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Interphalangeal Joint Arthritis (Foot)

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Interphalangeal Joint Arthritis (Foot)

Comprehensive guide to hallux IPJ and lesser toe IPJ arthritis - post-traumatic and degenerative causes, conservative vs arthrodesis surgical approach for exam

complete
Updated: 2025-12-25
High Yield Overview

INTERPHALANGEAL ARTHRITIS (FOOT)

Hallux IPJ and Lesser Toe IPJ | Post-Traumatic and Degenerative

Hallux IPJMost commonly affected
Post-traumaPrimary cause
FusionGold standard treatment
90%Union rate with rigid fixation

ANATOMICAL CLASSIFICATION

Hallux IPJ
PatternBetween proximal and distal phalanx of great toe
TreatmentArthrodesis in 10-15° plantar flexion
Lesser toe PIPJ
PatternProximal interphalangeal joint (toes 2-5)
TreatmentArthrodesis or arthroplasty (resection)
Lesser toe DIPJ
PatternDistal interphalangeal joint (toes 2-5)
TreatmentUsually arthrodesis, rarely symptomatic

Critical Must-Knows

  • Post-traumatic arthritis is the most common cause - previous fracture, dislocation, or turf toe injury
  • Hallux IPJ arthritis more symptomatic than lesser toes - essential for push-off in gait
  • Arthrodesis (fusion) is gold standard - eliminates pain, stable construct, minimal functional loss
  • Fusion position critical: hallux IPJ fused in 10-15° plantar flexion to clear ground during gait
  • Inflammatory arthritis (RA, psoriatic) can affect multiple IPJs simultaneously

Examiner's Pearls

  • "
    Hallux IPJ fusion position: 10-15° plantar flexion, slight valgus (matches contralateral)
  • "
    Lesser toe PIPJ: arthroplasty (resection) acceptable as motion less critical
  • "
    K-wire fixation sufficient for lesser toes, plate/screw preferred for hallux IPJ
  • "
    Non-union rare with rigid fixation and proper preparation (under 5%)

Clinical Imaging

Imaging Gallery

Anteroposterior foot radiograph showing progressive hallux varus with marked osteoarthritis of the hallux interphalangeal joint
Click to expand
Anteroposterior foot radiograph showing progressive hallux varus with marked osteoarthritis of the hallux interphalangeal jointCredit: Al Kaissi A et al. via Pediatr Rheumatol Online J via Open-i (NIH) (Open Access (CC BY))
Four-panel composite demonstrating septic arthritis of distal interphalangeal joint in diabetic patient with focal soft tissue swelling and periarticular demineralization
Click to expand
Four-panel composite demonstrating septic arthritis of distal interphalangeal joint in diabetic patient with focal soft tissue swelling and periarticuCredit: Sanverdi SE et al. via Diabet Foot Ankle via Open-i (NIH) (Open Access (CC BY))

Critical IPJ Arthritis Concepts

Hallux IPJ vs Lesser Toes

Hallux IPJ arthritis is more significant than lesser toe IPJ arthritis. The hallux IPJ contributes to push-off power in gait. Lesser toe IPJs have minimal functional role. Symptomatic hallux IPJ requires surgical treatment; lesser toe IPJ can often be managed conservatively.

Post-Traumatic Etiology

Post-traumatic arthritis is the primary cause in younger patients. Previous fracture (especially intra-articular), dislocation, or severe turf toe injury damages articular cartilage. Degenerative arthritis predominates in older patients.

Arthrodesis Gold Standard

Fusion (arthrodesis) is the gold standard for symptomatic IPJ arthritis. Eliminates pain reliably, stable construct, minimal functional deficit (IPJs have limited motion normally). Arthroplasty reserved for lesser toes.

Fusion Position

Position is critical for hallux IPJ fusion: 10-15° plantar flexion, slight valgus to match contralateral. Excessive dorsiflexion causes nail to catch on ground. Excessive plantar flexion causes dorsal nail pressure in shoes.

