY POINTS TO SCORE
Differentiate adhesions (limited passive and active) from weakness (limited active only)
Conservative management first: 3 months intensive therapy before surgery
Tenolysis timing: Not before 3 months (need healing), not after 12 months (matured scar)
Tenolysis technique: Local anesthesia allows immediate active motion verification
COMMON TRAPS
✗Operating too early before allowing therapy trial and tendon maturation
✗Not counseling realistic expectations - tenolysis rarely achieves normal motion
✗Missing differential diagnosis: Could weakness alone cause this? (No - passive would be full)
LIKELY FOLLOW-UPS
"What is the optimal timing window for tenolysis?"
"Why perform tenolysis under local rather than general anesthesia?"
"What are predictors of poor outcome after tenolysis?"

MCQ Practice Points

Zone 2 Definition

Q: Zone 2 of the flexor tendon system extends from which landmarks? A: A1 pulley to FDS insertion. Zone 2 (no man's land) encompasses the area where both FDS and FDP tendons run within the restrictive fibro-osseous sheath. This zone has the highest risk of adhesions and poorest outcomes historically.

Kleinert Protocol Mechanism

Q: In the Kleinert protocol, what motion does the patient actively perform? A: Active extension against rubber band traction. The rubber band maintains flexion passively; the patient actively extends the finger to neutral against the band resistance. This protects the repair from active flexion forces while maintaining gliding.

TAM Calculation

Q: How is Total Active Motion (TAM) calculated for flexor tendon outcomes? A: TAM equals (Active PIPJ flex plus Active DIPJ flex) minus (PIPJ extension lag plus DIPJ extension lag). Good outcome is 70-84% of normal, excellent is greater than 85%. This standardized measurement allows comparison across studies.

Rupture Risk Comparison

Q: What is the approximate rupture rate for early active motion protocols compared to passive motion? A: 10-15% for EAM vs 3-5% for passive motion. Despite higher rupture rates, EAM protocols produce superior functional outcomes (TAM typically 20-30° better) and are preferred for compliant patients with strong repairs.

Critical Healing Phase

Q: During which period is the flexor tendon repair weakest and most vulnerable to rupture? A: Weeks 1-3 post-repair. During this inflammatory phase, the repair has minimal intrinsic strength and depends entirely on suture holding power. Tensile strength increases significantly during the fibroplasia phase (weeks 3-8).

Australian Context

Epidemiology and Burden

Flexor tendon injuries in Australia predominantly affect working-age males (18-45 years) in manual occupations including construction, manufacturing, and agriculture. Zone 2 injuries constitute approximately 40% of all flexor tendon lacerations treated in Australian emergency departments. The incidence is estimated at 15-20 cases per 100,000 population annually, with higher rates in regional and remote areas where industrial and agricultural work predominates.

Healthcare System Considerations

Access to specialized hand therapy services varies significantly across Australian states and territories. Metropolitan areas typically have excellent access to certified hand therapists (CHTs) through both public hospital hand units and private practice networks. Regional centers may have limited hand therapy expertise, requiring patients to travel significant distances for weekly supervised sessions during the critical 0-6 week protected motion phase. Telehealth has emerged as an important adjunct for remote protocol monitoring, though it cannot fully replace in-person assessment of range of motion and differential gliding.

Management Approaches

Public hospital hand units in major Australian cities generally favor early active motion protocols for appropriately selected patients, supported by daily inpatient hand therapy during the first week followed by intensive outpatient therapy. Private practice management often utilizes place-and-hold or passive protocols due to concerns about therapy compliance and medicolegal risk. Rural and remote practitioners more commonly employ passive motion protocols (Kleinert or Duran) given limited access to frequent hand therapy supervision.

The Australian Hand Surgery Society guidelines recommend individualized protocol selection based on repair strength, zone of injury, and realistic assessment of therapy access and patient compliance. Medicare funding for hand therapy under chronic disease management plans provides up to five allied health sessions per calendar year, though this is often insufficient for optimal flexor tendon rehabilitation requiring 12-15 supervised sessions during the first three months.

Occupational Rehabilitation and Return to Work

WorkCover schemes across Australian jurisdictions vary in their support for prolonged rehabilitation periods. Most schemes fund hand therapy and occupational therapy assessment for graduated return to work. Manual laborers face particular challenges returning to full duties, often requiring 3-4 months before tolerating repetitive forceful grip activities. Vocational retraining may be necessary for workers with poor functional outcomes (TAM under 50%) who cannot meet the physical demands of their pre-injury occupation.

Medicolegal Considerations

Documentation requirements:

  • Informed consent must include rupture risk (10-15% for EAM, 3-5% passive)
  • Document patient compliance assessment and protocol selection rationale
  • Record therapy attendance and progression at each visit
  • Photograph any ruptures with notation of timing and circumstances

Common litigation issues:

  • Rupture without documented consent about protocol risks
  • Inadequate therapy follow-up leading to adhesions
  • Delayed recognition of rupture with missed re-repair window

FLEXOR TENDON REHABILITATION

High-Yield Exam Summary

Protocol Selection

  • •Passive Motion (Kleinert/Duran) = 3-5% rupture, non-compliant patients
  • •Place-and-Hold = 6-8% rupture, intermediate compliance
  • •Early Active Motion (EAM) = 10-15% rupture, best outcomes, compliant patients
  • •Zone 2 (no man's land) = both FDS and FDP in sheath, highest adhesion risk

Critical Timelines

  • •Days 0-2: Splint immobilization, no exercises
  • •Days 3-5: Begin EAM or passive protocol
  • •Weeks 0-6: Protected motion phase, strict splint compliance
  • •Weeks 6-8: Transition to unrestricted active motion
  • •Weeks 8-12: Progressive strengthening, return to work
  • •Weeks 1-3: Weakest repair, highest rupture risk

Splint Positioning

  • •Wrist: 20-30° flexion
  • •MPs: 50-70° flexion
  • •IPs: Neutral to slight flexion
  • •Kleinert: Add rubber band from nail to volar forearm

Exercise Components

  • •Differential gliding: Isolate FDS (PIPJ flex) vs FDP (DIPJ flex)
  • •Tenodesis: Passive wrist motion with finger motion
  • •Composite fist: All joints flexed together
  • •Place-and-hold: Passive positioning, active maintenance

Complications

  • •Rupture 10-15% EAM: Flexion lag, loss of active flexion, palpable gap
  • •Adhesions 20-30%: Limited passive ROM, consider tenolysis after 3 months therapy
  • •Flexion contracture: Cannot extend passively, extension splinting
  • •Re-repair window: Under 2 weeks best, after 6 weeks need reconstruction

Outcome Measurement

  • •TAM = (PIPJ flex + DIPJ flex) - (PIPJ lag + DIPJ lag)
  • •Excellent = over 85% (over 220°)
  • •Good = 70-84% (180-219°)
  • •Fair = 50-69% (130-179°)
  • •Poor = under 50% (under 130°)
Quick Stats
Reading Time112 min
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