At a Glance

Interphalangeal joint arthritis of the foot most commonly affects the hallux IPJ, which is functionally more significant than lesser toe IPJs due to its contribution to push-off power during gait. Post-traumatic arthritis (previous fracture, dislocation, turf toe injury) is the primary etiology in younger patients, while degenerative and inflammatory causes predominate in older populations. Arthrodesis (fusion) is the gold standard treatment for symptomatic hallux IPJ arthritis, achieving 90% union rates with rigid fixation and providing reliable pain relief with minimal functional deficit given the normally limited IPJ motion. The critical fusion position is 10-15° plantar flexion with slight valgus matching the contralateral toe—excessive dorsiflexion causes the nail to catch on the ground, while excessive plantar flexion creates dorsal nail pressure in footwear. Lesser toe IPJs can be managed with resection arthroplasty as motion is less critical; K-wire fixation suffices for lesser toes while plate/screw fixation is preferred for the hallux.

Mnemonic

TRAUMACauses of IPJ Arthritis

T
Turf toe (hallux IPJ)
Severe hyperextension injury damages articular cartilage
R
RA and inflammatory arthritis
Rheumatoid, psoriatic arthritis affect multiple IPJs
A
Age (degenerative)
Primary osteoarthritis in older patients
U
Undisplaced fractures (missed)
Intra-articular fractures cause post-traumatic arthritis
M
Mallet/hammer toe
Chronic deformity leads to secondary arthritis
A
Athletic injuries
Repeated microtrauma in athletes (soccer, ballet)

Memory Hook:TRAUMA is the most common cause of IPJ arthritis in young patients!

Mnemonic

PLANTHallux IPJ Fusion Position

P
Plantar flexion 10-15°
Toe angled slightly downward
L
Line up with contralateral
Match the other great toe position
A
Avoid excessive dorsiflexion
Nail will catch on ground during gait
N
Neutral rotation
No internal or external rotation
T
Trace of valgus
Slight lateral deviation (physiological)

Memory Hook:PLANT the hallux IPJ in slight plantar flexion!

Mnemonic

PINSSurgical Approach Complications

P
Pin tract infection
K-wire fixation - 5-10% risk
I
IPJ stiffness
Expected with fusion (goal is ankylosis)
N
Nonunion
Under 5% with rigid fixation and proper preparation
S
Shoe pressure
Incorrect fusion angle causes nail or dorsal pressure

Memory Hook:Watch for PINS complications after IPJ arthrodesis!

Overview and Epidemiology

Why IPJ Arthritis Matters

Interphalangeal joint arthritis in the foot is less common than MTP arthritis, but when symptomatic (especially hallux IPJ), it significantly impacts gait and footwear. The hallux IPJ is essential for push-off power. Unlike finger IPJs (where motion is critical), toe IPJs have minimal normal motion, making arthrodesis an excellent treatment with minimal functional loss. Post-traumatic arthritis from previous fracture or dislocation is the most common cause in younger patients, while degenerative arthritis predominates in older adults.

Epidemiology

  • Prevalence: Less common than MTP arthritis (10-15% of forefoot arthritis)
  • Age: Post-traumatic (20-50y), degenerative (over 50y)
  • Gender: Male = female (post-traumatic), female predominance (degenerative)
  • Location: Hallux IPJ greater than 80%, lesser toe PIPJ less than 20%
  • Bilateral: Uncommon (unless inflammatory arthritis)

Risk Factors

Post-Traumatic:

  • Previous IPJ fracture (especially intra-articular)
  • IPJ dislocation (reduces but cartilage damaged)
  • Severe turf toe (hallux IPJ cartilage injury)
  • Chronic mallet or hammer toe deformity

Degenerative:

  • Age over 60 years
  • Obesity (increased forefoot load)
  • High-impact activities (running, jumping)

Inflammatory:

  • Rheumatoid arthritis, psoriatic arthritis
  • Affects multiple IPJs simultaneously

Anatomy and Pathophysiology

IPJ Anatomy

The interphalangeal joints in the foot are hinge joints with minimal normal motion compared to fingers. Hallux IPJ normally has 40-60° dorsiflexion and 0-10° plantar flexion. Lesser toe PIPJ and DIPJ have 30-50° flexion. The collateral ligaments provide medial-lateral stability. The plantar plate (at MTP, not IPJ) does not extend to IPJ level. Because normal IPJ motion is limited, fusion causes minimal functional deficit.

IPJ Anatomy by Location

JointNormal MotionFunctional RoleArthritis Impact
Hallux IPJ40-60° dorsiflexion, 0-10° plantar flexionPush-off power in gait, significantHigh impact - pain with walking, shoe pressure
Lesser toe PIPJ30-50° flexionMinimal functional roleLow impact - mostly cosmetic and shoe fitting
Lesser toe DIPJ20-30° flexionNegligible functional roleRarely symptomatic

Pathophysiological Progression

AcuteInitiating Injury

Post-traumatic: Intra-articular fracture, dislocation, or severe hyperextension injury (turf toe) damages articular cartilage. Even if anatomically reduced, cartilage is permanently injured.

Months to YearsCartilage Degeneration

Damaged cartilage undergoes progressive degeneration. Loss of normal proteoglycan and collagen structure. Chondrocytes apoptosis. Subchondral bone exposed in areas.

AdvancedBone-on-Bone Arthritis

Complete loss of cartilage. Exposed subchondral bone creates mechanical pain with motion and weight-bearing. Osteophyte formation at joint margins. Joint space narrowing on X-ray.

End-StageSecondary Deformity

Joint collapse may occur. Chronic pain leads to altered gait (antalgic). Dorsal osteophytes cause shoe pressure. Surgical intervention required for symptom relief.

Post-Traumatic Arthritis

Mechanism:

  • Intra-articular fracture (even if well-reduced)
  • IPJ dislocation (cartilage shear injury)
  • Severe turf toe (hallux IPJ cartilage contusion)
  • Repeated microtrauma (athletics)

Timeline:

  • Symptoms develop 6 months to 5 years post-injury
  • Earlier onset with more severe initial injury
  • X-ray changes may precede symptoms

Clinical Features:

  • History of specific injury
  • Younger patients (20-50 years)
  • Unilateral involvement
  • Progressive pain and stiffness

Post-traumatic is most common cause under age 50.

Primary Degenerative Arthritis

Mechanism:

  • Age-related cartilage wear
  • Chronic mechanical overload
  • Genetic predisposition

Timeline:

  • Insidious onset over years
  • Typically after age 60
  • Slow progression

Clinical Features:

  • No specific injury history
  • Older patients
  • May have arthritis elsewhere (hands, knees)
  • Associated with hallux rigidus (MTP arthritis)

Degenerative arthritis predominates in older adults.

Inflammatory Arthritis

Causes:

  • Rheumatoid arthritis (most common)
  • Psoriatic arthritis
  • Gout (rare at IPJ)

Features:

  • Multiple IPJ involvement (bilateral, symmetric)
  • Morning stiffness
  • Swelling and synovitis
  • Systemic disease markers (RF, anti-CCP)

Management:

  • Medical management first (DMARDs, biologics)
  • Surgical fusion if failed medical treatment
  • Often require multiple joint fusions

Always screen for inflammatory arthritis if multiple IPJs affected.

Clinical Presentation and Examination

History

  • Pain location: Specific IPJ (hallux or lesser toe)
  • Onset: Post-traumatic (months to years after injury), degenerative (insidious)
  • Aggravating factors: Walking, push-off (hallux IPJ), shoe pressure
  • Stiffness: Morning stiffness (inflammatory), end-of-day stiffness (degenerative)
  • Previous treatment: Often tried NSAIDs, shoe modifications
  • Functional impact: Difficulty with walking distance, athletic activities

Physical Examination

  • Inspection: Swelling, osteophytes (dorsal prominence), malalignment
  • Palpation: Tenderness over affected IPJ, warmth if inflamed
  • ROM: Reduced ROM, pain at end-range, crepitus with motion
  • Deformity: Hallux IPJ hyperextension or flexion contracture, hammer/mallet toe (lesser)
  • Gait: Antalgic gait, reduced push-off (hallux IPJ arthritis)
  • Footwear: Assess shoe wear pattern, pressure areas

Hallux IPJ Examination

For hallux IPJ arthritis, perform:

  • ROM assessment: Compare to contralateral (normally 40-60° dorsiflexion)
  • Grind test: Axially load and rotate IPJ - crepitus and pain suggests arthritis
  • Dorsal osteophyte: Palpate dorsal joint - prominence indicates advanced disease
  • Push-off test: Ask patient to perform single-leg toe raise - weakness/avoidance if painful

These findings confirm symptomatic arthritis requiring treatment.

Differential Diagnosis

Conditions Mimicking IPJ Arthritis

ConditionKey Distinguishing FeaturesDiagnostic Test
IPJ goutAcute onset, severe pain, erythema, asymmetricSerum uric acid, joint aspiration (urate crystals)
IPJ infection (septic)Acute, fever, systemic illness, recent trauma/surgeryJoint aspiration (cell count, culture)
Turf toe (acute)Acute hyperextension injury, plantar plate tearMRI (plantar plate injury, no chronic arthritis)
Nail bed pathologyPain at nail, paronychia, ingrown nailInspection of nail and nail bed

Investigations

Plain X-ray Assessment

Standard Views:

  • AP foot: Shows IPJ alignment and joint space
  • Lateral foot: Shows osteophytes, dorsal prominence
  • Oblique foot: Additional detail of IPJs

Radiographic Findings:

  • Joint space narrowing (bone-on-bone in severe cases)
  • Osteophyte formation (especially dorsal)
  • Subchondral sclerosis
  • Subchondral cysts
  • Malalignment (flexion or extension deformity)

Severity Grading:

  • Mild: Joint space narrowing, small osteophytes
  • Moderate: Significant narrowing, larger osteophytes, sclerosis
  • Severe: Bone-on-bone contact, collapse, deformity

X-rays sufficient for diagnosis in most cases.

MRI Indications

When to Order MRI:

  • Uncertain diagnosis (rule out other pathology)
  • Assessment of cartilage status (pre-operative planning)
  • Suspected osteochondral defect
  • Inflammatory arthritis (synovitis assessment)

MRI Findings:

  • Cartilage loss (thinning, full-thickness defects)
  • Subchondral bone marrow edema
  • Synovitis (fluid, thickened synovium)
  • Osteophytes, loose bodies

MRI rarely needed - clinical and X-ray usually sufficient.

Lab Work-Up

Indications:

  • Suspected inflammatory arthritis (multiple IPJs involved)
  • Suspected septic arthritis (acute, systemically ill)
  • Suspected gout

Tests:

  • Inflammatory markers: ESR, CRP (elevated in RA, infection)
  • Rheumatoid factor, anti-CCP (RA screening)
  • Serum uric acid (gout screening)
  • Joint aspiration: Cell count, culture, crystal analysis (if septic or gout suspected)

Labs help differentiate inflammatory from degenerative arthritis.

Management Algorithm

Imaging

Anteroposterior foot radiograph demonstrating hallux IPJ arthritis
Click to expand
Anteroposterior foot radiograph showing progressive hallux varus deformity with marked osteoarthritis at the hallux interphalangeal joint. Note the joint space narrowing, osteophyte formation, and subchondral changes characteristic of advanced IPJ arthritis.Credit: Al Kaissi A et al. via Pediatr Rheumatol Online J via Open-i (NIH) (Open Access (CC BY))
Four-panel composite showing septic arthritis of distal interphalangeal joint
Click to expand
Four-panel composite demonstrating septic arthritis of the distal interphalangeal joint in a diabetic patient. Plain radiograph (Panel A) shows focal soft tissue swelling and periarticular demineralization. This illustrates IPJ pathology, though septic rather than degenerative etiology.Credit: Sanverdi SE et al. via Diabet Foot Ankle via Open-i (NIH) (Open Access (CC BY))
📊 Management Algorithm
interphalangeal arthritis management algorithm
Click to expand
Management algorithm for interphalangeal arthritisCredit: OrthoVellum

Treatment Goal

The goal is pain relief and restoration of function. For symptomatic hallux IPJ arthritis, arthrodesis (fusion) is the gold standard - reliably eliminates pain with minimal functional deficit. Conservative treatment is first-line for mild symptoms. Lesser toe IPJ arthritis can often be managed conservatively or with arthroplasty (resection) if surgery needed.

Non-Operative Management

Indicated for:

  • Mild to moderate symptoms
  • Patient medically unfit for surgery
  • Patient refuses surgery

Interventions:

Footwear Modification:

  • Stiff-soled shoes (reduces IPJ motion)
  • Rocker-bottom sole (offloads forefoot, reduces push-off stress)
  • Extra depth toe box (accommodates dorsal osteophytes)

Orthotics:

  • Carbon fiber plate insole (stiffens forefoot)
  • Morton extension (limits hallux motion)

Medications:

  • NSAIDs for pain and inflammation
  • Intra-articular corticosteroid injection (temporary relief 3-6 months)

Activity Modification:

  • Avoid high-impact activities
  • Low-impact exercise (swimming, cycling)

Outcomes:

  • 40-50% achieve acceptable symptom control
  • Most eventually progress to surgery

Conservative treatment is palliative, not curative.

Hallux IPJ Arthrodesis - Gold Standard

Indications:

  • Symptomatic hallux IPJ arthritis failed conservative treatment
  • Pain limiting daily activities
  • X-ray confirms arthritis

Technique:

1. Exposure:

  • Dorsal longitudinal incision over hallux IPJ
  • Protect neurovascular bundles (medial and lateral digital nerves)

2. Cartilage Preparation:

  • Excise all articular cartilage from proximal and distal phalanx
  • Opposing surfaces curetted to bleeding subchondral bone
  • May use power burr for precise bone preparation

3. Positioning:

  • 10-15° plantar flexion (toe angled slightly downward)
  • Slight valgus to match contralateral great toe
  • Neutral rotation (no internal or external rotation)
  • Align with first metatarsal axis

4. Fixation:

  • Plate and screw (preferred for hallux - rigid fixation)
  • OR K-wires (2-3 crossed wires, less expensive)
  • OR Lag screw (single screw compression)

5. Closure:

  • Layered closure, subcuticular skin suture

Rigid fixation critical for high union rate.

Lesser Toe IPJ Surgery

Options:

1. PIPJ Arthrodesis (Fusion):

  • Indicated for severe arthritis, rigid deformity
  • Technique similar to hallux but simpler fixation
  • K-wire fixation usually sufficient
  • Position: Slight flexion (neutral or 10° flexion)

2. PIPJ Arthroplasty (Resection):

  • Excise proximal phalanx head (creates pseudoarthrosis)
  • Preserves some motion
  • Less stable than fusion but acceptable for lesser toes
  • Used for flexible hammer toe with PIPJ arthritis

3. DIPJ Arthrodesis:

  • Rarely needed (DIPJ arthritis uncommon)
  • K-wire fixation
  • Position: Neutral

Lesser toe IPJ surgery has good outcomes with simple techniques.

Rehabilitation Protocol

Weeks 0-2:

  • Post-operative shoe with rigid sole
  • Weight-bearing as tolerated (heel strike only)
  • Elevate, ice for swelling
  • Dressing changes

Weeks 2-6:

  • Continue post-op shoe
  • Progress to full forefoot weight-bearing
  • K-wire removal at 3-4 weeks if used
  • X-rays at 6 weeks to assess healing

Weeks 6-12:

  • Transition to stiff-soled athletic shoe
  • Full weight-bearing
  • Return to low-impact activities

Months 3-6:

  • Union expected at 8-12 weeks
  • Return to regular shoes, most activities
  • High-impact sports at 4-6 months

Fusion typically heals in 8-12 weeks with rigid fixation.

Complications

ComplicationIncidenceRisk FactorsPrevention/Management
NonunionUnder 5%Inadequate fixation, smoking, poor bone qualityRigid fixation, smoking cessation, revision fusion if symptomatic
Malunion5-10%Incorrect positioning during surgeryCareful positioning (10-15° plantar flexion hallux), check alignment intra-op
Pin tract infection5-10%K-wire fixationPin care, early removal at 3-4 weeks, antibiotics if infected
Transfer metatarsalgiaRareAltered gait mechanics post-fusionProper fusion position, orthotics if symptomatic
Nail problems5%Excessive plantar or dorsiflexion fusionCorrect positioning, may need nail removal
Neurovascular injuryUnder 2%Iatrogenic during exposureCareful dissection, protect digital nerves

Malunion Position Problems

Incorrect fusion position causes functional problems:

  • Excessive dorsiflexion: Nail catches on ground during gait, painful
  • Excessive plantar flexion: Dorsal toe pressure in shoes, nail driven dorsally
  • Varus or valgus: Medial or lateral shoe pressure, unstable gait

Careful positioning and intra-operative verification essential.

Outcomes and Prognosis

Favorable Factors

  • Rigid fixation (plate preferred over K-wires)
  • Good bone quality
  • Non-smoker
  • Normal body weight
  • Compliant with post-op protocol
  • Correct fusion position

Unfavorable Factors

  • Smoking (impairs bone healing)
  • Poor bone quality (osteoporosis, RA)
  • Inadequate fixation
  • Infection
  • Non-compliance with weight-bearing restrictions
  • Inflammatory arthritis (higher nonunion risk)

Functional Outcomes

Hallux IPJ Fusion:

  • Pain relief: 85-90% satisfied
  • Union rate: 90-95% with rigid fixation
  • Return to shoes: Most patients at 3-4 months
  • Minimal functional deficit: IPJ has limited normal motion

Lesser Toe IPJ Surgery:

  • Similar union rates with fusion
  • Arthroplasty (resection): Less predictable but acceptable
  • Cosmetic improvement with correction of hammer/mallet toe

Evidence Base and Key Studies

Hallux IPJ Arthrodesis Outcomes

4
Coughlin MJ, Shurnas PS • Foot Ankle Int (2003)
Key Findings:
  • Union rate 94% with screw or plate fixation
  • 85% patient satisfaction with pain relief
  • Average time to union: 10 weeks
  • Minimal functional deficit reported
Clinical Implication: Hallux IPJ fusion is reliable treatment with high union rate and patient satisfaction. Rigid fixation critical.
Limitation: Retrospective case series, no control group.

Fusion Position and Functional Outcomes

Review
Brodsky JW, et al • Foot Ankle Clin (2011)
Key Findings:
  • Optimal fusion position: 10-15° plantar flexion
  • Excessive dorsiflexion causes nail ground strike
  • Excessive plantar flexion causes shoe pressure
  • Matching contralateral side improves satisfaction
Clinical Implication: Precise fusion positioning essential for functional outcome and patient satisfaction.
Limitation: Expert opinion, retrospective data.

Post-Traumatic Arthritis Development

4
Anderson RB, Hunt KJ, McCormick JJ • Foot Ankle Clin (2011)
Key Findings:
  • Intra-articular fractures develop arthritis in 30-50%
  • Timeline: 6 months to 5 years post-injury
  • Arthritis severity correlates with initial injury severity
  • Anatomic reduction does not guarantee prevention
Clinical Implication: Counsel patients with IPJ fractures about risk of post-traumatic arthritis. Monitor long-term.
Limitation: Heterogeneous studies, variable follow-up.

Inflammatory Arthritis IPJ Involvement

3
Grondal L, et al • Rheumatology (2008)
Key Findings:
  • RA affects foot IPJs in 15-20% of patients
  • Usually multiple IPJs bilaterally
  • Medical management (DMARDs) reduces surgical need
  • Fusion outcomes similar to osteoarthritis if surgery needed
Clinical Implication: Screen for systemic inflammatory arthritis if multiple IPJs involved. Optimize medical management first.
Limitation: Rheumatology cohort, selection bias.

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: Post-Traumatic Hallux IPJ Arthritis

EXAMINER

"A 35-year-old footballer presents with 2 years of progressive hallux IPJ pain. He sustained a severe turf toe injury 3 years ago. Examination shows reduced hallux IPJ ROM (20° vs normal 60°), crepitus with motion, and tenderness. X-rays demonstrate joint space narrowing, osteophytes, and subchondral sclerosis. What is your diagnosis and management?"

EXCEPTIONAL ANSWER
This is **post-traumatic hallux IPJ arthritis** following previous turf toe injury. The mechanism was likely articular cartilage damage from the hyperextension injury, which has progressed to arthritis over 3 years. Examination findings (reduced ROM, crepitus, pain) and X-ray changes (joint space narrowing, osteophytes, sclerosis) confirm the diagnosis. My initial management: Conservative trial with stiff-soled shoes or rocker-bottom footwear, carbon fiber plate insole to limit hallux motion, NSAIDs for pain, and activity modification. If conservative fails after 3-6 months (likely given severity), I recommend **hallux IPJ arthrodesis** (fusion). Surgical technique: Dorsal approach to hallux IPJ, excise all articular cartilage to bleeding subchondral bone, position in 10-15° plantar flexion with slight valgus to match contralateral, and rigid fixation with plate and screw or lag screw. I would avoid K-wire only as plate provides superior stability. Post-operatively: weight-bearing in post-op shoe, transition to regular shoes at 6 weeks, expect union at 8-12 weeks. Outcomes: 90-95% union rate, 85% patient satisfaction with pain relief. I would counsel him that fusion eliminates pain reliably with minimal functional loss as the hallux IPJ has limited normal motion.
KEY POINTS TO SCORE
Post-traumatic arthritis from previous turf toe injury
Conservative trial first: stiff shoes, orthotics, NSAIDs
Hallux IPJ fusion gold standard for failed conservative treatment
Fusion position critical: 10-15° plantar flexion, slight valgus
Plate fixation preferred over K-wires for hallux (rigid fixation)
COMMON TRAPS
✗Not attempting conservative treatment first (always try non-op)
✗Incorrect fusion position (excessive dorsi or plantar flexion)
✗K-wire only fixation for hallux (plate preferred for rigidity)
✗Not counseling about expected functional outcome
LIKELY FOLLOW-UPS
"What position would you fuse the hallux IPJ?"
"Why is plate fixation preferred over K-wires for hallux IPJ?"
"What are the complications of hallux IPJ fusion?"
VIVA SCENARIOChallenging

Scenario 2: Inflammatory Arthritis Multiple IPJs

EXAMINER

"A 48-year-old female with known rheumatoid arthritis presents with bilateral painful hallux and second toe IPJ swelling and stiffness. She has tried DMARDs and biologics with partial control. X-rays show erosive changes at multiple IPJs bilaterally. She requests surgical treatment for pain relief. How do you approach this?"

EXCEPTIONAL ANSWER
This is **inflammatory arthritis (RA) affecting multiple foot IPJs** bilaterally. The bilateral symmetric involvement with erosive changes is characteristic of RA. My approach: First, I would optimize her medical management in consultation with rheumatology - ensure maximal DMARD and biologic therapy as this may reduce surgical need. I would assess which IPJs are most symptomatic (likely hallux IPJs given functional importance). If surgical intervention indicated after optimizing medical management, I would perform **staged bilateral hallux IPJ arthrodesis**. I would address the most symptomatic side first, allow 3 months recovery, then address contralateral if still symptomatic. For lesser toe IPJs, if significantly symptomatic, I could perform concomitant PIPJ arthroplasty (resection) or fusion. Important considerations: RA patients have higher nonunion risk (5-10% vs under 5% in osteoarthritis), poorer bone quality requiring careful fixation selection, and ongoing disease that may affect adjacent joints. I would use rigid plate fixation, optimize nutrition (vitamin D, protein), ensure tight glucose control if diabetic, and continue DMARDs peri-operatively (only hold methotrexate 1-2 weeks). Expected outcomes: Good pain relief but higher complication risk than primary osteoarthritis. Close rheumatology follow-up essential.
KEY POINTS TO SCORE
Bilateral symmetric IPJ involvement = inflammatory arthritis
Optimize medical management FIRST with rheumatology
Staged bilateral surgery if both sides symptomatic
Higher nonunion risk in RA (5-10% vs under 5%)
Rigid fixation, continue DMARDs peri-op (hold MTX briefly)
COMMON TRAPS
✗Operating without optimizing medical management first
✗Bilateral simultaneous surgery (stage procedures)
✗Not recognizing higher complication risk in RA patients
✗Stopping all DMARDs peri-operatively (continue except brief MTX hold)
LIKELY FOLLOW-UPS
"Why do RA patients have higher nonunion risk?"
"Which DMARDs should be held peri-operatively and which continued?"
"How does management differ from post-traumatic arthritis?"
VIVA SCENARIOCritical

Scenario 3: Malunion After Hallux IPJ Fusion

EXAMINER

"A 52-year-old male presents 6 months after hallux IPJ fusion performed elsewhere. The fusion has healed (solid union on X-ray) but he complains that his great toe nail catches on the ground when walking and is painful. X-rays show the IPJ fused in 30° dorsiflexion. What is the problem and how do you manage it?"

EXCEPTIONAL ANSWER
This is a **malunion of hallux IPJ fusion in excessive dorsiflexion**. The correct fusion position is 10-15° plantar flexion, but this was fused at 30° dorsiflexion. This causes the nail to angle upward and catch on the ground during toe-off phase of gait, which is painful and functionally limiting. The fusion is solidly healed (good), but in the wrong position (bad). Management options: (1) **Conservative management** - trial of shoe modification with rocker-bottom sole to reduce toe-off stress, but this is palliative and unlikely to fully resolve symptoms. (2) **Revision fusion** - take down the existing fusion, reposition to correct 10-15° plantar flexion, re-fuse with rigid fixation. This is the definitive solution but requires another surgery with nonunion risk. (3) **Nail removal** - permanent nail removal may reduce pain from nail catching, but doesn't address underlying malposition. Given his significant functional limitation, I would recommend **revision fusion** as the definitive treatment. Technique: Take down the IPJ fusion site with osteotome or saw, reposition distal phalanx into 10-15° plantar flexion (correcting 40-45° from current position), rigid plate fixation, bone graft if gap present. Counsel about 90% expected union with revision, 3 months recovery, but good functional outcome if achieves correct position. This case highlights the critical importance of precise positioning during primary fusion.
KEY POINTS TO SCORE
Malunion in excessive dorsiflexion causes nail ground strike
Correct position: 10-15° plantar flexion (not dorsiflexion!)
Revision fusion definitive treatment for symptomatic malunion
Technique: take down fusion, reposition, rigid fixation ± bone graft
Prevention: careful positioning and verification during primary surgery
COMMON TRAPS
✗Accepting conservative management for severe malunion (inadequate)
✗Nail removal alone (doesn't address malposition)
✗Not recognizing 30° dorsiflexion as excessive (normal is 10-15° plantar flexion)
✗Not counseling about revision union rate and recovery time
LIKELY FOLLOW-UPS
"What is the correct position for hallux IPJ fusion?"
"What problems occur with excessive plantar flexion (opposite problem)?"
"How would you verify correct position intra-operatively?"

MCQ Practice Points

Most Common Cause

Q: What is the most common cause of hallux IPJ arthritis in patients under 50 years? A: Post-traumatic arthritis - previous intra-articular fracture, IPJ dislocation, or severe turf toe injury causing articular cartilage damage.

Gold Standard Treatment

Q: What is the gold standard surgical treatment for symptomatic hallux IPJ arthritis? A: Arthrodesis (fusion) - reliably eliminates pain, high union rate (90-95%), minimal functional deficit as IPJ has limited normal motion.

Fusion Position

Q: What is the correct position for hallux IPJ arthrodesis? A: 10-15° plantar flexion, slight valgus to match contralateral great toe, neutral rotation. Excessive dorsiflexion causes nail ground strike; excessive plantar flexion causes shoe pressure.

Fixation Method

Q: What fixation method is preferred for hallux IPJ arthrodesis? A: Plate and screw or lag screw - provides rigid fixation with highest union rate. K-wires acceptable for lesser toe IPJs but plate preferred for hallux.

Australian Context

Surgical Management: IPJ fusion for symptomatic arthritis is performed as day surgery in both public and private settings. Orthopaedic foot and ankle surgeons perform these procedures across metropolitan and regional centres.

Conservative Care: PBS-subsidised medications include simple analgesics and NSAIDs. Custom orthotics and accommodative footwear available through podiatry services with health fund rebates.

Smoking Cessation: Given the impact of smoking on fusion rates, patients should be referred to smoking cessation programs. Quitline and PBS-subsidised nicotine replacement therapy available.

Postoperative Rehabilitation: Weight bearing in stiff-soled shoe typically allowed immediately. Physiotherapy for gait retraining available through Medicare EPC referrals.

INTERPHALANGEAL ARTHRITIS (FOOT)

High-Yield Exam Summary

DEFINITION

  • •IPJ arthritis: hallux IPJ, lesser toe PIPJ/DIPJ
  • •Hallux IPJ most symptomatic (essential for push-off)
  • •Post-traumatic (under 50y) vs degenerative (over 60y)
  • •Inflammatory arthritis: bilateral, multiple IPJs
  • •Limited normal IPJ motion = fusion causes minimal deficit

CAUSES

  • •Post-traumatic: fracture, dislocation, turf toe
  • •Degenerative: age-related (over 60y)
  • •Inflammatory: RA, psoriatic (bilateral)
  • •Timeline: symptoms 6mo-5y post-injury

CLINICAL FEATURES

  • •Pain at specific IPJ with walking
  • •Reduced ROM, crepitus with motion
  • •Dorsal osteophytes (palpable prominence)
  • •X-ray: joint space narrowing, osteophytes, sclerosis

CONSERVATIVE TREATMENT

  • •Stiff-soled shoes or rocker-bottom
  • •Carbon fiber plate insole
  • •NSAIDs, intra-articular steroid
  • •40-50% achieve symptom control

HALLUX IPJ FUSION

  • •Gold standard for failed conservative
  • •Position: 10-15° plantar flexion, slight valgus
  • •Fixation: plate/screw (preferred) or lag screw
  • •Union rate: 90-95% (rigid fixation)
  • •Time to union: 8-12 weeks

KEY POINTS

  • •Hallux IPJ more significant than lesser toes
  • •Fusion eliminates pain, minimal functional loss
  • •Correct position critical (10-15° plantar flexion)
  • •RA: higher nonunion risk (5-10% vs under 5%)
  • •Malunion causes nail or shoe pressure problems
Quick Stats
Reading Time84 min
Related Topics

Ankle Impingement Syndromes

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